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Raising the ‘Good’ Oxidants for Immune Protection

Frontiers in immunology, 2021-06, Vol.12, p.698042-698042

2021

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Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair

Redox biology, 2015-08, Vol.5, p.275-289

2015

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Defensive Mutualism Rescues NADPH Oxidase Inactivation in Gut Infection

Cell host & microbe, 2016-05, Vol.19 (5), p.651-663

2016

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Pharmacological characterization of the seven human NOX isoforms and their inhibitors

Redox biology, 2019-09, Vol.26, p.101272-101272, Article 101272

2019

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Isoform-selective NADPH oxidase inhibitor panel for pharmacological target validation

Free radical biology & medicine, 2020-02, Vol.148, p.60-69

2020

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Hydrogen peroxide production by lactobacilli promotes epithelial restitution during colitis

Redox biology, 2018-06, Vol.16, p.11-20

2018

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Oxidants in Physiological Processes

Handbook of experimental pharmacology, 2021, Vol.264, p.27-47

2021

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NADPH oxidases and ROS signaling in the gastrointestinal tract

Mucosal immunology, 2018-07, Vol.11 (4), p.1011-1023

2018

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Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases

Cellular signalling, 2006, Vol.18 (1), p.69-82

2006

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House dust mite‐treated PAR2 over‐expressor mouse: A novel model of atopic dermatitis

Experimental dermatology, 2019-11, Vol.28 (11), p.1298-1308

2019

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Silencing of DUOX NADPH Oxidases by Promoter Hypermethylation in Lung Cancer

Cancer research (Chicago, Ill.), 2008-02, Vol.68 (4), p.1037-1045

2008

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Mucosal Reactive Oxygen Species Decrease Virulence by Disrupting Campylobacter jejuni Phosphotyrosine Signaling

Cell host & microbe, 2012-07, Vol.12 (1), p.47-59

2012

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VEO-IBD NOX1 variant highlights a structural region essential for NOX/DUOX catalytic activity

Redox biology, 2023-11, Vol.67, p.102905-102905, Article 102905

2023

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RhoA determines disease progression by controlling neutrophil motility and restricting hyperresponsiveness

Blood, 2014-06, Vol.123 (23), p.3635-3645

2014

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Structural Insights into Nox4 and Nox2: Motifs Involved in Function and Cellular Localization

Molecular and Cellular Biology, 2010-02, Vol.30 (4), p.961-975

2010

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Mouse Macrophages Completely Lacking Rho Subfamily GTPases (RhoA, RhoB, and RhoC) Have Severe Lamellipodial Retraction Defects, but Robust Chemotactic Navigation and Altered Motility

The Journal of biological chemistry, 2014-10, Vol.289 (44), p.30772-30784

2014

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Case report: Neonatal-onset inflammatory bowel disease due to novel compound heterozygous mutations in DUOX2

Frontiers in genetics, 2023-11, Vol.14, p.1276697

2023

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Ubiquitin C-terminal hydrolase-L1 increases cancer cell invasion by modulating hydrogen peroxide generated via NADPH oxidase 4

Oncotarget, 2015-06, Vol.6 (18), p.16287-16303

2015

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Roles of the Rac1 and Rac3 GTPases in human tumor cell invasion

Oncogene, 2005-11, Vol.24 (53), p.7821-7829

2005

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Heterodimerization controls localization of Duox-DuoxA NADPH oxidases in airway cells

Journal of cell science, 2009-04, Vol.122 (8), p.1238-1247

2009

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NADPH oxidase 4 is protective and not fibrogenic in intestinal inflammation

Redox biology, 2020-10, Vol.37, p.101752-101752, Article 101752

2020

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Defects in Nicotinamide-adenine Dinucleotide Phosphate Oxidase Genes NOX1 and DUOX2 in Very Early Onset Inflammatory Bowel Disease

Cellular and molecular gastroenterology and hepatology, 2015-09, Vol.1 (5), p.489-502

2015

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Impaired function of the Tie-2 receptor contributes to vascular leakage and lethality in anthrax

Proceedings of the National Academy of Sciences - PNAS, 2012-06, Vol.109 (25), p.10024-10029

2012

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NAPDH Oxidases in Inflammatory Bowel Disease

Methods in molecular biology (Clifton, N.J.), 2019, Vol.1982, p.695

2019

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Lung epithelial injury by B. anthracis lethal toxin is caused by MKK-dependent loss of cytoskeletal integrity

PloS one, 2009-03, Vol.4 (3), p.e4755-e4755

2009

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Raising the ‘Good’ Oxidants for Immune Protection

Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair

Defensive Mutualism Rescues NADPH Oxidase Inactivation in Gut Infection

Pharmacological characterization of the seven human NOX isoforms and their inhibitors

Isoform-selective NADPH oxidase inhibitor panel for pharmacological target validation

Hydrogen peroxide production by lactobacilli promotes epithelial restitution during colitis

Oxidants in Physiological Processes

NADPH oxidases and ROS signaling in the gastrointestinal tract

Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases

House dust mite‐treated PAR2 over‐expressor mouse: A novel model of atopic dermatitis

Silencing of DUOX NADPH Oxidases by Promoter Hypermethylation in Lung Cancer

Mucosal Reactive Oxygen Species Decrease Virulence by Disrupting Campylobacter jejuni Phosphotyrosine Signaling

VEO-IBD NOX1 variant highlights a structural region essential for NOX/DUOX catalytic activity

RhoA determines disease progression by controlling neutrophil motility and restricting hyperresponsiveness

Structural Insights into Nox4 and Nox2: Motifs Involved in Function and Cellular Localization

Mouse Macrophages Completely Lacking Rho Subfamily GTPases (RhoA, RhoB, and RhoC) Have Severe Lamellipodial Retraction Defects, but Robust Chemotactic Navigation and Altered Motility

Case report: Neonatal-onset inflammatory bowel disease due to novel compound heterozygous mutations in DUOX2

Ubiquitin C-terminal hydrolase-L1 increases cancer cell invasion by modulating hydrogen peroxide generated via NADPH oxidase 4

Roles of the Rac1 and Rac3 GTPases in human tumor cell invasion

Heterodimerization controls localization of Duox-DuoxA NADPH oxidases in airway cells

NADPH oxidase 4 is protective and not fibrogenic in intestinal inflammation

Defects in Nicotinamide-adenine Dinucleotide Phosphate Oxidase Genes NOX1 and DUOX2 in Very Early Onset Inflammatory Bowel Disease

Impaired function of the Tie-2 receptor contributes to vascular leakage and lethality in anthrax

NAPDH Oxidases in Inflammatory Bowel Disease

Lung epithelial injury by B. anthracis lethal toxin is caused by MKK-dependent loss of cytoskeletal integrity

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