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Autophagy in Chronically Ischemic Myocardium

Proceedings of the National Academy of Sciences - PNAS, 2005-09, Vol.102 (39), p.13807-13812

2005

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Venous thromboembolism prophylaxis usage among surgical patients in both private and public hospitals in Ethiopia

Heliyon, 2024-07, Vol.10 (13), p.e33450, Article e33450

2024

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Computer model of action potential of mouse ventricular myocytes

American journal of physiology. Heart and circulatory physiology, 2004-09, Vol.287 (3), p.H1378-H1403

2004

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Aging increases stiffness of cardiac myocytes measured by atomic force microscopy nanoindentation

American journal of physiology. Heart and circulatory physiology, 2004-08, Vol.287 (2), p.H645-H651

2004

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Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy

The Journal of clinical investigation, 2003-05, Vol.111 (10), p.1463-1474

2003

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Program of Cell Survival Underlying Human and Experimental Hibernating Myocardium

Circulation Research, 2004-08, Vol.95 (4), p.433-440

2004

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Mechanism of Enhanced Cardiac Function in Mice with Hypertrophy Induced by Overexpressed Akt

The Journal of biological chemistry, 2003-11, Vol.278 (48), p.47622-47628

2003

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Gene Program for Cardiac Cell Survival Induced by Transient Ischemia in Conscious Pigs

Proceedings of the National Academy of Sciences - PNAS, 2001-07, Vol.98 (16), p.9336-9341

2001

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Evidence for enhanced eNOS function in coronary microvessels during the second window of protection

American journal of physiology. Heart and circulatory physiology, 2007-05, Vol.292 (5), p.H2152-H2158

2007

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Chronic treatment with insulin-like growth factor I enhances myocyte contraction by upregulation of Akt-SERCA2a signaling pathway

American journal of physiology. Heart and circulatory physiology, 2008-07, Vol.295 (1), p.H130-H135

2008

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Novel mechanisms mediating stunned myocardium

Heart failure reviews, 2003-04, Vol.8 (2), p.143-153

2003

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In vivo gene delivery of XIAP protects against myocardial apoptosis and infarction following ischemia/reperfusion in conscious rabbits

Life sciences (1973), 2011-03, Vol.88 (13-14), p.572-577

2011

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Nitric oxide has no obligatory role in isoflurane late preconditioning against myocardial stunning

Life sciences (1973), 2012-12, Vol.91 (23-24), p.1201-1206

2012

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Altered Excitation–Contraction Coupling in Myocytes from Remodeled Myocardium after Chronic Myocardial Infarction

Journal of molecular and cellular cardiology, 2002-01, Vol.34 (1), p.63-73

2002

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Delayed Enhanced Nitric Oxide-Mediated Coronary Vasodilation Following Brief Ischemia and Prolonged Reperfusion in Conscious Dogs

Circulation research, 1997-07, Vol.81 (1), p.53-59

1997

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Sleep-Induced Hypotension Precipitates Severe Myocardial Ischemia

Sleep (New York, N.Y.), 2008-09, Vol.31 (9), p.1215-1220

2008

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Persistent Stunning Induces Myocardial Hibernation and Protection: Flow/Function and Metabolic Mechanisms

Circulation research, 2003-06, Vol.92 (11), p.1233-1239

2003

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Persistency and pathway of isoflurane-induced inhibition of superoxide production by neutrophils

Canadian journal of anesthesia, 2010, Vol.57 (1), p.50-57

2010

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Paradoxical Cellular Ca2+ Signaling in Severe but Compensated Canine Left Ventricular Hypertrophy

Circulation research, 2005-09, Vol.97 (5), p.457-464

2005

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A novel mechanism for myocardial stunning involving impaired Ca(2+) handling

Circulation research, 2001-10, Vol.89 (9), p.831-837

2001

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A Novel Mechanism for Myocardial Stunning Involving Impaired Ca2+ Handling

Circulation research, 2001-10, Vol.89 (9), p.831-837

2001

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Hemodilution does not alter the coronary vasodilating effects of endogenous or exogenous nitric oxide

Canadian journal of anesthesia, 2008-08, Vol.55 (8), p.507-514

2008

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Computer model of action potential of mouse ventricular myocytes

American journal of physiology. Heart and circulatory physiology, 2004-09, Vol.56 (3), p.H1378-H1403

2004

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Enhanced iNOS function in myocytes one day after brief ischemic episode

American journal of physiology. Heart and circulatory physiology, 2002-02, Vol.282 (2), p.H423-H428

2002

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Accelerated cardiomyopathy in mice with overexpression of cardiac Gsα and a missense mutation in the α-myosin heavy chain

Circulation (New York, N.Y.), 2002-02, Vol.105 (5), p.614-620

2002

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Autophagy in Chronically Ischemic Myocardium

Venous thromboembolism prophylaxis usage among surgical patients in both private and public hospitals in Ethiopia

Computer model of action potential of mouse ventricular myocytes

Aging increases stiffness of cardiac myocytes measured by atomic force microscopy nanoindentation

Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy

Program of Cell Survival Underlying Human and Experimental Hibernating Myocardium

Mechanism of Enhanced Cardiac Function in Mice with Hypertrophy Induced by Overexpressed Akt

Gene Program for Cardiac Cell Survival Induced by Transient Ischemia in Conscious Pigs

Evidence for enhanced eNOS function in coronary microvessels during the second window of protection

Chronic treatment with insulin-like growth factor I enhances myocyte contraction by upregulation of Akt-SERCA2a signaling pathway

Novel mechanisms mediating stunned myocardium

In vivo gene delivery of XIAP protects against myocardial apoptosis and infarction following ischemia/reperfusion in conscious rabbits

Nitric oxide has no obligatory role in isoflurane late preconditioning against myocardial stunning

Altered Excitation–Contraction Coupling in Myocytes from Remodeled Myocardium after Chronic Myocardial Infarction

Delayed Enhanced Nitric Oxide-Mediated Coronary Vasodilation Following Brief Ischemia and Prolonged Reperfusion in Conscious Dogs

Sleep-Induced Hypotension Precipitates Severe Myocardial Ischemia

Persistent Stunning Induces Myocardial Hibernation and Protection: Flow/Function and Metabolic Mechanisms

Persistency and pathway of isoflurane-induced inhibition of superoxide production by neutrophils

Paradoxical Cellular Ca2+ Signaling in Severe but Compensated Canine Left Ventricular Hypertrophy

A novel mechanism for myocardial stunning involving impaired Ca(2+) handling

A Novel Mechanism for Myocardial Stunning Involving Impaired Ca2+ Handling

Hemodilution does not alter the coronary vasodilating effects of endogenous or exogenous nitric oxide

Computer model of action potential of mouse ventricular myocytes

Enhanced iNOS function in myocytes one day after brief ischemic episode

Accelerated cardiomyopathy in mice with overexpression of cardiac Gsα and a missense mutation in the α-myosin heavy chain

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