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Sarcolemma-localized nNOS is required to maintain activity after mild exercise

Nature, 2008-11, Vol.456 (7221), p.511-515

2008

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Like-acetylglucosaminyltransferase (LARGE)-dependent modification of dystroglycan at Thr-317/319 is required for laminin binding and arenavirus infection

Proceedings of the National Academy of Sciences - PNAS, 2011-10, Vol.108 (42), p.17426-17431

2011

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Molecular Recognition by LARGE Is Essential for Expression of Functional Dystroglycan

Cell, 2004-06, Vol.117 (7), p.953-964

2004

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PGC-1α regulates the neuromuscular junction program and ameliorates Duchenne muscular dystrophy

Genes & development, 2007-04, Vol.21 (7), p.770-783

2007

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Triadins Modulate Intracellular Ca2+ Homeostasis but Are Not Essential for Excitation-Contraction Coupling in Skeletal Muscle

The Journal of biological chemistry, 2007-12, Vol.282 (52), p.37864-37874

2007

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Organization of Ca2+ release units in excitable smooth muscle of the guinea-pig urinary bladder

Biophysical journal, 2004-09, Vol.87 (3), p.1836-1847

2004

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Endpoint measures in the mdx mouse relevant for muscular dystrophy pre-clinical studies

Neuromuscular disorders : NMD, 2012-01, Vol.22 (1), p.34-42

2012

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Triadins modulate intracellular Ca(2+) homeostasis but are not essential for excitation-contraction coupling in skeletal muscle

The Journal of biological chemistry, 2007-12, Vol.282 (52), p.37864-37874

2007

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Altered function in atrium of transgenic mice overexpressing triadin 1

American journal of physiology. Heart and circulatory physiology, 2002-10, Vol.283 (4), p.H1334-H1343

2002

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Identification of Triadin 1 as the Predominant Triadin Isoform Expressed in Mammalian Myocardium

The Journal of biological chemistry, 1999-10, Vol.274 (40), p.28660-28668

1999

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A Leucine Zipper Stabilizes the Pentameric Membrane Domain of Phospholamban and Forms a Coiled-coil Pore Structure (∗)

The Journal of biological chemistry, 1996-03, Vol.271 (10), p.5941-5946

1996

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Cardiac Hypertrophy and Impaired Relaxation in Transgenic Mice Overexpressing Triadin 1

The Journal of biological chemistry, 2001-02, Vol.276 (6), p.4142-4149

2001

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Cellular Mechanisms of Altered Contractility in the Hypertrophied Heart: Big Hearts, Big Sparks

Circulation research, 1999-03, Vol.84 (4), p.424-434

1999

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Mat-8, a Novel Phospholemman-like Protein Expressed in Human Breast Tumors, Induces a Chloride Conductance in Xenopus Oocytes (∗)

The Journal of biological chemistry, 1995-02, Vol.270 (5), p.2176-2182

1995

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Gene structure and expression of phospholemman in mouse

Gene, 2001-06, Vol.271 (1), p.69-79

2001

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Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane

The Journal of biological chemistry, 1997-09, Vol.272 (37), p.23389-23397

1997

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Localization and Characterization of the Calsequestrin-binding Domain of Triadin 1

The Journal of biological chemistry, 2000-06, Vol.275 (23), p.17639-17646

2000

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Localization and characterization of the calsequestrin-binding domain of triadin 1. Evidence for a charged beta-strand in mediating the protein-protein interaction

The Journal of biological chemistry, 2000-06, Vol.275 (23), p.17639

2000

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Complex Formation between Junctin, Triadin, Calsequestrin, and the Ryanodine Receptor

The Journal of biological chemistry, 1997-09, Vol.272 (37), p.23389-23397

1997

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PGC-1alpha regulates the neuromuscular junction program and ameliorates Duchenne muscular dystrophy

Genes & development, 2007-04, Vol.21 (7), p.770-783

2007

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Organization of Ca^sup 2+^ Release Units in Excitable Smooth Muscle of the Guinea-Pig Urinary Bladder

Biophysical journal, 2004-09, Vol.87 (3), p.1836

2004

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Skeletal Muscle Dystrophin-Glycoprotein Complex and Muscular Dystrophy

Muscle: Fundamental Biology and Mechanisms of Disease, 2012, p.935-942

2012

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Triadins Modulate Intracellular Ca super(2+) Homeostasis but Are Not Essential for Excitation-Contraction Coupling in Skeletal Muscle

The Journal of biological chemistry, 2007-12, Vol.282 (52), p.37864-37874

2007

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Organization of Ca 2+ Release Units in Excitable Smooth Muscle of the Guinea-Pig Urinary Bladder

Biophysical journal, 2004, Vol.87 (3), p.1836-1847

2004

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Sarcolemma-localized nNOS is required to maintain activity after mild exercise

Like-acetylglucosaminyltransferase (LARGE)-dependent modification of dystroglycan at Thr-317/319 is required for laminin binding and arenavirus infection

Molecular Recognition by LARGE Is Essential for Expression of Functional Dystroglycan

PGC-1α regulates the neuromuscular junction program and ameliorates Duchenne muscular dystrophy

Triadins Modulate Intracellular Ca2+ Homeostasis but Are Not Essential for Excitation-Contraction Coupling in Skeletal Muscle

Progranulin AAV gene therapy for frontotemporal dementia: translational studies and phase 1/2 trial interim results

Organization of Ca2+ release units in excitable smooth muscle of the guinea-pig urinary bladder

Endpoint measures in the mdx mouse relevant for muscular dystrophy pre-clinical studies

Triadins modulate intracellular Ca(2+) homeostasis but are not essential for excitation-contraction coupling in skeletal muscle

Altered function in atrium of transgenic mice overexpressing triadin 1

Identification of Triadin 1 as the Predominant Triadin Isoform Expressed in Mammalian Myocardium

A Leucine Zipper Stabilizes the Pentameric Membrane Domain of Phospholamban and Forms a Coiled-coil Pore Structure (∗)

Cardiac Hypertrophy and Impaired Relaxation in Transgenic Mice Overexpressing Triadin 1

Cellular Mechanisms of Altered Contractility in the Hypertrophied Heart: Big Hearts, Big Sparks

Mat-8, a Novel Phospholemman-like Protein Expressed in Human Breast Tumors, Induces a Chloride Conductance in Xenopus Oocytes (∗)

Gene structure and expression of phospholemman in mouse

Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane

Localization and Characterization of the Calsequestrin-binding Domain of Triadin 1

Localization and characterization of the calsequestrin-binding domain of triadin 1. Evidence for a charged beta-strand in mediating the protein-protein interaction

Complex Formation between Junctin, Triadin, Calsequestrin, and the Ryanodine Receptor

PGC-1alpha regulates the neuromuscular junction program and ameliorates Duchenne muscular dystrophy

Organization of Ca^sup 2+^ Release Units in Excitable Smooth Muscle of the Guinea-Pig Urinary Bladder

Skeletal Muscle Dystrophin-Glycoprotein Complex and Muscular Dystrophy

Triadins Modulate Intracellular Ca super(2+) Homeostasis but Are Not Essential for Excitation-Contraction Coupling in Skeletal Muscle

Organization of Ca 2+ Release Units in Excitable Smooth Muscle of the Guinea-Pig Urinary Bladder

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