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WNT signaling in bone homeostasis and disease: from human mutations to treatments

Nature medicine, 2013-02, Vol.19 (2), p.179-192

2013

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Parathyroid hormone (PTH)–induced bone gain is blunted in SOST overexpressing and deficient mice

Journal of bone and mineral research, 2010-02, Vol.25 (2), p.178-189

2010

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Osteocyte Wnt/β-Catenin Signaling Is Required for Normal Bone Homeostasis

Molecular and Cellular Biology, 2010-06, Vol.30 (12), p.3071-3085

2010

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Disruption of Lrp4 function by genetic deletion or pharmacological blockade increases bone mass and serum sclerostin levels

Proceedings of the National Academy of Sciences - PNAS, 2014-12, Vol.111 (48), p.E5187-E5195

2014

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Anti‐Sclerostin Antibody Treatment in a Rat Model of Progressive Renal Osteodystrophy

Journal of bone and mineral research, 2015-03, Vol.30 (3), p.499-509

2015

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Cortical bone adaptation to a moderate level of mechanical loading in male Sost deficient mice

Scientific reports, 2020-12, Vol.10 (1), p.22299-22299, Article 22299

2020

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Mechanical Load Increases in Bone Formation via a Sclerostin‐Independent Pathway

Journal of bone and mineral research, 2014-11, Vol.29 (11), p.2456-2467

2014

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Sclerostin inhibition promotes TNF-dependent inflammatory joint destruction

Science translational medicine, 2016-03, Vol.8 (330), p.330ra35

2016

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Bone Overgrowth-associated Mutations in the LRP4 Gene Impair Sclerostin Facilitator Function

The Journal of biological chemistry, 2011-06, Vol.286 (22), p.19489-19500

2011

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Skeletal muscle in MuRF1 null mice is not spared in low-gravity conditions, indicating atrophy proceeds by unique mechanisms in space

Scientific reports, 2019-06, Vol.9 (1), p.9397-11, Article 9397

2019

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Mice Lacking Histone Deacetylase 6 Have Hyperacetylated Tubulin but Are Viable and Develop Normally

Molecular and Cellular Biology, 2008-03, Vol.28 (5), p.1688-1701

2008

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Pharmacological inhibition of fibroblast growth factor (FGF) receptor signaling ameliorates FGF23‐mediated hypophosphatemic rickets

Journal of bone and mineral research, 2013-04, Vol.28 (4), p.899-911

2013

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Effects of Long-Term Sclerostin Deficiency on Trabecular Bone Mass and Adaption to Limb Loading Differ in Male and Female Mice

Calcified tissue international, 2020-04, Vol.106 (4), p.415-430

2020

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Control of the SOST Bone Enhancer by PTH Using MEF2 Transcription Factors

Journal of bone and mineral research, 2007-12, Vol.22 (12), p.1957-1967

2007

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Mef2c deletion in osteocytes results in increased bone mass

Journal of bone and mineral research, 2012-02, Vol.27 (2), p.360-373

2012

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Genomic deletion of a long-range bone enhancer misregulates sclerostin in Van Buchem disease

Genome research, 2005-07, Vol.15 (7), p.928-935

2005

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Reversing LRP5‐Dependent Osteoporosis and SOST Deficiency–Induced Sclerosing Bone Disorders by Altering WNT Signaling Activity

Journal of bone and mineral research, 2014-01, Vol.29 (1), p.29-42

2014

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Sclerostin Deficiency Is Linked to Altered Bone Composition

Journal of bone and mineral research, 2014-10, Vol.29 (10), p.2144-2151

2014

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Isolation of mouse osteocytes using cell fractionation for gene expression analysis

Methods in molecular biology (Clifton, N.J.), 2012-01, Vol.816, p.55

2012

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Silencing of parathyroid hormone (PTH) receptor 1 in T cells blunts the bone anabolic activity of PTH

Proceedings of the National Academy of Sciences - PNAS, 2012-03, Vol.109 (12), p.E725-E733

2012

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Sost deficiency leads to reduced mechanical strains at the tibia midshaft in strain-matched in vivo loading experiments in mice

Journal of the Royal Society interface, 2018-04, Vol.15 (141), p.20180012

2018

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High Bone Resorption in Adult Aging Transgenic Mice Overexpressing Cbfa1/Runx2 in Cells of the Osteoblastic Lineage

Molecular and Cellular Biology, 2002-09, Vol.22 (17), p.6222-6233

2002

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Dental and periodontal phenotype in sclerostin knockout mice

International journal of oral science, 2014-04, Vol.6 (2), p.70-76

2014

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Inhibiting WNT secretion reduces high bone mass caused by Sost loss-of-function or gain-of-function mutations in Lrp5

Bone Research, 2023-08, Vol.11 (1), p.47-47, Article 47

2023

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WNT signaling in bone homeostasis and disease: from human mutations to treatments

Parathyroid hormone (PTH)–induced bone gain is blunted in SOST overexpressing and deficient mice

Osteocyte Wnt/β-Catenin Signaling Is Required for Normal Bone Homeostasis

Inhibiting the osteocyte-specific protein sclerostin increases bone mass and fracture resistance in multiple myeloma

Disruption of Lrp4 function by genetic deletion or pharmacological blockade increases bone mass and serum sclerostin levels

Anti‐Sclerostin Antibody Treatment in a Rat Model of Progressive Renal Osteodystrophy

Cortical bone adaptation to a moderate level of mechanical loading in male Sost deficient mice

Mechanical Load Increases in Bone Formation via a Sclerostin‐Independent Pathway

Sclerostin inhibition promotes TNF-dependent inflammatory joint destruction

Bone Overgrowth-associated Mutations in the LRP4 Gene Impair Sclerostin Facilitator Function

Skeletal muscle in MuRF1 null mice is not spared in low-gravity conditions, indicating atrophy proceeds by unique mechanisms in space

Mice Lacking Histone Deacetylase 6 Have Hyperacetylated Tubulin but Are Viable and Develop Normally

Pharmacological inhibition of fibroblast growth factor (FGF) receptor signaling ameliorates FGF23‐mediated hypophosphatemic rickets

Effects of Long-Term Sclerostin Deficiency on Trabecular Bone Mass and Adaption to Limb Loading Differ in Male and Female Mice

Control of the SOST Bone Enhancer by PTH Using MEF2 Transcription Factors

Mef2c deletion in osteocytes results in increased bone mass

Genomic deletion of a long-range bone enhancer misregulates sclerostin in Van Buchem disease

Reversing LRP5‐Dependent Osteoporosis and SOST Deficiency–Induced Sclerosing Bone Disorders by Altering WNT Signaling Activity

Sclerostin Deficiency Is Linked to Altered Bone Composition

Isolation of mouse osteocytes using cell fractionation for gene expression analysis

Silencing of parathyroid hormone (PTH) receptor 1 in T cells blunts the bone anabolic activity of PTH

Sost deficiency leads to reduced mechanical strains at the tibia midshaft in strain-matched in vivo loading experiments in mice

High Bone Resorption in Adult Aging Transgenic Mice Overexpressing Cbfa1/Runx2 in Cells of the Osteoblastic Lineage

Dental and periodontal phenotype in sclerostin knockout mice

Inhibiting WNT secretion reduces high bone mass caused by Sost loss-of-function or gain-of-function mutations in Lrp5

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