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A mouse model of myosin binding protein C human familial hypertrophic cardiomyopathy

The Journal of clinical investigation, 1998-10, Vol.102 (7), p.1292-1300

1998

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GDF15/MIC-1 Functions As a Protective and Antihypertrophic Factor Released From the Myocardium in Association With SMAD Protein Activation

Circulation research, 2006-02, Vol.98 (3), p.342-350

2006

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A treadmill exercise regimen for identifying cardiovascular phenotypes in transgenic mice

American journal of physiology. Heart and circulatory physiology, 1997-09, Vol.273 (3), p.H1595-H1605

1997

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Cardiac Myosin-Binding Protein-C Phosphorylation and Cardiac Function

Circulation research, 2005-11, Vol.97 (11), p.1156-1163

2005

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PKC-α regulates cardiac contractility and propensity toward heart failure

Nature medicine, 2004-03, Vol.10 (3), p.248-254

2004

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Reversal of Amyloid-Induced Heart Disease in Desmin-Related Cardiomyopathy

Proceedings of the National Academy of Sciences - PNAS, 2005-09, Vol.102 (38), p.13592-13597

2005

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Genetic Inhibition or Activation of JNK1/2 Protects the Myocardium from Ischemia-Reperfusion-induced Cell Death in Vivo

The Journal of biological chemistry, 2005-09, Vol.280 (38), p.32602-32608

2005

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The MEK1-ERK1/2 signaling pathway promotes compensated cardiac hypertrophy in transgenic mice

The EMBO journal, 2000-12, Vol.19 (23), p.6341-6350

2000

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Expression of R120G-αB-Crystallin Causes Aberrant Desmin and αB-Crystallin Aggregation and Cardiomyopathy in Mice

Circulation research, 2001-07, Vol.89 (1), p.84-91

2001

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αB-Crystallin Modulates Protein Aggregation of Abnormal Desmin

Circulation research, 2003-11, Vol.93 (10), p.998-1005

2003

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Cardiac-specific overexpression of fibroblast growth factor-2 protects against myocardial dysfunction and infarction in a murine model of low-flow ischemia

Circulation (New York, N.Y.), 2003-12, Vol.108 (25), p.3140-3148

2003

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Transgenic overexpression of cardiac actin in the mouse heart suggests coregulation of cardiac, skeletal and vascular actin expression

Transgenic research, 2004-12, Vol.13 (6), p.531-540

2004

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Forced expression of α-myosin heavy chain in the rabbit ventricle results in cardioprotection under cardiomyopathic conditions

Circulation (New York, N.Y.), 2005-05, Vol.111 (18), p.2339-2346

2005

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Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy

Circulation (New York, N.Y.), 2005-05, Vol.111 (18), p.2330-2338

2005

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Analysis of Myosin Heavy Chain Functionality in the Heart

The Journal of biological chemistry, 2003-05, Vol.278 (19), p.17466-17474

2003

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Transgenic Modeling of a Cardiac Troponin I Mutation Linked to Familial Hypertrophic Cardiomyopathy

Circulation research, 2000-10, Vol.87 (9), p.805-811

2000

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Altered focal adhesion regulation correlates with cardiomyopathy in mice expressing constitutively active rac1

The Journal of clinical investigation, 2000-04, Vol.105 (7), p.875-886

2000

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Role of the acidic N' region of cardiac troponin I in regulating myocardial function

The FASEB journal, 2008-04, Vol.22 (4), p.1246-1257

2008

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Calcineurin A β Gene Targeting Predisposes the Myocardium to Acute Ischemia-Induced Apoptosis and Dysfunction

Circulation research, 2004-01, Vol.94 (1), p.91-99

2004

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A mouse model of myosin binding protein C human familial hypertrophic cardiomyopathy

The Journal of clinical investigation, 1998-10, Vol.102 (7), p.1292-1300

1998

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Myofibril degeneration caused by tropomodulin overexpression leads to dilated cardiomyopathy in juvenile mice

The Journal of clinical investigation, 1998-01, Vol.101 (1), p.51-61

1998

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Disruption of Rho signaling results in progressive atrioventricular conduction defects while ventricular function remains preserved

The FASEB journal, 2004-05, Vol.18 (7), p.857-859

2004

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In Vivo Modeling of Myosin Binding Protein C Familial Hypertrophic Cardiomyopathy

Circulation research, 1999-10, Vol.85 (9), p.841-847

1999

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Proinflammatory consequences of transgenic fas ligand expression in the heart

The Journal of clinical investigation, 2000-05, Vol.105 (9), p.1199-1208

2000

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Charged residue changes in the carboxy‐terminus of α‐tropomyosin alter mouse cardiac muscle contractility

The Journal of physiology, 2004-04, Vol.556 (2), p.531-543

2004

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A mouse model of myosin binding protein C human familial hypertrophic cardiomyopathy

GDF15/MIC-1 Functions As a Protective and Antihypertrophic Factor Released From the Myocardium in Association With SMAD Protein Activation

A treadmill exercise regimen for identifying cardiovascular phenotypes in transgenic mice

Cardiac Myosin-Binding Protein-C Phosphorylation and Cardiac Function

PKC-α regulates cardiac contractility and propensity toward heart failure

Reversal of Amyloid-Induced Heart Disease in Desmin-Related Cardiomyopathy

Genetic Inhibition or Activation of JNK1/2 Protects the Myocardium from Ischemia-Reperfusion-induced Cell Death in Vivo

The MEK1-ERK1/2 signaling pathway promotes compensated cardiac hypertrophy in transgenic mice

Expression of R120G-αB-Crystallin Causes Aberrant Desmin and αB-Crystallin Aggregation and Cardiomyopathy in Mice

αB-Crystallin Modulates Protein Aggregation of Abnormal Desmin

Cardiac-specific overexpression of fibroblast growth factor-2 protects against myocardial dysfunction and infarction in a murine model of low-flow ischemia

Transgenic overexpression of cardiac actin in the mouse heart suggests coregulation of cardiac, skeletal and vascular actin expression

Forced expression of α-myosin heavy chain in the rabbit ventricle results in cardioprotection under cardiomyopathic conditions

Transgenic rabbit model for human troponin I-based hypertrophic cardiomyopathy

Analysis of Myosin Heavy Chain Functionality in the Heart

Transgenic Modeling of a Cardiac Troponin I Mutation Linked to Familial Hypertrophic Cardiomyopathy

Altered focal adhesion regulation correlates with cardiomyopathy in mice expressing constitutively active rac1

Role of the acidic N' region of cardiac troponin I in regulating myocardial function

Calcineurin A β Gene Targeting Predisposes the Myocardium to Acute Ischemia-Induced Apoptosis and Dysfunction

A mouse model of myosin binding protein C human familial hypertrophic cardiomyopathy

Myofibril degeneration caused by tropomodulin overexpression leads to dilated cardiomyopathy in juvenile mice

Disruption of Rho signaling results in progressive atrioventricular conduction defects while ventricular function remains preserved

In Vivo Modeling of Myosin Binding Protein C Familial Hypertrophic Cardiomyopathy

Proinflammatory consequences of transgenic fas ligand expression in the heart

Charged residue changes in the carboxy‐terminus of α‐tropomyosin alter mouse cardiac muscle contractility

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