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Therapeutic CDK4 6 inhibition in breast cancer: key mechanisms of response and failure

Oncogene, 2010-07, Vol.29 (28), p.4018-4032

2010

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Cyclin D1: polymorphism, aberrant splicing and cancer risk

Oncogene, 2006-03, Vol.25 (11), p.1620-1628

2006

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Retraction Note: Cyclin-dependent kinase 4/6 activity is a critical determinant of pre-replication complex assembly

Oncogene, 2023-04, Vol.42 (16), p.1332-1332

2023

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Cyclin D1b protein expression in breast cancer is independent of cyclin D1a and associated with poor disease outcome

Oncogene, 2009-04, Vol.28 (15), p.1812-1820

2009

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The retinoblastoma tumor suppressor pathway modulates the invasiveness of ErbB2-positive breast cancer

Oncogene, 2014-07, Vol.33 (30), p.3980-3991

2014

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Coordinate regulation of Fanconi anemia gene expression occurs through the Rb E2F pathway

Oncogene, 2008-08, Vol.27 (35), p.4798-4808

2008

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Therapeutically activating RB: reestablishing cell cycle control in endocrine therapy-resistant breast cancer

Endocrine-related cancer, 2011-06, Vol.18 (3), p.333-345

2011

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Loss of the retinoblastoma tumor suppressor: differential action on transcriptional programs related to cell cycle control and immune function

Oncogene, 2007-09, Vol.26 (43), p.6307-6318

2007

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RB has a critical role in mediating the in vivo checkpoint response, mitigating secondary DNA damage and suppressing liver tumorigenesis initiated by aflatoxin B1

Oncogene, 2009-12, Vol.28 (50), p.4434-4443

2009

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Expression of cyclin D1a and D1b as predictive factors for treatment response in colorectal cancer

British journal of cancer, 2012-11, Vol.107 (10), p.1684-1691

2012

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Cyclin-dependent kinase 4 6 activity is a critical determinant of pre-replication complex assembly

Oncogene, 2008-11, Vol.27 (56), p.7083-7093

2008

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Modeling the effect of the RB tumor suppressor on disease progression: dependence on oncogene network and cellular context

Oncogene, 2010-01, Vol.29 (1), p.68-80

2010

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RB-Dependent S-Phase Response to DNA Damage

Molecular and Cellular Biology, 2000-10, Vol.20 (20), p.7751-7763

2000

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Dual mechanisms for the inhibition of E2F binding to RB by cyclin-dependent kinase-mediated RB phosphorylation

Molecular and Cellular Biology, 1997-10, Vol.17 (10), p.5771-5783

1997

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The retinoblastoma tumor suppressor inhibits cellular proliferation through two distinct mechanisms: inhibition of cell cycle progression and induction of cell death

Oncogene, 1999-09, Vol.18 (37), p.5239-5245

1999

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Restoration of retinoblastoma mediated signaling to Cdk2 results in cell cycle arrest

Oncogene, 2000-04, Vol.19 (15), p.1857-1867

2000

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Nucleophosmin/B23 Is a Target of CDK2/Cyclin E in Centrosome Duplication

Cell, 2000-09, Vol.103 (1), p.127-140

2000

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The Aryl Hydrocarbon Receptor Displaces p300 from E2F-dependent Promoters and Represses S Phase-specific Gene Expression

The Journal of biological chemistry, 2004-07, Vol.279 (28), p.29013-29022

2004

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Cyclin D1 splice variants. Differential effects on localization, RB phosphorylation, and cellular transformation

The Journal of biological chemistry, 2003-08, Vol.278 (32), p.30339-30347

2003

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BRG-1 Is Required for RB-Mediated Cell Cycle Arrest

Proceedings of the National Academy of Sciences - PNAS, 2000-07, Vol.97 (14), p.7748-7753

2000

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Aromatic Hydrocarbon Receptor Interaction with the Retinoblastoma Protein Potentiates Repression of E2F-dependent Transcription and Cell Cycle Arrest

The Journal of biological chemistry, 2000-01, Vol.275 (4), p.2943-2950

2000

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RB signaling prevents replication-dependent DNA double-strand breaks following genotoxic insult

Nucleic acids research, 2004-01, Vol.32 (1), p.25-34

2004

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Differential Regulation of Retinoblastoma Protein Function by Specific Cdk Phosphorylation Sites

The Journal of biological chemistry, 1996-04, Vol.271 (14), p.8313-8320

1996

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Histone Deacetylation of RB-Responsive Promoters: Requisite for Specific Gene Repression but Dispensable for Cell Cycle Inhibition

Molecular and Cellular Biology, 2003-11, Vol.23 (21), p.7719-7731

2003

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Unbiased analysis of RB-mediated transcriptional repression identifies novel targets and distinctions from E2F action

Cancer research (Chicago, Ill.), 2002-11, Vol.62 (22), p.6587-6597

2002

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Therapeutic CDK4 6 inhibition in breast cancer: key mechanisms of response and failure

Cyclin D1: polymorphism, aberrant splicing and cancer risk

Retraction Note: Cyclin-dependent kinase 4/6 activity is a critical determinant of pre-replication complex assembly

Cyclin D1b protein expression in breast cancer is independent of cyclin D1a and associated with poor disease outcome

The retinoblastoma tumor suppressor pathway modulates the invasiveness of ErbB2-positive breast cancer

Coordinate regulation of Fanconi anemia gene expression occurs through the Rb E2F pathway

Therapeutically activating RB: reestablishing cell cycle control in endocrine therapy-resistant breast cancer

Loss of the retinoblastoma tumor suppressor: differential action on transcriptional programs related to cell cycle control and immune function

RB has a critical role in mediating the in vivo checkpoint response, mitigating secondary DNA damage and suppressing liver tumorigenesis initiated by aflatoxin B1

Expression of cyclin D1a and D1b as predictive factors for treatment response in colorectal cancer

Cyclin-dependent kinase 4 6 activity is a critical determinant of pre-replication complex assembly

Modeling the effect of the RB tumor suppressor on disease progression: dependence on oncogene network and cellular context

RB-Dependent S-Phase Response to DNA Damage

Dual mechanisms for the inhibition of E2F binding to RB by cyclin-dependent kinase-mediated RB phosphorylation

The retinoblastoma tumor suppressor inhibits cellular proliferation through two distinct mechanisms: inhibition of cell cycle progression and induction of cell death

Restoration of retinoblastoma mediated signaling to Cdk2 results in cell cycle arrest

Nucleophosmin/B23 Is a Target of CDK2/Cyclin E in Centrosome Duplication

The Aryl Hydrocarbon Receptor Displaces p300 from E2F-dependent Promoters and Represses S Phase-specific Gene Expression

Cyclin D1 splice variants. Differential effects on localization, RB phosphorylation, and cellular transformation

BRG-1 Is Required for RB-Mediated Cell Cycle Arrest

Aromatic Hydrocarbon Receptor Interaction with the Retinoblastoma Protein Potentiates Repression of E2F-dependent Transcription and Cell Cycle Arrest

RB signaling prevents replication-dependent DNA double-strand breaks following genotoxic insult

Differential Regulation of Retinoblastoma Protein Function by Specific Cdk Phosphorylation Sites

Histone Deacetylation of RB-Responsive Promoters: Requisite for Specific Gene Repression but Dispensable for Cell Cycle Inhibition

Unbiased analysis of RB-mediated transcriptional repression identifies novel targets and distinctions from E2F action

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