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Mitochondrial complex I inhibition triggers a mitophagy-dependent ROS increase leading to necroptosis and ferroptosis in melanoma cells

Cell death & disease, 2017-03, Vol.8 (3), p.e2716-e2716

2017

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Mitochondrial disorders in children: toward development of small‐molecule treatment strategies

EMBO molecular medicine, 2016-04, Vol.8 (4), p.311-327

2016

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Targeting mitochondrial complex I using BAY 87-2243 reduces melanoma tumor growth

Cancer & metabolism, 2015-10, Vol.3 (1), p.11-11, Article 11

2015

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Interactions between mitochondrial reactive oxygen species and cellular glucose metabolism

Archives of toxicology, 2015-08, Vol.89 (8), p.1209-1226

2015

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Monogenic Mitochondrial Disorders

The New England journal of medicine, 2012-03, Vol.366 (12), p.1132-1141

2012

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Mitochondrial Morphofunction in Mammalian Cells

Antioxidants & redox signaling, 2019-06, Vol.30 (18), p.2066-2109

2019

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Mitochondrial complex I deficiency: from organelle dysfunction to clinical disease

Brain (London, England : 1878), 2009-04, Vol.132 (4), p.833-842

2009

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Depletion of PINK1 affects mitochondrial metabolism, calcium homeostasis and energy maintenance

Journal of cell science, 2011-04, Vol.124 (Pt 7), p.1115-1125

2011

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The Hypoxia Response Pathway: A Potential Intervention Target in Parkinson's Disease?

Movement disorders, 2024-02, Vol.39 (2), p.273-293

2024

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Mitoenergetic Dysfunction Triggers a Rapid Compensatory Increase in Steady-State Glucose Flux

Biophysical journal, 2015-10, Vol.109 (7), p.1372-1386

2015

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Variants in NGLY1 lead to intellectual disability, myoclonus epilepsy, sensorimotor axonal polyneuropathy and mitochondrial dysfunction

Clinical genetics, 2020-04, Vol.97 (4), p.556-566

2020

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Insights into the genome of large sulfur bacteria revealed by analysis of single filaments

PLoS biology, 2007-09, Vol.5 (9), p.e230-e230

2007

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Frapbot: An open‐source application for FRAP data

Cytometry. Part A, 2017-08, Vol.91 (8), p.810-814

2017

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Cytosolic signaling protein Ecsit also localizes to mitochondria where it interacts with chaperone NDUFAF1 and functions in complex I assembly

Genes & development, 2007-03, Vol.21 (5), p.615-624

2007

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Computer-assisted live cell analysis of mitochondrial membrane potential, morphology and calcium handling

Methods (San Diego, Calif.), 2008-12, Vol.46 (4), p.304-311

2008

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An anaerobic mitochondrion that produces hydrogen

Nature, 2005-03, Vol.434 (7029), p.74-79

2005

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Sustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fates

Nucleus (Austin, Tex.), 2015-01, Vol.6 (3), p.236-246

2015

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The antioxidant Trolox restores mitochondrial membrane potential and Ca2+-stimulated ATP production in human complex I deficiency

Journal of molecular medicine (Berlin, Germany), 2009-05, Vol.87 (5), p.515-522

2009

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Increased mitochondrial ATP production capacity in brain of healthy mice and a mouse model of isolated complex I deficiency after isoflurane anesthesia

Journal of inherited metabolic disease, 2016-01, Vol.39 (1), p.59-65

2016

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Mitochondrial processes are impaired in hereditary inclusion body myopathy

Human molecular genetics, 2008-12, Vol.17 (23), p.3663-3674

2008

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Mitochondrial dynamics in human NADH:ubiquinone oxidoreductase deficiency

The international journal of biochemistry & cell biology, 2009-10, Vol.41 (10), p.1773-1782

2009

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Modeling mitochondrial dysfunctions in the brain: from mice to men

Journal of inherited metabolic disease, 2012-03, Vol.35 (2), p.193-210

2012

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Mouse models for nuclear DNA-encoded mitochondrial complex I deficiency

Journal of inherited metabolic disease, 2011-04, Vol.34 (2), p.293-307

2011

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Restoration of complex V deficiency caused by a novel deletion in the human TMEM70 gene normalizes mitochondrial morphology

Mitochondrion, 2011-11, Vol.11 (6), p.954-963

2011

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Ca super(2+)-mobilizing agonists increase mitochondrial ATP production to accelerate cytosolic Ca super(2+) removal: aberrations in human complex I deficiency

American Journal of Physiology: Cell Physiology, 2006-08, Vol.291 (2), p.C308-C316

2006

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Mitochondrial complex I inhibition triggers a mitophagy-dependent ROS increase leading to necroptosis and ferroptosis in melanoma cells

Mitochondrial disorders in children: toward development of small‐molecule treatment strategies

Targeting mitochondrial complex I using BAY 87-2243 reduces melanoma tumor growth

Interactions between mitochondrial reactive oxygen species and cellular glucose metabolism

Monogenic Mitochondrial Disorders

Mitochondrial Morphofunction in Mammalian Cells

Mitochondrial complex I deficiency: from organelle dysfunction to clinical disease

Depletion of PINK1 affects mitochondrial metabolism, calcium homeostasis and energy maintenance

The Hypoxia Response Pathway: A Potential Intervention Target in Parkinson's Disease?

Mitoenergetic Dysfunction Triggers a Rapid Compensatory Increase in Steady-State Glucose Flux

Variants in NGLY1 lead to intellectual disability, myoclonus epilepsy, sensorimotor axonal polyneuropathy and mitochondrial dysfunction

Insights into the genome of large sulfur bacteria revealed by analysis of single filaments

Frapbot: An open‐source application for FRAP data

Cytosolic signaling protein Ecsit also localizes to mitochondria where it interacts with chaperone NDUFAF1 and functions in complex I assembly

Computer-assisted live cell analysis of mitochondrial membrane potential, morphology and calcium handling

An anaerobic mitochondrion that produces hydrogen

Sustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fates

The antioxidant Trolox restores mitochondrial membrane potential and Ca2+-stimulated ATP production in human complex I deficiency

Increased mitochondrial ATP production capacity in brain of healthy mice and a mouse model of isolated complex I deficiency after isoflurane anesthesia

Mitochondrial processes are impaired in hereditary inclusion body myopathy

Mitochondrial dynamics in human NADH:ubiquinone oxidoreductase deficiency

Modeling mitochondrial dysfunctions in the brain: from mice to men

Mouse models for nuclear DNA-encoded mitochondrial complex I deficiency

Restoration of complex V deficiency caused by a novel deletion in the human TMEM70 gene normalizes mitochondrial morphology

Ca super(2+)-mobilizing agonists increase mitochondrial ATP production to accelerate cytosolic Ca super(2+) removal: aberrations in human complex I deficiency

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