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Trans-cellular Propagation of Tau Aggregation by Fibrillar Species

The Journal of biological chemistry, 2012-06, Vol.287 (23), p.19440-19451

2012

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Heparan sulfate proteoglycans mediate internalization and propagation of specific proteopathic seeds

Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (33), p.E3138-E3147

2013

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Reprogramming Medulloblastoma-Propagating Cells by a Combined Antagonism of Sonic Hedgehog and CXCR4

Cancer research (Chicago, Ill.), 2017-03, Vol.77 (6), p.1416-1426

2017

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Novel human neuronal tau model exhibiting neurofibrillary tangles and transcellular propagation

Neurobiology of disease, 2017-10, Vol.106, p.222-234

2017

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Cell-intrinsic, Bmal1-dependent Circadian Regulation of Temozolomide Sensitivity in Glioblastoma

Journal of biological rhythms, 2017-04, Vol.32 (2), p.121-129

2017

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Cooperative p16 and p21 action protects female astrocytes from transformation

Acta neuropathologica communications, 2018-02, Vol.6 (1), p.12-12, Article 12

2018

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Gonadal sex patterns p21-induced cellular senescence in mouse and human glioblastoma

Communications biology, 2022-08, Vol.5 (1), p.781-781, Article 781

2022

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Anti-Tau Antibodies that Block Tau Aggregate Seeding In Vitro Markedly Decrease Pathology and Improve Cognition In Vivo

Neuron (Cambridge, Mass.), 2013-10, Vol.80 (2), p.402-414

2013

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EPCO-29. KLF5 IS REQUIRED FOR THE SEX-DIFFERENCES ACROSS MULTIPLE TUMORIGENIC PHENOTYPES IN PEDIATRIC GLIOBLASTOMA (GBM)

Neuro-oncology (Charlottesville, Va.), 2022-11, Vol.24 (Supplement_7), p.vii122-vii122

2022

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HGG-25. Targeting KLF5 reduces tumorigenic phenotype in a murine glioblastoma (GBM) model

Neuro-oncology (Charlottesville, Va.), 2022-06, Vol.24 (Supplement_1), p.i66-i66

2022

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CBIO-08. ASTROCYTE SENESCENCE CONTRIBUTES TO SEX DIFFERENCES IN GLIOBLASTOMA INCIDENCE AND OUTCOME

Neuro-oncology (Charlottesville, Va.), 2020-11, Vol.22 (Supplement_2), p.ii17-ii17

2020

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Brd4-bound enhancers drive cell-intrinsic sex differences in glioblastoma

Proceedings of the National Academy of Sciences - PNAS, 2021-04, Vol.118 (16), p.1

2021

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CBIO-36p21 REGULATES STEM CELL FREQUENCY IN A SEX-SPECIFIC FASHION

Neuro-oncology (Charlottesville, Va.), 2015-11, Vol.17 (suppl 5), p.v62.3-v62

2015

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TMIC-34. SENESCENT ASTROCYTE INDUCED INFLAMMATION MAY UNDERLIE SEX DIFFERENCES IN THE AGE-DEPENDENT RISE IN GLIOBLASTOMA INCIDENCE

Neuro-oncology (Charlottesville, Va.), 2019-11, Vol.21 (Supplement_6), p.vi255-vi255

2019

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TMIC-17. ASTROCYTE SENESCENCE CONTRIBUTES TO SEX DIFFERENCES IN THE AGE-RELATED INCREASE IN GLIOBLASTOMA INCIDENCE

Neuro-oncology (Charlottesville, Va.), 2019-11, Vol.21 (Supplement_6), p.vi250-vi251

2019

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Identification of neuronal target genes for CCAAT/Enhancer Binding Proteins

Molecular and cellular neuroscience, 2009-03, Vol.40 (3), p.313-327

2009

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Abstract B011: Sex-specific chromatin remodeling drives tumorigenesis in glioblastoma

Cancer research (Chicago, Ill.), 2024-03, Vol.84 (5_Supplement_1), p.B011-B011

2024

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GENT-18. DIFFERENTIAL BRD4-BOUND STRETCH/SUPER ENHANCERS DRIVE CRITICAL SEX DIFFERENCES IN GBM PHENOTYPE

Neuro-oncology (Charlottesville, Va.), 2016-11, Vol.18 (suppl_6), p.vi77-vi77

2016

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GENE-16. Brd4-BOUND STRETCH/SUPER ENHANCER USAGE UNDERLIES SEXUAL DIMORPHISM IN GLIOBLASTOMA

Neuro-oncology (Charlottesville, Va.), 2017-11, Vol.19 (suppl_6), p.vi95-vi95

2017

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CBIO-31. SEX DIFFERENCES IN THE ACTIVITY OF CYCLIN DEPENDENT KINASE INHIBITORS UNDERLIE GREATER THRESHOLD FOR GLIOMIC TRANSFORMATION IN FEMALE ASTROCYTES

Neuro-oncology (Charlottesville, Va.), 2017-11, Vol.19 (suppl_6), p.vi38-vi38

2017

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Abstract 3658: Brd4-bound enhancers drive critical sex differences in glioblastoma

Cancer research (Chicago, Ill.), 2020-08, Vol.80 (16_Supplement), p.3658-3658

2020

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Heparan sulfate proteoglycans mediate internalization and propagation of specific proteopathic seeds

Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (33), p.13243-13243

2013

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Anti-Tau Antibodies that Block Tau Aggregate Seeding In Vitro Markedly Decrease Pathology and Improve Cognition In Vivo

Neuron (Cambridge, Mass.), 2013-12, Vol.80 (6), p.1572-1572

2013

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Quantification and assessment of viability of Cryptococcus neoformans by LightCycler amplification of capsule gene mRNA

Journal of medical microbiology, 2004-12, Vol.53 (12), p.1201-1206

2004

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Differential target gene recruitment and cellular distribution of CCAAT/Enhancer binding proteins α, β and δ in the rodent brain

Kfoury, Najla A

2009

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Trans-cellular Propagation of Tau Aggregation by Fibrillar Species

Heparan sulfate proteoglycans mediate internalization and propagation of specific proteopathic seeds

Reprogramming Medulloblastoma-Propagating Cells by a Combined Antagonism of Sonic Hedgehog and CXCR4

Novel human neuronal tau model exhibiting neurofibrillary tangles and transcellular propagation

Cell-intrinsic, Bmal1-dependent Circadian Regulation of Temozolomide Sensitivity in Glioblastoma

Cooperative p16 and p21 action protects female astrocytes from transformation

Gonadal sex patterns p21-induced cellular senescence in mouse and human glioblastoma

Anti-Tau Antibodies that Block Tau Aggregate Seeding In Vitro Markedly Decrease Pathology and Improve Cognition In Vivo

EPCO-29. KLF5 IS REQUIRED FOR THE SEX-DIFFERENCES ACROSS MULTIPLE TUMORIGENIC PHENOTYPES IN PEDIATRIC GLIOBLASTOMA (GBM)

HGG-25. Targeting KLF5 reduces tumorigenic phenotype in a murine glioblastoma (GBM) model

CBIO-08. ASTROCYTE SENESCENCE CONTRIBUTES TO SEX DIFFERENCES IN GLIOBLASTOMA INCIDENCE AND OUTCOME

Brd4-bound enhancers drive cell-intrinsic sex differences in glioblastoma

CBIO-36p21 REGULATES STEM CELL FREQUENCY IN A SEX-SPECIFIC FASHION

TMIC-34. SENESCENT ASTROCYTE INDUCED INFLAMMATION MAY UNDERLIE SEX DIFFERENCES IN THE AGE-DEPENDENT RISE IN GLIOBLASTOMA INCIDENCE

TMIC-17. ASTROCYTE SENESCENCE CONTRIBUTES TO SEX DIFFERENCES IN THE AGE-RELATED INCREASE IN GLIOBLASTOMA INCIDENCE

Identification of neuronal target genes for CCAAT/Enhancer Binding Proteins

Abstract B011: Sex-specific chromatin remodeling drives tumorigenesis in glioblastoma

GENT-18. DIFFERENTIAL BRD4-BOUND STRETCH/SUPER ENHANCERS DRIVE CRITICAL SEX DIFFERENCES IN GBM PHENOTYPE

GENE-16. Brd4-BOUND STRETCH/SUPER ENHANCER USAGE UNDERLIES SEXUAL DIMORPHISM IN GLIOBLASTOMA

CBIO-31. SEX DIFFERENCES IN THE ACTIVITY OF CYCLIN DEPENDENT KINASE INHIBITORS UNDERLIE GREATER THRESHOLD FOR GLIOMIC TRANSFORMATION IN FEMALE ASTROCYTES

Abstract 3658: Brd4-bound enhancers drive critical sex differences in glioblastoma

Heparan sulfate proteoglycans mediate internalization and propagation of specific proteopathic seeds

Anti-Tau Antibodies that Block Tau Aggregate Seeding In Vitro Markedly Decrease Pathology and Improve Cognition In Vivo

Quantification and assessment of viability of Cryptococcus neoformans by LightCycler amplification of capsule gene mRNA

Differential target gene recruitment and cellular distribution of CCAAT/Enhancer binding proteins α, β and δ in the rodent brain

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