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CRISPR screens in cancer spheroids identify 3D growth-specific vulnerabilities

Nature (London), 2020-04, Vol.580 (7801), p.136-141

2020

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Antitumor activity of an engineered decoy receptor targeting CLCF1-CNTFR signaling in lung adenocarcinoma

Nature medicine, 2019-11, Vol.25 (11), p.1783-1795

2019

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STAT2/IRF9 directs a prolonged ISGF3-like transcriptional response and antiviral activity in the absence of STAT1

Biochemical journal, 2015-03, Vol.466 (3), p.511-524

2015

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UHRF1 is a mediator of KRAS driven oncogenesis in lung adenocarcinoma

Nature communications, 2023-07, Vol.14 (1), p.3966-3966, Article 3966

2023

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Assays for DNA double-strand break repair by microhomology-based end-joining repair mechanisms

Nucleic acids research, 2016-04, Vol.44 (6), p.e56

2016

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An Expanded Tool Kit for Modeling the Oncogenic Functions of KRAS

Cancer discovery, 2020-11, Vol.10 (11), p.1626-1628

2020

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The unique role of STAT2 in constitutive and IFN-induced transcription and antiviral responses

Cytokine & growth factor reviews, 2016-06, Vol.29, p.71-81

2016

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siRNA-mediated engineering of DNA recombination for improved transgene integration and expression

New biotechnology, 2014-07, Vol.31, p.S193-S193

2014

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A role for homologous recombination proteins in cell cycle regulation

Cell cycle (Georgetown, Tex.), 2015, Vol.14 (17), p.2853-2861

2015

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Abstract 1083: MEKi and SOS1i as a combination treatment option for KRAS mutant endometrial cancer

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.1083-1083

2023

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Abstract B25: Combined proteomic and genetic interaction mapping reveals new Ras pathway effectors and regulators

Molecular cancer research, 2020-05, Vol.18 (5_Supplement), p.B25-B25

2020

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Abstract PR07: Combinatorial knockout of Rap1GDS1 and RhoA leads to lethality in KRAS-driven non-small cell lung cancer

Molecular cancer research, 2020-05, Vol.18 (5_Supplement), p.PR07-PR07

2020

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Abstract 959: Proteomic and genetic interaction mapping of the Ras pathway reveals new effectors and vulnerabilities

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.959-959

2019

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Combined Proteomic and Genetic Interaction Mapping Reveals New RAS Effector Pathways and Susceptibilities

Cancer discovery, 2020-12, Vol.10 (12), p.1950-1967

2020

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The Mir181ab1 cluster promotes KRAS-driven oncogenesis and progression in lung and pancreas

The Journal of clinical investigation, 2020-04, Vol.130 (4), p.1879-1895

2020

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Abstract PR12: A combined protein-protein interaction and genetic interaction map defines new and critical Kras effectors in non-small cell lung cancer

Clinical cancer research, 2018-09, Vol.24 (17_Supplement), p.PR12-PR12

2018

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Abstract B029: Functional analysis of the role of RAP1GDS1 and RhoA in KRAS-driven lung adenocarcinoma

Molecular cancer research, 2023-05, Vol.21 (5_Supplement), p.B029-B029

2023

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Abstract 3920: Functional analysis of the role of RAP1GDS1 and RhoA in KRAS-driven lung adenocarcinoma

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.3920-3920

2023

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Abstract 4362: Identification of novel combinatorial synthetic lethal vulnerabilities in KRAS-driven lung cancer

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.4362-4362

2018

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Abstract A27: Identification of novel KRAS-synthetic lethal targets for treatment of KRAS-dependent Non-Small Cell Lung Cancer

Molecular cancer therapeutics, 2017-10, Vol.16 (10_Supplement), p.A27-A27

2017

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MAR‐Mediated transgene integration into permissive chromatin and increased expression by recombination pathway engineering

Biotechnology and bioengineering, 2017-02, Vol.114 (2), p.384-396

2017

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Anti-tumor activity of an engineered decoy receptor targeting CLCF1-CNTFR signaling in lung adenocarcinoma

Nature medicine, 2019-11, Vol.25 (11), p.1783-1795

2019

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Abstract PR03: Linking AP/MS-driven protein-protein interaction networks and combination CRISPR/sgRNA screens defines new Kras effectors and target candidates for non-small cell lung cancer

Molecular cancer therapeutics, 2017-10, Vol.16 (10_Supplement), p.PR03-PR03

2017

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High-level transgene expression by homologous recombination-mediated gene transfer

Nucleic acids research, 2011-08, Vol.39 (15), p.e104

2011

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CRISPR screens in cancer spheroids identify 3D growth-specific vulnerabilities

Antitumor activity of an engineered decoy receptor targeting CLCF1-CNTFR signaling in lung adenocarcinoma

STAT2/IRF9 directs a prolonged ISGF3-like transcriptional response and antiviral activity in the absence of STAT1

UHRF1 is a mediator of KRAS driven oncogenesis in lung adenocarcinoma

Assays for DNA double-strand break repair by microhomology-based end-joining repair mechanisms

An Expanded Tool Kit for Modeling the Oncogenic Functions of KRAS

The unique role of STAT2 in constitutive and IFN-induced transcription and antiviral responses

siRNA-mediated engineering of DNA recombination for improved transgene integration and expression

A role for homologous recombination proteins in cell cycle regulation

Abstract 1083: MEKi and SOS1i as a combination treatment option for KRAS mutant endometrial cancer

Abstract B25: Combined proteomic and genetic interaction mapping reveals new Ras pathway effectors and regulators

Abstract PR07: Combinatorial knockout of Rap1GDS1 and RhoA leads to lethality in KRAS-driven non-small cell lung cancer

Abstract 959: Proteomic and genetic interaction mapping of the Ras pathway reveals new effectors and vulnerabilities

Combined Proteomic and Genetic Interaction Mapping Reveals New RAS Effector Pathways and Susceptibilities

The Mir181ab1 cluster promotes KRAS-driven oncogenesis and progression in lung and pancreas

Abstract PR12: A combined protein-protein interaction and genetic interaction map defines new and critical Kras effectors in non-small cell lung cancer

Abstract B029: Functional analysis of the role of RAP1GDS1 and RhoA in KRAS-driven lung adenocarcinoma

Abstract 3920: Functional analysis of the role of RAP1GDS1 and RhoA in KRAS-driven lung adenocarcinoma

Abstract 4362: Identification of novel combinatorial synthetic lethal vulnerabilities in KRAS-driven lung cancer

Abstract A27: Identification of novel KRAS-synthetic lethal targets for treatment of KRAS-dependent Non-Small Cell Lung Cancer

MAR‐Mediated transgene integration into permissive chromatin and increased expression by recombination pathway engineering

Anti-tumor activity of an engineered decoy receptor targeting CLCF1-CNTFR signaling in lung adenocarcinoma

Abstract PR03: Linking AP/MS-driven protein-protein interaction networks and combination CRISPR/sgRNA screens defines new Kras effectors and target candidates for non-small cell lung cancer

High-level transgene expression by homologous recombination-mediated gene transfer

Abstract B23: Combination of SOS1::KRAS inhibitor with a MEK inhibitor reconfigures the immune tumor microenvironment of KRASG12D pancreatic ductal adenocarcinomas and sensitizes to immunotherapy

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