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A drug repurposing screen identifies hepatitis C antivirals as inhibitors of the SARS-CoV2 main protease

PloS one, 2021-02, Vol.16 (2), p.e0245962-e0245962

2021

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Motor neuron disease-associated loss of nuclear TDP-43 is linked to DNA double-strand break repair defects

Proceedings of the National Academy of Sciences - PNAS, 2019-03, Vol.116 (10), p.4696-4705

2019

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The tau tubulin kinases TTBK1/2 promote accumulation of pathological TDP-43

PLoS genetics, 2014-12, Vol.10 (12), p.e1004803-e1004803

2014

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Protein Kinase CK‑1 Inhibitors As New Potential Drugs for Amyotrophic Lateral Sclerosis

Journal of medicinal chemistry, 2014-03, Vol.57 (6), p.2755-2772

2014

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Constitutive XBP-1s-mediated activation of the endoplasmic reticulum unfolded protein response protects against pathological tau

Nature communications, 2019-09, Vol.10 (1), p.4443-12, Article 4443

2019

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Phosphorylation promotes neurotoxicity in a Caenorhabditis elegans model of TDP-43 proteinopathy

The Journal of neuroscience, 2010-12, Vol.30 (48), p.16208-16219

2010

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TDP-43/FUS in motor neuron disease: Complexity and challenges

Progress in neurobiology, 2016-10, Vol.145-146, p.78-97

2016

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Molecular hydrogen in drinking water protects against neurodegenerative changes induced by traumatic brain injury

PloS one, 2014-09, Vol.9 (9), p.e108034

2014

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Regulation of TDP-43 phosphorylation in aging and disease

GeroScience, 2021-08, Vol.43 (4), p.1605-1614

2021

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Genome wide analysis reveals heparan sulfate epimerase modulates TDP-43 proteinopathy

PLoS genetics, 2019-12, Vol.15 (12), p.e1008526

2019

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Repetitive blast exposure in mice and combat veterans causes persistent cerebellar dysfunction

Science translational medicine, 2016-01, Vol.8 (321), p.321ra6

2016

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Loss of murine TDP-43 disrupts motor function and plays an essential role in embryogenesis

Acta neuropathologica, 2010-04, Vol.119 (4), p.409-419

2010

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CDC7 inhibition blocks pathological TDP-43 phosphorylation and neurodegeneration

Annals of neurology, 2013-07, Vol.74 (1), p.39-52

2013

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Synergistic toxicity between tau and amyloid drives neuronal dysfunction and neurodegeneration in transgenic C. elegans

Human molecular genetics, 2020-02, Vol.29 (3), p.495-505

2020

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Transgenic Dendra2::tau expression allows in vivo monitoring of tau proteostasis in Caenorhabditis elegans

Disease models & mechanisms, 2024-03, Vol.17 (3)

2024

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Targeting nuclear protein TDP-43 by cell division cycle kinase 7 inhibitors: A new therapeutic approach for amyotrophic lateral sclerosis

European journal of medicinal chemistry, 2021-01, Vol.210, p.112968, Article 112968

2021

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Human Ubiquilin 2 and TDP-43 copathology drives neurodegeneration in transgenic Caenorhabditis elegans

G3 : genes - genomes - genetics, 2021-08, Vol.11 (8)

2021

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Simple models to understand complex disease: 10 years of progress from Caenorhabditis elegans models of amyotrophic lateral sclerosis and frontotemporal lobar degeneration

Frontiers in neuroscience, 2024-01, Vol.17, p.1300705

2024

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Transcriptomic evaluation of tau and TDP-43 synergism shows tauopathy predominance and reveals potential modulating targets

Neurobiology of disease, 2024-04, Vol.193, p.106441-106441, Article 106441

2024

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Tau–RNA complexes inhibit microtubule polymerization and drive disease-relevant conformation change

Brain (London, England : 1878), 2023-08, Vol.146 (8), p.3206-3220

2023

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Pathological phosphorylation of tau and TDP-43 by TTBK1 and TTBK2 drives neurodegeneration

Molecular neurodegeneration, 2018-02, Vol.13 (1), p.7-7, Article 7

2018

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SPOP loss of function protects against tauopathy

Proceedings of the National Academy of Sciences - PNAS, 2023-01, Vol.120 (1), p.e2207250120

2023

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Tau tubulin kinases in proteinopathy

The FEBS journal, 2019-07, Vol.286 (13), p.2434-2446

2019

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Distinct Poly(A) nucleases have differential impact on sut-2 dependent tauopathy phenotypes

Neurobiology of disease, 2021-01, Vol.147, p.105148-105148, Article 105148

2021

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Blast exposure causes early and persistent aberrant phospho- and cleaved-tau expression in a murine model of mild blast-induced traumatic brain injury

Journal of Alzheimer's disease, 2013-01, Vol.37 (2), p.309-323

2013

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A drug repurposing screen identifies hepatitis C antivirals as inhibitors of the SARS-CoV2 main protease

Motor neuron disease-associated loss of nuclear TDP-43 is linked to DNA double-strand break repair defects

The tau tubulin kinases TTBK1/2 promote accumulation of pathological TDP-43

Protein Kinase CK‑1 Inhibitors As New Potential Drugs for Amyotrophic Lateral Sclerosis

Constitutive XBP-1s-mediated activation of the endoplasmic reticulum unfolded protein response protects against pathological tau

Phosphorylation promotes neurotoxicity in a Caenorhabditis elegans model of TDP-43 proteinopathy

TDP-43/FUS in motor neuron disease: Complexity and challenges

Molecular hydrogen in drinking water protects against neurodegenerative changes induced by traumatic brain injury

Regulation of TDP-43 phosphorylation in aging and disease

Genome wide analysis reveals heparan sulfate epimerase modulates TDP-43 proteinopathy

Repetitive blast exposure in mice and combat veterans causes persistent cerebellar dysfunction

Loss of murine TDP-43 disrupts motor function and plays an essential role in embryogenesis

CDC7 inhibition blocks pathological TDP-43 phosphorylation and neurodegeneration

Synergistic toxicity between tau and amyloid drives neuronal dysfunction and neurodegeneration in transgenic C. elegans

Transgenic Dendra2::tau expression allows in vivo monitoring of tau proteostasis in Caenorhabditis elegans

Targeting nuclear protein TDP-43 by cell division cycle kinase 7 inhibitors: A new therapeutic approach for amyotrophic lateral sclerosis

Human Ubiquilin 2 and TDP-43 copathology drives neurodegeneration in transgenic Caenorhabditis elegans

Simple models to understand complex disease: 10 years of progress from Caenorhabditis elegans models of amyotrophic lateral sclerosis and frontotemporal lobar degeneration

Transcriptomic evaluation of tau and TDP-43 synergism shows tauopathy predominance and reveals potential modulating targets

Tau–RNA complexes inhibit microtubule polymerization and drive disease-relevant conformation change

Pathological phosphorylation of tau and TDP-43 by TTBK1 and TTBK2 drives neurodegeneration

SPOP loss of function protects against tauopathy

Tau tubulin kinases in proteinopathy

Distinct Poly(A) nucleases have differential impact on sut-2 dependent tauopathy phenotypes

Blast exposure causes early and persistent aberrant phospho- and cleaved-tau expression in a murine model of mild blast-induced traumatic brain injury

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