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Crucial role for autophagy in degranulation of mast cells

Journal of allergy and clinical immunology, 2011-05, Vol.127 (5), p.1267-1276.e6

2011

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The scaffold-dependent function of RIPK1 in dendritic cells promotes injury-induced colitis

Mucosal immunology, 2022-01, Vol.15 (1), p.84-95

2022

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Depletion of myeloid cells exacerbates hepatitis and induces an aberrant increase in histone H3 in mouse serum

Hepatology (Baltimore, Md.), 2017-01, Vol.65 (1), p.237-252

2017

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MIND bomb 2 prevents RIPK1 kinase activity-dependent and -independent apoptosis through ubiquitylation of cFLIPL

Communications biology, 2021-01, Vol.4 (1), p.80-80, Article 80

2021

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An antiapoptotic protein, c-FLIPL, directly binds to MKK7 and inhibits the JNK pathway

The EMBO journal, 2006-11, Vol.25 (23), p.5549-5559

2006

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Proscillaridin A Sensitizes Human Colon Cancer Cells to TRAIL-Induced Cell Death

International journal of molecular sciences, 2022-06, Vol.23 (13), p.6973

2022

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Fibroblast growth factor 18 stimulates the proliferation of hepatic stellate cells, thereby inducing liver fibrosis

Nature communications, 2023-10, Vol.14 (1), p.6304-6304, Article 6304

2023

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Lewis glycosphingolipids as critical determinants of TRAIL sensitivity in cancer cells

Oncogene, 2022-09, Vol.41 (38), p.4385-4396

2022

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Cellular FLICE-Inhibitory Protein Regulates Tissue Homeostasis

Apoptotic and Non-Apoptotic Cell Death, 2017-01, Vol.403, p.119-141

2017

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c-FLIP maintains tissue homeostasis by preventing apoptosis and programmed necrosis

Science signaling, 2012-12, Vol.5 (255), p.ra93

2012

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Generation of transgenic mice expressing a FRET biosensor, SMART, that responds to necroptosis

Communications biology, 2022-12, Vol.5 (1), p.1331-14, Article 1331

2022

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Novel method to rescue a lethal phenotype through integration of target gene onto the X-chromosome

Scientific reports, 2016-11, Vol.6 (1), p.37200-37200, Article 37200

2016

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Regenerating islet-derived protein (Reg)3β plays a crucial role in attenuation of ileitis and colitis in mice

Biochemistry and biophysics reports, 2020-03, Vol.21, p.100738-100738, Article 100738

2020

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Necroptosis of Intestinal Epithelial Cells Induces Type 3 Innate Lymphoid Cell-Dependent Lethal Ileitis

iScience, 2019-05, Vol.15, p.536-551

2019

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Fusion of OTT to BSAC Results in Aberrant Up-regulation of Transcriptional Activity

The Journal of biological chemistry, 2008-09, Vol.283 (39), p.26820-26828

2008

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Tumor Necrosis Factor α (TNFα) Induces the Unfolded Protein Response (UPR) in a Reactive Oxygen Species (ROS)-dependent Fashion, and the UPR Counteracts ROS Accumulation by TNFα

The Journal of biological chemistry, 2005-10, Vol.280 (40), p.33917-33925

2005

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A high-sensitivity ELISA for detection of human FGF18 in culture supernatants from tumor cell lines

Biochemical and biophysical research communications, 2023-10, Vol.675, p.71-77

2023

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Blockade of TNF receptor superfamily 1 (TNFR1)–dependent and TNFR1-independent cell death is crucial for normal epidermal differentiation

Journal of allergy and clinical immunology, 2019-01, Vol.143 (1), p.213-228.e10

2019

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MIND bomb 2 prevents RIPK1 kinase activity-dependent and -independent apoptosis through ubiquitylation of cFLIP L

Communications biology, 2021-01, Vol.4 (1), p.80

2021

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Genome wide analysis of TNF-inducible genes reveals that antioxidant enzymes are induced by TNF and responsible for elimination of ROS

Molecular immunology, 2004-07, Vol.41 (5), p.547-551

2004

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An antiapoptotic protein, c-FLIP L , directly binds to MKK7 and inhibits the JNK pathway

The EMBO journal, 2006-11, Vol.25 (23), p.5549-5559

2006

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Fusion of OTT to BSAC Results in Aberrant Up-regulation of Transcriptional ActivityS

The Journal of biological chemistry, 2008-09, Vol.283 (39), p.26820-26828

2008

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An antiapoptotic protein, c-FLIP sub(L), directly binds to MKK7 and inhibits the JNK pathway

The EMBO journal, 2006-01, Vol.25 (23), p.5549-5559

2006

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Tumor Necrosis Factor Î± (TNFÎ±) Induces the Unfolded Protein Response (UPR) in a Reactive Oxygen Species (ROS)-dependent Fashion, and the UPR Counteracts ROS Accumulation by TNFÎ

The Journal of biological chemistry, 2005-10, Vol.280 (40), p.33917

2005

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Tumor necrosis factor alpha (TNFalpha) induces the unfolded protein response (UPR) in a reactive oxygen species (ROS)-dependent fashion, and the UPR counteracts ROS accumulation by TNFalpha

The Journal of biological chemistry, 2005-10, Vol.280 (40), p.33917

2005

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Crucial role for autophagy in degranulation of mast cells

The scaffold-dependent function of RIPK1 in dendritic cells promotes injury-induced colitis

Depletion of myeloid cells exacerbates hepatitis and induces an aberrant increase in histone H3 in mouse serum

MIND bomb 2 prevents RIPK1 kinase activity-dependent and -independent apoptosis through ubiquitylation of cFLIPL

An antiapoptotic protein, c-FLIPL, directly binds to MKK7 and inhibits the JNK pathway

Proscillaridin A Sensitizes Human Colon Cancer Cells to TRAIL-Induced Cell Death

Fibroblast growth factor 18 stimulates the proliferation of hepatic stellate cells, thereby inducing liver fibrosis

Lewis glycosphingolipids as critical determinants of TRAIL sensitivity in cancer cells

Cellular FLICE-Inhibitory Protein Regulates Tissue Homeostasis

c-FLIP maintains tissue homeostasis by preventing apoptosis and programmed necrosis

Generation of transgenic mice expressing a FRET biosensor, SMART, that responds to necroptosis

Novel method to rescue a lethal phenotype through integration of target gene onto the X-chromosome

Regenerating islet-derived protein (Reg)3β plays a crucial role in attenuation of ileitis and colitis in mice

Necroptosis of Intestinal Epithelial Cells Induces Type 3 Innate Lymphoid Cell-Dependent Lethal Ileitis

Fusion of OTT to BSAC Results in Aberrant Up-regulation of Transcriptional Activity

Tumor Necrosis Factor α (TNFα) Induces the Unfolded Protein Response (UPR) in a Reactive Oxygen Species (ROS)-dependent Fashion, and the UPR Counteracts ROS Accumulation by TNFα

A high-sensitivity ELISA for detection of human FGF18 in culture supernatants from tumor cell lines

Blockade of TNF receptor superfamily 1 (TNFR1)–dependent and TNFR1-independent cell death is crucial for normal epidermal differentiation

MIND bomb 2 prevents RIPK1 kinase activity-dependent and -independent apoptosis through ubiquitylation of cFLIP L

Genome wide analysis of TNF-inducible genes reveals that antioxidant enzymes are induced by TNF and responsible for elimination of ROS

An antiapoptotic protein, c-FLIP L , directly binds to MKK7 and inhibits the JNK pathway

Fusion of OTT to BSAC Results in Aberrant Up-regulation of Transcriptional ActivityS

An antiapoptotic protein, c-FLIP sub(L), directly binds to MKK7 and inhibits the JNK pathway

Tumor Necrosis Factor Î± (TNFÎ±) Induces the Unfolded Protein Response (UPR) in a Reactive Oxygen Species (ROS)-dependent Fashion, and the UPR Counteracts ROS Accumulation by TNFÎ

Tumor necrosis factor alpha (TNFalpha) induces the unfolded protein response (UPR) in a reactive oxygen species (ROS)-dependent fashion, and the UPR counteracts ROS accumulation by TNFalpha

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