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Failure of Costimulatory Blockade-induced Regulatory T Cells to Sustain Long-term Survival of High Ischemic Allografts

Transplantation, 2023-09, Vol.107 (9), p.1935-1944

2023

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Sex-Biased T-cell Exhaustion Drives Differential Immune Responses in Glioblastoma

Cancer discovery, 2023-09, Vol.13 (9), p.2090-2105

2023

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CD8 T cells producing IL-17 and IFN-gamma initiate the innate immune response required for responses to antigen skin challenge

The Journal of immunology (1950), 2009-05, Vol.182 (10), p.5949-5959

2009

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Myeloid‐derived suppressor cells increase and inhibit donor‐reactive T cell responses to graft intestinal epithelium in intestinal transplant patients

American journal of transplantation, 2018-10, Vol.18 (10), p.2544-2558

2018

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Neutrophil Cathepsin G Regulates Dendritic Cell Production of IL-12 during Development of CD4 T Cell Responses to Antigens in the Skin

The Journal of immunology (1950), 2019-02, Vol.202 (4), p.1045-1056

2019

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Neutrophil expression of Fas ligand and perforin directs effector CD8 T cell infiltration into antigen-challenged skin

The Journal of immunology (1950), 2012-09, Vol.189 (5), p.2191-2202

2012

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The Toll–Interleukin-1 Receptor Member SIGIRR Regulates Colonic Epithelial Homeostasis, Inflammation, and Tumorigenesis

Immunity (Cambridge, Mass.), 2007-04, Vol.26 (4), p.461-475

2007

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IL-1 receptor signaling is required at multiple stages of sensitization and elicitation of the contact hypersensitivity response

The Journal of immunology (1950), 2012-02, Vol.188 (4), p.1761-1771

2012

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CXC Chemokine Ligand (CXCL) 9 and CXCL10 Are Antagonistic Costimulation Molecules during the Priming of Alloreactive T Cell Effectors

The Journal of immunology (1950), 2010-04, Vol.184 (7), p.3450-3460

2010

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TLR4 dependent generation of a novel bone marrow-derived inflammatory monocyte population required for elicitation of skin contact hypersensitivity responses

The Journal of immunology (1950), 2023-05, Vol.210 (1_Supplement), p.71-71.18

2023

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Hapten application to the skin induces an inflammatory program directing hapten-primed effector CD8 T cell interaction with hapten-presenting endothelial cells

The Journal of immunology (1950), 2011-02, Vol.186 (4), p.2117-2126

2011

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The intensity of neutrophil infiltration controls the number of antigen-primed CD8 T cells recruited into cutaneous antigen challenge sites

Journal of leukocyte biology, 2004-11, Vol.76 (5), p.941-949

2004

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Hapten skin sensitization induces development of TLR4-dependent inflammatory monocytes that are required for elicitation of skin contact hypersensitivity responses αααα

The Journal of immunology (1950), 2022-05, Vol.208 (1_Supplement), p.105-105.15

2022

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IL-1 Receptor Signaling on Graft Parenchymal Cells Regulates Memory and De Novo Donor-Reactive CD8 T Cell Responses to Cardiac Allografts

The Journal of immunology (1950), 2016-03, Vol.196 (6), p.2827-2837

2016

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A novel population of myeloid-derived Gr-1+ CXCR2+CD11c+Ly6C+cells expressing FasL and perforin is required for contact hypersensitivity responses (CAM4P.150)

The Journal of immunology (1950), 2015-05, Vol.194 (1_Supplement), p.185-185.8

Kish, Danielle

2015

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Roles of the CCL20-CCR6 axis and CXCR3 in CD8 T cell-mediated contact hypersensitivity (CCR5P.211)

The Journal of immunology (1950), 2015-05, Vol.194 (1_Supplement), p.186-186.13

Kish, Danielle

2015

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CD8+ T cells produce IL-2, which is required for CD4+CD25+ T cell regulation of effector CD8+ T cell development for contact hypersensitivity responses

Journal of leukocyte biology, 2005-09, Vol.78 (3), p.725-735

2005

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The bone marrow-derived Gr-1+CXCR2+ cells are required for T cell infiltration into the skin challenge site to elicit contact hypersensitivity responses are inflammatory monocytes expressing FasL and perforin

The Journal of immunology (1950), 2019-05, Vol.202 (1_Supplement), p.55-55.8

2019

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Effector CD8 T Cell Infiltration into the Skin to Mediate Contact Hypersensitivity Responses Requires TLR4 Signaling on Precursors of Gr-1+CXCR2+Bone Marrow Cells

The Journal of immunology (1950), 2019-05, Vol.202 (1_Supplement), p.55-55.9

2019

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Regulatory function of CD4+CD25+ T cells from Class II MHC-deficient mice in contact hypersensitivity responses

Journal of leukocyte biology, 2007-07, Vol.82 (1), p.85-92

2007

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Alloreactive T Cell Responses and Acute Rejection of Single Class II MHC-Disparate Heart Allografts Are under Strict Regulation by CD4+CD25+ T Cells

The Journal of immunology (1950), 2005-03, Vol.174 (6), p.3741-3748

2005

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Trafficking of IL-17-producing CD4+, but not CD8+, T cells to the skin requires CCR6 expression

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.119-119.16

2016

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Hapten-induced generation of a novel myeloid-derived FasL+ perforin+cell population in the bone marrow is induced by TNFa

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.191-191.15

2016

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Neutrophil cathepsin G inhibits hapten-presenting dendritic cell production of IL-12 and skewing of hapten-reactive CD4 T Cell development to IFN-γ- and IL-17-producing effector cells in allergic contact sensitivity

The Journal of immunology (1950), 2018-05, Vol.200 (1_Supplement), p.49-49.6

2018

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A novel population of myeloid Gr-1+ cells expressing FasL and perforin mediate contact hypersensitivity responses in skin (CAM5P.236)

The Journal of immunology (1950), 2014-05, Vol.192 (1_Supplement), p.180-180.7

2014

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Failure of Costimulatory Blockade-induced Regulatory T Cells to Sustain Long-term Survival of High Ischemic Allografts

Sex-Biased T-cell Exhaustion Drives Differential Immune Responses in Glioblastoma

CD8 T cells producing IL-17 and IFN-gamma initiate the innate immune response required for responses to antigen skin challenge

Myeloid‐derived suppressor cells increase and inhibit donor‐reactive T cell responses to graft intestinal epithelium in intestinal transplant patients

Neutrophil Cathepsin G Regulates Dendritic Cell Production of IL-12 during Development of CD4 T Cell Responses to Antigens in the Skin

Neutrophil expression of Fas ligand and perforin directs effector CD8 T cell infiltration into antigen-challenged skin

The Toll–Interleukin-1 Receptor Member SIGIRR Regulates Colonic Epithelial Homeostasis, Inflammation, and Tumorigenesis

IL-1 receptor signaling is required at multiple stages of sensitization and elicitation of the contact hypersensitivity response

CXC Chemokine Ligand (CXCL) 9 and CXCL10 Are Antagonistic Costimulation Molecules during the Priming of Alloreactive T Cell Effectors

TLR4 dependent generation of a novel bone marrow-derived inflammatory monocyte population required for elicitation of skin contact hypersensitivity responses

Hapten application to the skin induces an inflammatory program directing hapten-primed effector CD8 T cell interaction with hapten-presenting endothelial cells

The intensity of neutrophil infiltration controls the number of antigen-primed CD8 T cells recruited into cutaneous antigen challenge sites

Hapten skin sensitization induces development of TLR4-dependent inflammatory monocytes that are required for elicitation of skin contact hypersensitivity responses αααα

IL-1 Receptor Signaling on Graft Parenchymal Cells Regulates Memory and De Novo Donor-Reactive CD8 T Cell Responses to Cardiac Allografts

A novel population of myeloid-derived Gr-1+ CXCR2+CD11c+Ly6C+cells expressing FasL and perforin is required for contact hypersensitivity responses (CAM4P.150)

Roles of the CCL20-CCR6 axis and CXCR3 in CD8 T cell-mediated contact hypersensitivity (CCR5P.211)

CD8+ T cells produce IL-2, which is required for CD4+CD25+ T cell regulation of effector CD8+ T cell development for contact hypersensitivity responses

The bone marrow-derived Gr-1+CXCR2+ cells are required for T cell infiltration into the skin challenge site to elicit contact hypersensitivity responses are inflammatory monocytes expressing FasL and perforin

Effector CD8 T Cell Infiltration into the Skin to Mediate Contact Hypersensitivity Responses Requires TLR4 Signaling on Precursors of Gr-1+CXCR2+Bone Marrow Cells

Regulatory function of CD4+CD25+ T cells from Class II MHC-deficient mice in contact hypersensitivity responses

Alloreactive T Cell Responses and Acute Rejection of Single Class II MHC-Disparate Heart Allografts Are under Strict Regulation by CD4+CD25+ T Cells

Trafficking of IL-17-producing CD4+, but not CD8+, T cells to the skin requires CCR6 expression

Hapten-induced generation of a novel myeloid-derived FasL+ perforin+cell population in the bone marrow is induced by TNFa

Neutrophil cathepsin G inhibits hapten-presenting dendritic cell production of IL-12 and skewing of hapten-reactive CD4 T Cell development to IFN-γ- and IL-17-producing effector cells in allergic contact sensitivity

A novel population of myeloid Gr-1+ cells expressing FasL and perforin mediate contact hypersensitivity responses in skin (CAM5P.236)

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