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Mitochondrial spare respiratory capacity: Mechanisms, regulation, and significance in non‐transformed and cancer cells

The FASEB journal, 2020-10, Vol.34 (10), p.13106-13124

2020

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Implication of Rac1 GTPase in molecular and cellular mitochondrial functions

Life sciences (1973), 2024-04, Vol.342, p.122510-122510, Article 122510

2024

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Lactate Inhibits the Pro-Inflammatory Response and Metabolic Reprogramming in Murine Macrophages in a GPR81-Independent Manner

PloS one, 2016-11, Vol.11 (11), p.e0163694

2016

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TRPC3 shapes the ER-mitochondria Ca2+ transfer characterizing tumour-promoting senescence

Nature communications, 2022-02, Vol.13 (1), p.956-956, Article 956

2022

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Lipid Metabolism and Resistance to Anticancer Treatment

Biology (Basel, Switzerland), 2020-12, Vol.9 (12), p.474

2020

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Farnesoid X receptor inhibits glucagon-like peptide-1 production by enteroendocrine L cells

Nature communications, 2015-07, Vol.6 (1), p.7629-7629, Article 7629

2015

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Comparison of the in vivo genotoxicity of electronic and conventional cigarettes aerosols after subacute, subchronic and chronic exposures

Journal of hazardous materials, 2022-02, Vol.423 (Pt B), p.127246-127246, Article 127246

2022

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The troglitazone derivative EP13 disrupts energy metabolism through respiratory chain complex I inhibition in breast cancer cells and potentiates the antiproliferative effect of glycolysis inhibitntriors

Cancer cell international, 2024-04, Vol.24 (1), p.132-132

2024

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Glucose metabolism and NRF2 coordinate the antioxidant response in melanoma resistant to MAPK inhibitors

Cell death & disease, 2018-02, Vol.9 (3), p.325-14, Article 325

2018

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Mitochondrial electron transport is the cellular target of the oncology drug elesclomol

PloS one, 2012-01, Vol.7 (1), p.e29798-e29798

2012

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p65/RelA NF‐κB fragments generated by RIPK3 activity regulate tumorigenicity, cell metabolism, and stemness characteristics

Journal of cellular biochemistry, 2022-03, Vol.123 (3), p.543-556

2022

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A New Strategy to Preserve and Assess Oxygen Consumption in Murine Tissues

International journal of molecular sciences, 2021-12, Vol.23 (1), p.109

2021

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Metabolic features of melanoma: a gold mine of new therapeutic targets?

Current cancer drug targets, 2014-01, Vol.14 (4), p.357

2014

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Mitochondrial inhibitors circumvent adaptive resistance to venetoclax and cytarabine combination therapy in acute myeloid leukemia

Nature cancer, 2021-11, Vol.2 (11), p.1204

2021

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Clinically Relevant Oxygraphic Assay to Assess Mitochondrial Energy Metabolism in Acute Myeloid Leukemia Patients

Cancers, 2021-12, Vol.13 (24), p.6353

2021

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Integration of Mitochondrial Targeting for Molecular Cancer Therapeutics

International Journal of Cell Biology, 2015, Vol.2015, p.283145-17

2015

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Antimetabolic cooperativity with the clinically approved l-asparaginase and tyrosine kinase inhibitors to eradicate CML stem cells

Molecular metabolism (Germany), 2022-01, Vol.55, p.101410-101410, Article 101410

2022

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Cancer cell mitochondria are direct proapoptotic targets for the marine antitumor drug lamellarin D

Cancer research (Chicago, Ill.), 2006-03, Vol.66 (6), p.3177-3187

2006

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Mitochondrial adaptation decreases drug sensitivity of persistent triple negative breast cancer cells surviving combinatory and sequential chemotherapy

Neoplasia (New York, N.Y.), 2023-12, Vol.46, p.100949-100949, Article 100949

2023

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NOD2 in monocytes negatively regulates macrophage development through TNFalpha

Frontiers in immunology, 2023-06, Vol.14, p.1181823-1181823

2023

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Inhibition of DNA binding of the NF-Y transcription factor by the pyrrolobenzodiazepine-polyamide conjugate GWL-78

Molecular cancer therapeutics, 2008-05, Vol.7 (5), p.1319-1328

2008

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Melanoma metabolism contributes to the cellular responses to MAPK/ERK pathway inhibitors

Biochimica et biophysica acta. General subjects, 2018-04, Vol.1862 (4), p.999-1005

2018

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Inactivation of the HIF-1α/PDK3 Signaling Axis Drives Melanoma toward Mitochondrial Oxidative Metabolism and Potentiates the Therapeutic Activity of Pro-Oxidants

Cancer research (Chicago, Ill.), 2012-10, Vol.72 (19), p.5035-5047

2012

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Ventricular myocyte caspases are directly responsible for endotoxin-induced cardiac dysfunction

Circulation (New York, N.Y.), 2005-05, Vol.111 (20), p.2596-2604

2005

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Mitochondrial spare respiratory capacity: Mechanisms, regulation, and significance in non‐transformed and cancer cells

Implication of Rac1 GTPase in molecular and cellular mitochondrial functions

Lactate Inhibits the Pro-Inflammatory Response and Metabolic Reprogramming in Murine Macrophages in a GPR81-Independent Manner

TRPC3 shapes the ER-mitochondria Ca2+ transfer characterizing tumour-promoting senescence

Lipid Metabolism and Resistance to Anticancer Treatment

Farnesoid X receptor inhibits glucagon-like peptide-1 production by enteroendocrine L cells

Comparison of the in vivo genotoxicity of electronic and conventional cigarettes aerosols after subacute, subchronic and chronic exposures

The troglitazone derivative EP13 disrupts energy metabolism through respiratory chain complex I inhibition in breast cancer cells and potentiates the antiproliferative effect of glycolysis inhibitntriors

Glucose metabolism and NRF2 coordinate the antioxidant response in melanoma resistant to MAPK inhibitors

Mitochondrial electron transport is the cellular target of the oncology drug elesclomol

p65/RelA NF‐κB fragments generated by RIPK3 activity regulate tumorigenicity, cell metabolism, and stemness characteristics

A New Strategy to Preserve and Assess Oxygen Consumption in Murine Tissues

Metabolic features of melanoma: a gold mine of new therapeutic targets?

Mitochondrial inhibitors circumvent adaptive resistance to venetoclax and cytarabine combination therapy in acute myeloid leukemia

Targeting chelatable iron as a therapeutic modality in Parkinson's disease

Clinically Relevant Oxygraphic Assay to Assess Mitochondrial Energy Metabolism in Acute Myeloid Leukemia Patients

Integration of Mitochondrial Targeting for Molecular Cancer Therapeutics

Antimetabolic cooperativity with the clinically approved l-asparaginase and tyrosine kinase inhibitors to eradicate CML stem cells

Cancer cell mitochondria are direct proapoptotic targets for the marine antitumor drug lamellarin D

Mitochondrial adaptation decreases drug sensitivity of persistent triple negative breast cancer cells surviving combinatory and sequential chemotherapy

NOD2 in monocytes negatively regulates macrophage development through TNFalpha

Inhibition of DNA binding of the NF-Y transcription factor by the pyrrolobenzodiazepine-polyamide conjugate GWL-78

Melanoma metabolism contributes to the cellular responses to MAPK/ERK pathway inhibitors

Inactivation of the HIF-1α/PDK3 Signaling Axis Drives Melanoma toward Mitochondrial Oxidative Metabolism and Potentiates the Therapeutic Activity of Pro-Oxidants

Ventricular myocyte caspases are directly responsible for endotoxin-induced cardiac dysfunction

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