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The ATP-sensitive K +-channel (K ATP) controls early left–right patterning in Xenopus and chick embryos

Developmental biology, 2010-10, Vol.346 (1), p.39-53

2010

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Kir6.2 Variant E23K Increases ATP-Sensitive K+ Channel Activity and Is Associated With Impaired Insulin Release and Enhanced Insulin Sensitivity in Adults With Normal Glucose Tolerance

Diabetes (New York, N.Y.), 2009-08, Vol.58 (8), p.1869-1878

2009

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Muscle KATP channels: recent insights to energy sensing and myoprotection

Physiological reviews, 2010-07, Vol.90 (3), p.799-829

2010

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An ATP-Binding Mutation (G334D) in KCNJ11 Is Associated With a Sulfonylurea-Insensitive Form of Developmental Delay, Epilepsy, and Neonatal Diabetes

Diabetes (New York, N.Y.), 2007-02, Vol.56 (2), p.328-336

2007

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Critical role of gap junction coupled KATP channel activity for regulated insulin secretion

PLoS biology, 2006-02, Vol.4 (2), p.e26-e26

2006

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HIV Protease Inhibitors Acutely Impair Glucose-Stimulated Insulin Release

Diabetes (New York, N.Y.), 2003-07, Vol.52 (7), p.1695-1700

2003

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The G53D Mutation in Kir6.2 (KCNJ11) Is Associated with Neonatal Diabetes and Motor Dysfunction in Adulthood that Is Improved with Sulfonylurea Therapy

The journal of clinical endocrinology and metabolism, 2008-03, Vol.93 (3), p.1054-1061

2008

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Effects of the HIV Protease Inhibitor Ritonavir on GLUT4 Knock-out Mice

The Journal of biological chemistry, 2010-11, Vol.285 (47), p.36395-36400

2010

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Diet-Induced Glucose Intolerance in Mice With Decreased β-Cell ATP-Sensitive K+ Channels

Diabetes (New York, N.Y.), 2004-12, Vol.53 (12), p.3159-3167

2004

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Expression of ATP-Insensitive KATP Channels in Pancreatic β-Cells Underlies a Spectrum of Diabetic Phenotypes

Diabetes (New York, N.Y.), 2006-11, Vol.55 (11), p.2957-2964

2006

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Diabetes and insulin secretion : The ATP-sensitive K+ channel (KATP) connection

Diabetes (New York, N.Y.), 2005-11, Vol.54 (11), p.3065-3072

2005

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KATP channelopathies in the pancreas

Pflügers Archiv, 2010-07, Vol.460 (2), p.307-320

2010

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ATP and sulfonylurea sensitivity of mutant ATP-sensitive K+ channels in neonatal diabetes : Implications for pharmacogenomic therapy

Diabetes (New York, N.Y.), 2005-09, Vol.54 (9), p.2645-2654

2005

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ATP-Sensitive K+ Channel Signaling in Glucokinase-Deficient Diabetes

Diabetes (New York, N.Y.), 2005-10, Vol.54 (10), p.2925-2931

2005

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Hypotension Due to Kir6.1 Gain‐of‐Function in Vascular Smooth Muscle

Journal of the American Heart Association, 2013-08, Vol.2 (4), p.e000365-n/a

2013

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DEND mutation in Kir6.2 (KCNJ11) reveals a flexible N-terminal region critical for ATP-sensing of the KATP channel

Biophysical journal, 2008-11, Vol.95 (10), p.4689-4697

2008

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Successful sulfonylurea treatment of an insulin-naïve neonate with diabetes mellitus due to a KCNJ11 mutation

Pediatric diabetes, 2010-06, Vol.11 (4), p.286-288

2010

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Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 ( KCNJ11 ) Mutation V290M Mutation

Diabetes (New York, N.Y.), 2010-10, Vol.60 (1), p.209-217

2010

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Tolerance for ATP-Insensitive KATP Channels in Transgenic Mice

Circulation research, 2001-11, Vol.89 (11), p.1022-1029

2001

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Diabetes and Insulin Secretion

Diabetes (New York, N.Y.), 2005-11, Vol.54 (11), p.3065-3072

2005

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ATP and Sulfonylurea Sensitivity of Mutant ATP-Sensitive K+ Channels in Neonatal Diabetes

Diabetes (New York, N.Y.), 2005-09, Vol.54 (9), p.2645-2654

2005

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Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of KATP-Induced Neonatal Diabetes Mellitus

Cell metabolism, 2009-02, Vol.9 (2), p.140-151

2009

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Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of K ATP-Induced Neonatal Diabetes Mellitus

Cell metabolism, 2009-02, Vol.9 (2), p.140-151

2009

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K sub(ATP) channelopathies in the pancreas

Pflügers Archiv, 2010-07, Vol.460 (2), p.307-320

2010

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DEND Mutation in Kir6.2 ( KCNJ11) Reveals a Flexible N-Terminal Region Critical for ATP-Sensing of the K ATP Channel

Biophysical journal, 2008-11, Vol.95 (10), p.4689-4697

2008

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The ATP-sensitive K +-channel (K ATP) controls early left–right patterning in Xenopus and chick embryos

Kir6.2 Variant E23K Increases ATP-Sensitive K+ Channel Activity and Is Associated With Impaired Insulin Release and Enhanced Insulin Sensitivity in Adults With Normal Glucose Tolerance

Muscle KATP channels: recent insights to energy sensing and myoprotection

An ATP-Binding Mutation (G334D) in KCNJ11 Is Associated With a Sulfonylurea-Insensitive Form of Developmental Delay, Epilepsy, and Neonatal Diabetes

Critical role of gap junction coupled KATP channel activity for regulated insulin secretion

HIV Protease Inhibitors Acutely Impair Glucose-Stimulated Insulin Release

The G53D Mutation in Kir6.2 (KCNJ11) Is Associated with Neonatal Diabetes and Motor Dysfunction in Adulthood that Is Improved with Sulfonylurea Therapy

Effects of the HIV Protease Inhibitor Ritonavir on GLUT4 Knock-out Mice

Diet-Induced Glucose Intolerance in Mice With Decreased β-Cell ATP-Sensitive K+ Channels

Expression of ATP-Insensitive KATP Channels in Pancreatic β-Cells Underlies a Spectrum of Diabetic Phenotypes

Diabetes and insulin secretion : The ATP-sensitive K+ channel (KATP) connection

KATP channelopathies in the pancreas

ATP and sulfonylurea sensitivity of mutant ATP-sensitive K+ channels in neonatal diabetes : Implications for pharmacogenomic therapy

ATP-Sensitive K+ Channel Signaling in Glucokinase-Deficient Diabetes

Hypotension Due to Kir6.1 Gain‐of‐Function in Vascular Smooth Muscle

DEND mutation in Kir6.2 (KCNJ11) reveals a flexible N-terminal region critical for ATP-sensing of the KATP channel

Successful sulfonylurea treatment of an insulin-naïve neonate with diabetes mellitus due to a KCNJ11 mutation

Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 ( KCNJ11 ) Mutation V290M Mutation

Tolerance for ATP-Insensitive KATP Channels in Transgenic Mice

Diabetes and Insulin Secretion

ATP and Sulfonylurea Sensitivity of Mutant ATP-Sensitive K+ Channels in Neonatal Diabetes

Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of KATP-Induced Neonatal Diabetes Mellitus

Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of K ATP-Induced Neonatal Diabetes Mellitus

K sub(ATP) channelopathies in the pancreas

DEND Mutation in Kir6.2 ( KCNJ11) Reveals a Flexible N-Terminal Region Critical for ATP-Sensing of the K ATP Channel

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