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A novel peripheral biomarker for depression and antidepressant response

Molecular psychiatry, 2022-03, Vol.27 (3), p.1640-1646

2022

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Enterohemorrhagic E. coli alters murine intestinal epithelial tight junction protein expression and barrier function in a Shiga toxin independent manner

Laboratory investigation, 2010-08, Vol.90 (8), p.1152-1168

2010

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Enteropathogenic E. coli disrupts tight junction barrier function and structure in vivo

Laboratory investigation, 2005-10, Vol.85 (10), p.1308-1324

2005

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Probiotic Lactobacillus acidophilus Stimulates Chloride/Hydroxyl Exchange Activity in Human Intestinal Epithelial Cells

The Journal of nutrition, 2008-07, Vol.138 (7), p.1355-1359

2008

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Enteropathogenic Escherichia coli dephosphorylates and dissociates occludin from intestinal epithelial tight junctions

Cellular microbiology, 2000-08, Vol.2 (4), p.305-315

2000

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Comparative Analysis of EspF from Enteropathogenic and Enterohemorrhagic Escherichia coli in Alteration of Epithelial Barrier Function

Infection and Immunity, 2004-06, Vol.72 (6), p.3218-3227

2004

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Disruption of Cell Polarity by Enteropathogenic Escherichia coli Enables Basolateral Membrane Proteins To Migrate Apically and To Potentiate Physiological Consequences

Infection and Immunity, 2003-12, Vol.71 (12), p.7069-7078

2003

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The bacterial virulence factor NleA's involvement in intestinal tight junction disruption during Enteropathogenic E. coli infection is independent of its putative PDZ binding domain

Gut microbes, 2010-03, Vol.1 (2), p.114-118

2010

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Enteropathogenic Escherichia coli-Induced Epidermal Growth Factor Receptor Activation Contributes to Physiological Alterations in Intestinal Epithelial Cells

Infection and Immunity, 2007-05, Vol.75 (5), p.2316-2324

2007

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Enteropathogenic Escherichia coli Activates Ezrin, Which Participates in Disruption of Tight Junction Barrier Function

Infection and Immunity, 2001-09, Vol.69 (9), p.5679-5688

2001

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A Gene from the Locus of Enterocyte Effacement That Is Required for Enteropathogenic Escherichia coli To Increase Tight-Junction Permeability Encodes a Chaperone for EspF

Infection and Immunity, 2002-05, Vol.70 (5), p.2271-2277

2002

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bacterial virulence factor NleA is required for the disruption of intestinal tight junctions by enteropathogenic Escherichia coli

Cellular microbiology, 2010, Vol.12 (1), p.31-41

2010

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Activation of Microtubule Dynamics Increases Neuronal Growth via the Nerve Growth Factor (NGF)- and Gαs-mediated Signaling Pathways

The Journal of biological chemistry, 2015-04, Vol.290 (16), p.10045-10056

2015

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Clostridium difficile toxin B disrupts the barrier function of T84 monolayers

Gastroenterology (New York, N.Y. 1943), 1992-02, Vol.102 (2), p.416-423

1992

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A membrane-permeant peptide that inhibits MLC kinase restores barrier function in in vitro models of intestinal disease

Gastroenterology (New York, N.Y. 1943), 2002-07, Vol.123 (1), p.163-172

2002

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Enteropathogenic E. coli non‐LEE encoded effectors NleH1 and NleH2 attenuate NF‐κB activation

Molecular microbiology, 2010-12, Vol.78 (5), p.1232-1245

2010

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Translocated EspF protein from enteropathogenic Escherichia coli disrupts host intestinal barrier function

The Journal of clinical investigation, 2001-03, Vol.107 (5), p.621-629

2001

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EPEC ESPF Disrupts Tight Junction Barrier Function Through Direct Interaction With 14-3-3

Gastroenterology (New York, N.Y. 1943), 2011, Vol.140 (5), p.S-8-S-8

2011

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Enterohemorrhagic Escherichia coli suppresses inflammatory response to cytokines and its own toxin

American journal of physiology: Gastrointestinal and liver physiology, 2009-09, Vol.297 (3), p.G576-G581

2009

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Activation of NF-kappa B in intestinal epithelial cells by enteropathogenic Escherichia coli

American Journal of Physiology: Cell Physiology, 1997-10, Vol.273 (4), p.C1160-C1167

1997

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Infection by Bacterial Pathogens Expressing Type III Secretion Decreases Luciferase Activity: Ramifications for Reporter Gene Studies

BioTechniques, 2000-09, Vol.29 (3), p.514-522

2000

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Enteropathogenic E. coli-induced barrier function alteration is not a consequence of host cell apoptosis

American journal of physiology: Gastrointestinal and liver physiology, 2008-05, Vol.294 (5), p.G1165-G1170

2008

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EPEC-activated ERK1/2 participate in inflammatory response but not tight junction barrier disruption

American journal of physiology: Gastrointestinal and liver physiology, 2001-10, Vol.281 (4), p.G890-G898

2001

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The bacterial virulence factor NleA is required for the disruption of intestinal tight junctions by Enteropathogenic E.coli

Cellular microbiology, 2009-08, Vol.12 (1), p.31-41

2009

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Enteropathogenic E. coli attenuates secretagogue-induced net intestinal ion transport but not Cl- secretion

American journal of physiology: Gastrointestinal and liver physiology, 1999-03, Vol.276 (3), p.G781-G788

1999

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A novel peripheral biomarker for depression and antidepressant response

Enterohemorrhagic E. coli alters murine intestinal epithelial tight junction protein expression and barrier function in a Shiga toxin independent manner

Enteropathogenic E. coli disrupts tight junction barrier function and structure in vivo

Probiotic Lactobacillus acidophilus Stimulates Chloride/Hydroxyl Exchange Activity in Human Intestinal Epithelial Cells

Enteropathogenic Escherichia coli dephosphorylates and dissociates occludin from intestinal epithelial tight junctions

Comparative Analysis of EspF from Enteropathogenic and Enterohemorrhagic Escherichia coli in Alteration of Epithelial Barrier Function

Disruption of Cell Polarity by Enteropathogenic Escherichia coli Enables Basolateral Membrane Proteins To Migrate Apically and To Potentiate Physiological Consequences

The bacterial virulence factor NleA's involvement in intestinal tight junction disruption during Enteropathogenic E. coli infection is independent of its putative PDZ binding domain

Enteropathogenic Escherichia coli-Induced Epidermal Growth Factor Receptor Activation Contributes to Physiological Alterations in Intestinal Epithelial Cells

Enteropathogenic Escherichia coli Activates Ezrin, Which Participates in Disruption of Tight Junction Barrier Function

A Gene from the Locus of Enterocyte Effacement That Is Required for Enteropathogenic Escherichia coli To Increase Tight-Junction Permeability Encodes a Chaperone for EspF

bacterial virulence factor NleA is required for the disruption of intestinal tight junctions by enteropathogenic Escherichia coli

Activation of Microtubule Dynamics Increases Neuronal Growth via the Nerve Growth Factor (NGF)- and Gαs-mediated Signaling Pathways

Clostridium difficile toxin B disrupts the barrier function of T84 monolayers

A membrane-permeant peptide that inhibits MLC kinase restores barrier function in in vitro models of intestinal disease

Enteropathogenic E. coli non‐LEE encoded effectors NleH1 and NleH2 attenuate NF‐κB activation

Translocated EspF protein from enteropathogenic Escherichia coli disrupts host intestinal barrier function

EPEC ESPF Disrupts Tight Junction Barrier Function Through Direct Interaction With 14-3-3

Enterohemorrhagic Escherichia coli suppresses inflammatory response to cytokines and its own toxin

Activation of NF-kappa B in intestinal epithelial cells by enteropathogenic Escherichia coli

Infection by Bacterial Pathogens Expressing Type III Secretion Decreases Luciferase Activity: Ramifications for Reporter Gene Studies

Enteropathogenic E. coli-induced barrier function alteration is not a consequence of host cell apoptosis

EPEC-activated ERK1/2 participate in inflammatory response but not tight junction barrier disruption

The bacterial virulence factor NleA is required for the disruption of intestinal tight junctions by Enteropathogenic E.coli

Enteropathogenic E. coli attenuates secretagogue-induced net intestinal ion transport but not Cl- secretion

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