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The Role of Heparanase and Sulfatases in the Modification of Heparan Sulfate Proteoglycans within the Tumor Microenvironment and Opportunities for Novel Cancer Therapeutics

Frontiers in oncology, 2014-01, Vol.4, p.195-195

2014

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Quinacrine-Induced Autophagy in Ovarian Cancer Triggers Cathepsin-L Mediated Lysosomal/Mitochondrial Membrane Permeabilization and Cell Death

Cancers, 2021-04, Vol.13 (9), p.2004

2021

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HtrA1 sensitizes ovarian cancer cells to cisplatin-induced cytotoxicity by targeting XIAP for degradation

International journal of cancer, 2012-03, Vol.130 (5), p.1029-1035

2012

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Quinacrine in endometrial cancer: Repurposing an old antimalarial drug

Gynecologic oncology, 2017-07, Vol.146 (1), p.187-195

2017

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Heregulin in breast cancer: old story, new paradigm

Current pharmaceutical design, 2014-01, Vol.20 (30), p.4874-4878

2014

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Coiled-Coil and C2 Domain-Containing Protein 1A (CC2D1A) Promotes Chemotherapy Resistance in Ovarian Cancer

Frontiers in oncology, 2019-10, Vol.9, p.986-986

2019

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Loss of HSulf-1: The Missing Link between Autophagy and Lipid Droplets in Ovarian Cancer

Scientific reports, 2017-02, Vol.7 (1), p.41977, Article 41977

2017

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Metformin is synthetically lethal with glucose withdrawal in cancer cells

Cell cycle (Georgetown, Tex.), 2012-08, Vol.11 (15), p.2779-2779

2012

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Expression and Functional Significance of HtrA1 Loss in Endometrial Cancer

Clinical cancer research, 2011-02, Vol.17 (3), p.427-436

2011

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Regulation of the Ring Finger E3 Ligase Siah2 by p38 MAPK

The Journal of biological chemistry, 2006-11, Vol.281 (46), p.35316-35326

2006

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Regulation of HSulf-1 Expression by Variant Hepatic Nuclear Factor 1 in Ovarian Cancer

Cancer research (Chicago, Ill.), 2009-06, Vol.69 (11), p.4843-4850

2009

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Silencing of HSulf-2 expression in MCF10DCIS.com cells attenuate ductal carcinoma in situ progression to invasive ductal carcinoma in vivo

Breast cancer research : BCR, 2012-03, Vol.14 (2), p.R43-R43, Article R43

2012

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Therapeutic targeting of PFKFB3 with a novel glycolytic inhibitor PFK158 promotes lipophagy and chemosensitivity in gynecologic cancers

International journal of cancer, 2019-01, Vol.144 (1), p.178-189

2019

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Abstract 1937: Quinacrine-induced autophagy in ovarian cancer triggers cathepsin-L mediated lysosomal/mitochondrial membrane permeabilization and cell death

Cancer research (Chicago, Ill.), 2021-07, Vol.81 (13_Supplement), p.1937-1937

2021

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Loss of HSulf-1 promotes altered lipid metabolism in ovarian cancer

Cancer & metabolism, 2014-08, Vol.2 (1), p.13-13, Article 13

2014

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Quinacrine upregulates p21/p27 independent of p53 through autophagy-mediated downregulation of p62-Skp2 axis in ovarian cancer

Scientific reports, 2018-02, Vol.8 (1), p.2487-12, Article 2487

2018

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Involvement of c-Jun N-terminal kinase activities in skeletal muscle differentiation

Journal of muscle research and cell motility, 2004-11, Vol.25 (8), p.645-655

2004

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Quinacrine promotes autophagic cell death and chemosensitivity in ovarian cancer and attenuates tumor growth

Oncotarget, 2015-11, Vol.6 (34), p.36354-36369

2015

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JAMP, a Jun N-Terminal Kinase 1 (JNK1)-Associated Membrane Protein, Regulates Duration of JNK Activity

Molecular and Cellular Biology, 2005-10, Vol.25 (19), p.8619-8630

2005

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Loss of HSulf‐1 expression enhances tumorigenicity by inhibiting Bim expression in ovarian cancer

International journal of cancer, 2014-10, Vol.135 (8), p.1783-1789

2014

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Metformin is synthetically lethal with glucose withdrawal in cancer cells

Cell cycle (Georgetown, Tex.), 2012-08, Vol.11 (15), p.2782-2792

2012

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Abstract 3506: Heregulin induces transformation of mammary epithelial cells via HER3 activation

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (19_Supplement), p.3506-3506

2014

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Loss of HSulf-1 expression enhances tumorigenicity by inhibiting Bim expression in ovarian cancer: Bim is a downstream signaling molecule of HSulf-1

International journal of cancer, 2014-10, Vol.135 (8), p.1783-1789

2014

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Abstract B74: Can we repurpose anti-malarial drug quinacrine to treat ovarian cancer?

Clinical cancer research, 2016-01, Vol.22 (2_Supplement), p.B74-B74

2016

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Abstract 1135: Loss of HSulf-1 promotes defective autophagy and increased lipid droplet biogenesis in ovarian cancer

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.1135-1135

2015

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The Role of Heparanase and Sulfatases in the Modification of Heparan Sulfate Proteoglycans within the Tumor Microenvironment and Opportunities for Novel Cancer Therapeutics

Quinacrine-Induced Autophagy in Ovarian Cancer Triggers Cathepsin-L Mediated Lysosomal/Mitochondrial Membrane Permeabilization and Cell Death

HtrA1 sensitizes ovarian cancer cells to cisplatin-induced cytotoxicity by targeting XIAP for degradation

Quinacrine in endometrial cancer: Repurposing an old antimalarial drug

Heregulin in breast cancer: old story, new paradigm

Coiled-Coil and C2 Domain-Containing Protein 1A (CC2D1A) Promotes Chemotherapy Resistance in Ovarian Cancer

Loss of HSulf-1: The Missing Link between Autophagy and Lipid Droplets in Ovarian Cancer

Metformin is synthetically lethal with glucose withdrawal in cancer cells

Expression and Functional Significance of HtrA1 Loss in Endometrial Cancer

Regulation of the Ring Finger E3 Ligase Siah2 by p38 MAPK

Regulation of HSulf-1 Expression by Variant Hepatic Nuclear Factor 1 in Ovarian Cancer

Silencing of HSulf-2 expression in MCF10DCIS.com cells attenuate ductal carcinoma in situ progression to invasive ductal carcinoma in vivo

Therapeutic targeting of PFKFB3 with a novel glycolytic inhibitor PFK158 promotes lipophagy and chemosensitivity in gynecologic cancers

Abstract 1937: Quinacrine-induced autophagy in ovarian cancer triggers cathepsin-L mediated lysosomal/mitochondrial membrane permeabilization and cell death

Loss of HSulf-1 promotes altered lipid metabolism in ovarian cancer

Quinacrine upregulates p21/p27 independent of p53 through autophagy-mediated downregulation of p62-Skp2 axis in ovarian cancer

Involvement of c-Jun N-terminal kinase activities in skeletal muscle differentiation

Quinacrine promotes autophagic cell death and chemosensitivity in ovarian cancer and attenuates tumor growth

JAMP, a Jun N-Terminal Kinase 1 (JNK1)-Associated Membrane Protein, Regulates Duration of JNK Activity

Loss of HSulf‐1 expression enhances tumorigenicity by inhibiting Bim expression in ovarian cancer

Metformin is synthetically lethal with glucose withdrawal in cancer cells

Abstract 3506: Heregulin induces transformation of mammary epithelial cells via HER3 activation

Loss of HSulf-1 expression enhances tumorigenicity by inhibiting Bim expression in ovarian cancer: Bim is a downstream signaling molecule of HSulf-1

Abstract B74: Can we repurpose anti-malarial drug quinacrine to treat ovarian cancer?

Abstract 1135: Loss of HSulf-1 promotes defective autophagy and increased lipid droplet biogenesis in ovarian cancer

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