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siRNA Library Screening Identifies a Druggable Immune-Signature Driving Esophageal Adenocarcinoma Cell Growth

Cellular and molecular gastroenterology and hepatology, 2018-01, Vol.5 (4), p.569-590

2018

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Sa1654 - Gdf15 Mrna Expression is Enriched in Esophageal Adenocarcinoma Patients Presenting with a Smad4 Somatic Variation

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-343-S-343

2018

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Golgi phosphoprotein 2 (GOLPH2) is a novel bile acid-responsive modulator of oesophageal cell migration and invasion

British journal of cancer, 2015-11, Vol.113 (9), p.1332-1342

2015

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The characterization of an intestine-like genomic signature maintained during Barrett’s-associated adenocarcinogenesis reveals an NR5A2-mediated promotion of cancer cell survival

Scientific reports, 2016-09, Vol.6 (1), p.32638-32638, Article 32638

2016

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Correction: Corrigendum: The characterization of an intestine-like genomic signature maintained during Barrett’s-associated adenocarcinogenesis reveals an NR5A2-mediated promotion of cancer cell survival

Scientific reports, 2016-10, Vol.6 (1), Article 35330

2016

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An integrative genomic approach in oesophageal cells identifies TRB3 as a bile acid responsive gene, downregulated in Barrett's oesophagus, which regulates NF-κB activation and cytokine levels

Carcinogenesis (New York), 2010-05, Vol.31 (5), p.936-945

2010

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Su1960 The Expression of Dual Specificity Phosphatases 1, 5 and 6 Is Modulated by Acid and Bile Acid Exposure and Varies at Different Stages of Esophageal Carcinogenesis

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-561-S-561

2015

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siRNA library screening to define esophageal adenocarcinoma cell survival factors

Journal of clinical oncology, 2015-01, Vol.33 (3_suppl), p.70-70

2015

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The diagnostic accuracy of endoscopic ultrasound in suspected biliary obstruction and its impact on endoscopic retrograde cholangiopancreatography burden in real clinical practice: a consecutive analysis

European journal of gastroenterology & hepatology, 2013-07, Vol.25 (7), p.850-857

2013

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859 A Genomic Intestinal Signature of Barrett's Esophagus (BE) Defines Reflux Inducible Transcription Factors Proximally Encoded to BE and Adenocarcinoma Associated Genetic Loci

Gastroenterology (New York, N.Y. 1943), 2014-05, Vol.146 (5), p.S-148-S-148

2014

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Su1174 Members of the Dual Specificity Protein Phosphatase Subfamily 1,5 and 6 are Influenced by Bile Acids and Over-Expressed in Barrett's Esophagus

Gastroenterology (New York, N.Y. 1943), 2012-05, Vol.142 (5), p.S-443-S-443

2012

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Inherited Syndromes Predisposing to Inflammation and GI Cancer

Recent results in cancer research, 2011-01, Vol.185, p.35

2011

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S1638 A Novel Genomic Intestinal Filter Identifies NR5A2 and TEAD1 as Novel Intestinal Transcription Factors With the Potential to Interact With Gata6 in the Development of Barrett's Esophagus (BE) and Esophageal. Adenocarcinoma (EAC)

Gastroenterology (New York, N.Y. 1943), 2010, Vol.138 (5), p.S-243-S-244

2010

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siRNA Library Screening Identifies a Druggable Immune-Signature Driving Esophageal Adenocarcinoma Cell GrowthSummary

Cellular and molecular gastroenterology and hepatology, 2018-01, Vol.5 (4), p.569-590

2018

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An integrative genomic approach in oesophageal cells identifies TRB3 as a bile acid responsive gene, downregulated in Barrett's oesophagus, which regulates NF-B activation and cytokine levels

Carcinogenesis (New York), 2010-05, Vol.31 (5), p.936-945

2010

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Down-Regulation of Rora Transcriptional Target, Aryl Hydrocarbon Receptor Nuclear Translocator-Like 2 (ARNTL2), in Barrett's Metaplasia, Implications for Circadian Rhythm

Gastroenterology (New York, N.Y. 1943), 2011, Vol.140 (5), p.S-221-S-221

2011

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Gdf15 is Over-Expressed in Barrett's Esophagus (BE), Induces SMAD Activation and May Play a Crucial Role in the Transcriptional Cascade Leading to Cell Differentiation and Transformation

Gastroenterology (New York, N.Y. 1943), 2011, Vol.140 (5), p.S-221-S-221

2011

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S1635 Loss of Bile Acid Responsive Gene Retinoic Acid Receptor-Related Orphan Receptor A (Rora) in Barrett's Metaplasia, Implications for Refluxed Induced Signaling in Esophageal Cells

Gastroenterology (New York, N.Y. 1943), 2010, Vol.138 (5), p.S-243-S-243

2010

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T1939 Use of a Combined Microarray and Informatics Approach to Define a Novel Genomic Filter Identifying An Intestinal Signature of Gene Expression in Barrett's Esophagus and Esophageal Adenocarcinoma

Gastroenterology (New York, N.Y. 1943), 2009, Vol.136 (5), p.A-604-A-605

2009

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T1905 Gene Groups Modulated By Bile Acid Signaling Show Altered Expression in Tissue from Barrett's Esophagus

Gastroenterology (New York, N.Y. 1943), 2009, Vol.136 (5), p.A-597-A-598

2009

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An integrative genomic approach in oesophageal cells identifies TRB3 as a bile acid responsive gene, downregulated in Barrett's oesophagus, which regulates NF-KB activation and cytokine levels

Carcinogenesis (New York), 2010, Vol.31 (5), p.936-945

2010

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ÇEVRESEL YÖNETİM VE İÇSEL PAZARLAMA – YERLİ HAVAYOLU ÇALIŞANLARI ÖRNEĞİ

Third Sector Social Economic Review, 2023

2023

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Mo1390 EUS Significantly Reduces the Burden of ERCP and Accurately Predicts CBD Stones in Patients With Suspected Biliary Obstruction: Consecutive Analysis of 418 Patients

Gastrointestinal endoscopy, 2011-04, Vol.73 (4), p.AB328-AB329

2011

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Corrigendum: The characterization of an intestine-like genomic signature maintained during Barrett's-associated adenocarcinogenesis reveals an NR5A2-mediated promotion of cancer cell survival

Scientific reports, 2016-10, Vol.6, p.35330-35330

2016

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An integrative genomic approach in oesophageal cells identifies TRB3 as a bile acid responsive gene, downregulated in Barrett's oesophagus, which regulates NF-kappaB activation and cytokine levels

Carcinogenesis (New York), 2010-05, Vol.31 (5), p.936

2010

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siRNA Library Screening Identifies a Druggable Immune-Signature Driving Esophageal Adenocarcinoma Cell Growth

Sa1654 - Gdf15 Mrna Expression is Enriched in Esophageal Adenocarcinoma Patients Presenting with a Smad4 Somatic Variation

Golgi phosphoprotein 2 (GOLPH2) is a novel bile acid-responsive modulator of oesophageal cell migration and invasion

The characterization of an intestine-like genomic signature maintained during Barrett’s-associated adenocarcinogenesis reveals an NR5A2-mediated promotion of cancer cell survival

Correction: Corrigendum: The characterization of an intestine-like genomic signature maintained during Barrett’s-associated adenocarcinogenesis reveals an NR5A2-mediated promotion of cancer cell survival

An integrative genomic approach in oesophageal cells identifies TRB3 as a bile acid responsive gene, downregulated in Barrett's oesophagus, which regulates NF-κB activation and cytokine levels

Su1960 The Expression of Dual Specificity Phosphatases 1, 5 and 6 Is Modulated by Acid and Bile Acid Exposure and Varies at Different Stages of Esophageal Carcinogenesis

siRNA library screening to define esophageal adenocarcinoma cell survival factors

The diagnostic accuracy of endoscopic ultrasound in suspected biliary obstruction and its impact on endoscopic retrograde cholangiopancreatography burden in real clinical practice: a consecutive analysis

859 A Genomic Intestinal Signature of Barrett's Esophagus (BE) Defines Reflux Inducible Transcription Factors Proximally Encoded to BE and Adenocarcinoma Associated Genetic Loci

Su1174 Members of the Dual Specificity Protein Phosphatase Subfamily 1,5 and 6 are Influenced by Bile Acids and Over-Expressed in Barrett's Esophagus

Inherited Syndromes Predisposing to Inflammation and GI Cancer

S1638 A Novel Genomic Intestinal Filter Identifies NR5A2 and TEAD1 as Novel Intestinal Transcription Factors With the Potential to Interact With Gata6 in the Development of Barrett's Esophagus (BE) and Esophageal. Adenocarcinoma (EAC)

siRNA Library Screening Identifies a Druggable Immune-Signature Driving Esophageal Adenocarcinoma Cell GrowthSummary

An integrative genomic approach in oesophageal cells identifies TRB3 as a bile acid responsive gene, downregulated in Barrett's oesophagus, which regulates NF-B activation and cytokine levels

Down-Regulation of Rora Transcriptional Target, Aryl Hydrocarbon Receptor Nuclear Translocator-Like 2 (ARNTL2), in Barrett's Metaplasia, Implications for Circadian Rhythm

Gdf15 is Over-Expressed in Barrett's Esophagus (BE), Induces SMAD Activation and May Play a Crucial Role in the Transcriptional Cascade Leading to Cell Differentiation and Transformation

S1635 Loss of Bile Acid Responsive Gene Retinoic Acid Receptor-Related Orphan Receptor A (Rora) in Barrett's Metaplasia, Implications for Refluxed Induced Signaling in Esophageal Cells

T1939 Use of a Combined Microarray and Informatics Approach to Define a Novel Genomic Filter Identifying An Intestinal Signature of Gene Expression in Barrett's Esophagus and Esophageal Adenocarcinoma

T1905 Gene Groups Modulated By Bile Acid Signaling Show Altered Expression in Tissue from Barrett's Esophagus

An integrative genomic approach in oesophageal cells identifies TRB3 as a bile acid responsive gene, downregulated in Barrett's oesophagus, which regulates NF-KB activation and cytokine levels

ÇEVRESEL YÖNETİM VE İÇSEL PAZARLAMA – YERLİ HAVAYOLU ÇALIŞANLARI ÖRNEĞİ

Mo1390 EUS Significantly Reduces the Burden of ERCP and Accurately Predicts CBD Stones in Patients With Suspected Biliary Obstruction: Consecutive Analysis of 418 Patients

Corrigendum: The characterization of an intestine-like genomic signature maintained during Barrett's-associated adenocarcinogenesis reveals an NR5A2-mediated promotion of cancer cell survival

An integrative genomic approach in oesophageal cells identifies TRB3 as a bile acid responsive gene, downregulated in Barrett's oesophagus, which regulates NF-kappaB activation and cytokine levels

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