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Integrated pharmaco-proteogenomics defines two subgroups in isocitrate dehydrogenase wild-type glioblastoma with prognostic and therapeutic opportunities

Nature communications, 2020-07, Vol.11 (1), p.3288-3288, Article 3288

2020

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Salinomycin sensitizes cancer cells to the effects of doxorubicin and etoposide treatment by increasing DNA damage and reducing p21 protein

British journal of pharmacology, 2011-02, Vol.162 (3), p.773-784

2011

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Salinomycin sensitizes antimitotic drugs-treated cancer cells by increasing apoptosis via the prevention of G2 arrest

Biochemical and biophysical research communications, 2012-02, Vol.418 (1), p.98-103

2012

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A new anthropometric index to predict percent body fat in young adults

Public health nutrition, 2020-06, Vol.23 (9), p.1507-1514

2020

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Meta-Analysis of Large-Scale Toxicogenomic Data Finds Neuronal Regeneration Related Protein and Cathepsin D to Be Novel Biomarkers of Drug-Induced Toxicity

PloS one, 2015-09, Vol.10 (9), p.e0136698

2015

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Correction: Meta-Analysis of Large-Scale Toxicogenomic Data Finds Neuronal Regeneration Related Protein and Cathepsin D to Be Novel Biomarkers of Drug-Induced Toxicity

PloS one, 2016-09, Vol.11 (9), p.e0163403

2016

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Low amount of salinomycin greatly increases Akt activation, but reduces activated p70S6K levels

International journal of molecular sciences, 2013-08, Vol.14 (9), p.17304-17318

2013

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Lower salinomycin concentration increases apoptotic detachment in high-density cancer cells

International journal of molecular sciences, 2012-10, Vol.13 (10), p.13169-13182

2012

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Anti-malarial Drugs Primaquine and Chloroquine Have Different Sensitization Effects with Anti-mitotic Drugs in Resistant Cancer Cells

Anticancer research, 2016-04, Vol.36 (4), p.1641-1648

2016

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SP600125, an inhibitor of Jnk pathway, reduces viability of relatively resistant cancer cells to doxorubicin

Biochemical and biophysical research communications, 2009-09, Vol.387 (3), p.450-455

2009

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SOCS5 and SOCS6 have similar expression patterns in normal and cancer tissues

Tumor biology, 2012-02, Vol.33 (1), p.215-221

2012

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Attenuation of Colchicine Toxicity in Drug-resistant Cancer Cells by Co-treatment with Anti-malarial Drugs

Anticancer research, 2016-11, Vol.36 (11), p.5859-5866

2016

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Salinomycin, a p-glycoprotein inhibitor, sensitizes radiation-treated cancer cells by increasing DNA damage and inducing G2 arrest

Investigational new drugs, 2012-08, Vol.30 (4), p.1311-1318

2012

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Sensitization of Cancer Cells through Reduction of Total Akt and Downregulation of Salinomycin-Induced pAkt, pGSk3β, pTSC2, and p4EBP1 by Cotreatment with MK-2206

BioMed research international, 2014-01, Vol.2014 (2014), p.1-8

2014

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Co-treatment with the anti-malarial drugs mefloquine and primaquine highly sensitizes drug-resistant cancer cells by increasing P-gp inhibition

Biochemical and biophysical research communications, 2013-11, Vol.441 (3), p.655-660

2013

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Co-treatment of LY294002 or MK-2206 with AZD5363 Attenuates AZD5363-induced Increase in the Level of Phosphorylated AKT

Anticancer research, 2016-11, Vol.36 (11), p.5849-5858

2016

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Thioridazine specifically sensitizes drug-resistant cancer cells through highly increase in apoptosis and P-gp inhibition

Tumor biology, 2014-10, Vol.35 (10), p.9831-9838

2014

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Jnk signaling pathway-mediated regulation of Stat3 activation is linked to the development of doxorubicin resistance in cancer cell lines

Biochemical pharmacology, 2010-02, Vol.79 (3), p.373-380

2010

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SP600125 overcomes antimitotic drug-resistance in cancer cells by increasing apoptosis with independence of P-gp inhibition

European journal of pharmacology, 2014-01, Vol.723, p.141-147

2014

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Co-treatment of Salinomycin Sensitizes AZD5363-treated Cancer Cells Through Increased Apoptosis

Anticancer research, 2015-09, Vol.35 (9), p.4741-4747

2015

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A novel carbazole derivative, MHY407, sensitizes cancer cells to doxorubicin-, etoposide-, and radiation treatment via DNA damage

European journal of pharmacology, 2012-12, Vol.697 (1-3), p.24-31

2012

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Corrigendum to "sensitization of cancer cells through reduction of total akt and downregulation of salinomycin-induced pAkt, pGSk3[beta], pTSC2, and p4EBP1 by Cotreatment with MK-2206"

BioMed research international, 2015-01, Vol.2015

2015

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Erratum to: Thioridazine specifically sensitizes drug-resistant cancer cells through highly increase in apoptosis and P-gp inhibition

Tumor biology, 2015-09, Vol.36 (9), p.7331-7331

2015

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A 16b 10MS/s digitally self-calibrated ADC with time constant control

2008 IEEE Custom Integrated Circuits Conference, 2008, p.113-116

2008

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Integrated pharmaco-proteogenomics defines two subgroups in isocitrate dehydrogenase wild-type glioblastoma with prognostic and therapeutic opportunities

Salinomycin sensitizes cancer cells to the effects of doxorubicin and etoposide treatment by increasing DNA damage and reducing p21 protein

Chemistry-First Approach for Nomination of Personalized Treatment in Lung Cancer

Salinomycin sensitizes antimitotic drugs-treated cancer cells by increasing apoptosis via the prevention of G2 arrest

A new anthropometric index to predict percent body fat in young adults

Meta-Analysis of Large-Scale Toxicogenomic Data Finds Neuronal Regeneration Related Protein and Cathepsin D to Be Novel Biomarkers of Drug-Induced Toxicity

Correction: Meta-Analysis of Large-Scale Toxicogenomic Data Finds Neuronal Regeneration Related Protein and Cathepsin D to Be Novel Biomarkers of Drug-Induced Toxicity

Low amount of salinomycin greatly increases Akt activation, but reduces activated p70S6K levels

Lower salinomycin concentration increases apoptotic detachment in high-density cancer cells

Anti-malarial Drugs Primaquine and Chloroquine Have Different Sensitization Effects with Anti-mitotic Drugs in Resistant Cancer Cells

SP600125, an inhibitor of Jnk pathway, reduces viability of relatively resistant cancer cells to doxorubicin

SOCS5 and SOCS6 have similar expression patterns in normal and cancer tissues

Attenuation of Colchicine Toxicity in Drug-resistant Cancer Cells by Co-treatment with Anti-malarial Drugs

Salinomycin, a p-glycoprotein inhibitor, sensitizes radiation-treated cancer cells by increasing DNA damage and inducing G2 arrest

Sensitization of Cancer Cells through Reduction of Total Akt and Downregulation of Salinomycin-Induced pAkt, pGSk3β, pTSC2, and p4EBP1 by Cotreatment with MK-2206

Co-treatment with the anti-malarial drugs mefloquine and primaquine highly sensitizes drug-resistant cancer cells by increasing P-gp inhibition

Co-treatment of LY294002 or MK-2206 with AZD5363 Attenuates AZD5363-induced Increase in the Level of Phosphorylated AKT

Thioridazine specifically sensitizes drug-resistant cancer cells through highly increase in apoptosis and P-gp inhibition

Jnk signaling pathway-mediated regulation of Stat3 activation is linked to the development of doxorubicin resistance in cancer cell lines

SP600125 overcomes antimitotic drug-resistance in cancer cells by increasing apoptosis with independence of P-gp inhibition

Co-treatment of Salinomycin Sensitizes AZD5363-treated Cancer Cells Through Increased Apoptosis

A novel carbazole derivative, MHY407, sensitizes cancer cells to doxorubicin-, etoposide-, and radiation treatment via DNA damage

Corrigendum to "sensitization of cancer cells through reduction of total akt and downregulation of salinomycin-induced pAkt, pGSk3[beta], pTSC2, and p4EBP1 by Cotreatment with MK-2206"

Erratum to: Thioridazine specifically sensitizes drug-resistant cancer cells through highly increase in apoptosis and P-gp inhibition

A 16b 10MS/s digitally self-calibrated ADC with time constant control

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