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Alveolar Epithelial Cell Mesenchymal Transition Develops in vivo during Pulmonary Fibrosis and Is Regulated by the Extracellular Matrix

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (35), p.13180-13185

2006

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Inhibition of Myocardin-Related Transcription Factor/Serum Response Factor Signaling Decreases Lung Fibrosis and Promotes Mesenchymal Cell Apoptosis

The American journal of pathology, 2015-04, Vol.185 (4), p.969-986

2015

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TGF-β1 Signaling and Tissue Fibrosis

Cold Spring Harbor perspectives in biology, 2018-04, Vol.10 (4), p.a022293

2018

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Activated alveolar epithelial cells initiate fibrosis through autocrine and paracrine secretion of connective tissue growth factor

American journal of physiology. Lung cellular and molecular physiology, 2014-04, Vol.306 (8), p.L786-L796

2014

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CD36/Lyn kinase interactions within macrophages promotes pulmonary fibrosis in response to oxidized phospholipid

Respiratory research, 2023-12, Vol.24 (1), p.314-17, Article 314

2023

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Efferocytosis of apoptotic alveolar epithelial cells is sufficient to initiate lung fibrosis

Cell death & disease, 2018-10, Vol.9 (11), p.1056-12, Article 1056

2018

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Inhibition of discoidin domain receptor 2 reveals kinase-dependent and kinase-independent functions in regulating fibroblast activity

American journal of physiology. Lung cellular and molecular physiology, 2023-09, Vol.325 (3), p.L342-L351

2023

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Computational Modeling Predicts Simultaneous Targeting of Fibroblasts and Epithelial Cells Is Necessary for Treatment of Pulmonary Fibrosis

Frontiers in pharmacology, 2016-06, Vol.7, p.183-183

2016

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Paracrine functions of fibrocytes to promote lung fibrosis

Expert review of respiratory medicine, 2014-04, Vol.8 (2), p.163-172

2014

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The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice

Blood, 2011-08, Vol.118 (8), p.2313-2321

2011

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Discoidin domain receptor 2 signaling through PIK3C2α in fibroblasts promotes lung fibrosis

The Journal of pathology, 2024-04, Vol.262 (4), p.505-516

2024

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Lung epithelial cell focal adhesion kinase signaling inhibits lung injury and fibrosis

American journal of physiology. Lung cellular and molecular physiology, 2017-05, Vol.312 (5), p.L722-L730

2017

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Diverse Injury Pathways Induce Alveolar Epithelial Cell CCL2/12, Which Promotes Lung Fibrosis

American journal of respiratory cell and molecular biology, 2020-05, Vol.62 (5), p.622-632

2020

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Discoidin Domain Receptor 2 Signaling Regulates Fibroblast Apoptosis through PDK1/Akt

American journal of respiratory cell and molecular biology, 2018-09, Vol.59 (3), p.295-305

2018

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Telomerase reverse transcriptase ameliorates lung fibrosis by protecting alveolar epithelial cells against senescence

The Journal of biological chemistry, 2019-05, Vol.294 (22), p.8861-8871

2019

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Characteristics and Outcomes of Mechanically Ventilated COVID-19 Patients—An Observational Cohort Study

Journal of intensive care medicine, 2021-03, Vol.36 (3), p.271-276

2021

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Fibroblast growth factors and pulmonary fibrosis: it's more complex than it sounds

The Journal of pathology, 2017-01, Vol.241 (1), p.6-9

2017

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Type I Collagen Signaling Regulates Opposing Fibrotic Pathways through α2β1 Integrin

American journal of respiratory cell and molecular biology, 2020-11, Vol.63 (5), p.613-622

2020

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Type I Collagen Signaling Regulates Opposing Fibrotic Pathways through α 2 β 1 Integrin

American journal of respiratory cell and molecular biology, 2020-11, Vol.63 (5), p.613-622

2020

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Epithelial cell α3β1 integrin links β-catenin and Smad signaling to promote myofibroblast formation and pulmonary fibrosis

The Journal of clinical investigation, 2009-01, Vol.119 (1), p.213-224

2009

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Spatial heterogeneity of bone marrow endothelial cells unveils a distinct subtype in the epiphysis

Nature cell biology, 2023-10, Vol.25 (10), p.1415-1425

2023

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IL‐17A deficiency mitigates bleomycin‐induced complement activation during lung fibrosis

The FASEB journal, 2017-12, Vol.31 (12), p.5543-5556

2017

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IL‐17A deficiency mitigates bleomycin‐induced complement activation during lung fibrosis

The FASEB journal, 2017-12, Vol.31 (12), p.5543-5556

2017

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Axin Pathway Activity Regulates in Vivo pY654-β-catenin Accumulation and Pulmonary Fibrosis

The Journal of biological chemistry, 2012-02, Vol.287 (7), p.5164-5172

2012

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Fibrocytes are not an essential source of type I collagen during lung fibrosis

The Journal of immunology (1950), 2014-11, Vol.193 (10), p.5229-5239

2014

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Alveolar Epithelial Cell Mesenchymal Transition Develops in vivo during Pulmonary Fibrosis and Is Regulated by the Extracellular Matrix

Inhibition of Myocardin-Related Transcription Factor/Serum Response Factor Signaling Decreases Lung Fibrosis and Promotes Mesenchymal Cell Apoptosis

TGF-β1 Signaling and Tissue Fibrosis

Activated alveolar epithelial cells initiate fibrosis through autocrine and paracrine secretion of connective tissue growth factor

CD36/Lyn kinase interactions within macrophages promotes pulmonary fibrosis in response to oxidized phospholipid

Efferocytosis of apoptotic alveolar epithelial cells is sufficient to initiate lung fibrosis

Inhibition of discoidin domain receptor 2 reveals kinase-dependent and kinase-independent functions in regulating fibroblast activity

Computational Modeling Predicts Simultaneous Targeting of Fibroblasts and Epithelial Cells Is Necessary for Treatment of Pulmonary Fibrosis

Paracrine functions of fibrocytes to promote lung fibrosis

The vitronectin-binding function of PAI-1 exacerbates lung fibrosis in mice

Discoidin domain receptor 2 signaling through PIK3C2α in fibroblasts promotes lung fibrosis

Lung epithelial cell focal adhesion kinase signaling inhibits lung injury and fibrosis

Diverse Injury Pathways Induce Alveolar Epithelial Cell CCL2/12, Which Promotes Lung Fibrosis

Discoidin Domain Receptor 2 Signaling Regulates Fibroblast Apoptosis through PDK1/Akt

Telomerase reverse transcriptase ameliorates lung fibrosis by protecting alveolar epithelial cells against senescence

Characteristics and Outcomes of Mechanically Ventilated COVID-19 Patients—An Observational Cohort Study

Fibroblast growth factors and pulmonary fibrosis: it's more complex than it sounds

Type I Collagen Signaling Regulates Opposing Fibrotic Pathways through α2β1 Integrin

Type I Collagen Signaling Regulates Opposing Fibrotic Pathways through α 2 β 1 Integrin

Epithelial cell α3β1 integrin links β-catenin and Smad signaling to promote myofibroblast formation and pulmonary fibrosis

Spatial heterogeneity of bone marrow endothelial cells unveils a distinct subtype in the epiphysis

IL‐17A deficiency mitigates bleomycin‐induced complement activation during lung fibrosis

IL‐17A deficiency mitigates bleomycin‐induced complement activation during lung fibrosis

Axin Pathway Activity Regulates in Vivo pY654-β-catenin Accumulation and Pulmonary Fibrosis

Fibrocytes are not an essential source of type I collagen during lung fibrosis

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