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Cathepsin B and its endogenous inhibitor cystatin C in rheumatoid arthritis synovium

Journal of rheumatology, 2000-04, Vol.27 (4), p.859

2000

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Expression of vascular cell adhesion molecule-1 mRNA and protein in rheumatoid synovium demonstrated by in situ hybridization and immunohistochemistry

Laboratory investigation, 1995-02, Vol.72 (2), p.209

1995

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Detection of insulin-like growth factor I and II in synovial tissue specimens of patients with rheumatoid arthritis and osteoarthritis by in situ hybridization

Journal of rheumatology, 1995-02, Vol.22 (2), p.275

1995

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Inhibition of RNA polymerases by digoxigenin-labeled UTP

BioTechniques, 1994-01, Vol.16 (1), p.54

1994

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Expression of Cysteine Proteinases Cathepsins B and K and of Cysteine Proteinase Inhibitor Cystatin C in Giant Cell Tumor of Tendon Sheath

Modern pathology, 2001-04, Vol.14 (4), p.318-324

2001

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Cytokines and oncogenes in cellular interactions of rheumatoid arthritis

Stem cells (Dayton, Ohio), 1994-01, Vol.12 (1), p.75

1994

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A new model for rheumatoid arthritis generated by engraftment of rheumatoid synovial tissue and normal human cartilage into scid mice

Arthritis and rheumatism, 1994-11, Vol.37 (11), p.1664-1671

1994

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Comparative analysis of cathepsin l, cathepsin d, and collagenase messenger rna expression in synovial tissues of patients with rheumatoid arthritis and osteoarthritis, by in situ hybridization

Arthritis and rheumatism, 1995-07, Vol.38 (7), p.976-984

1995

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Expression of e‐selectin messenger RNA and protein in rheumatoid arthritis

Arthritis and rheumatism, 1995-06, Vol.38 (6), p.750-754

1995

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Cathepsin K Is a Critical Protease in Synovial Fibroblast-Mediated Collagen Degradation

The American journal of pathology, 2001-12, Vol.159 (6), p.2167-2177

2001

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Circulating levels of matrix metalloproteinases MMP-3 and MMP-1, tissue inhibitor of metalloproteinases 1 (TIMP-1), and MMP-1/TIMP-1 complex in rheumatic disease. Correlation with clinical activity of rheumatoid arthritis versus other surrogate markers

Journal of rheumatology, 1999-02, Vol.26 (2), p.251

1999

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Neurovascular mechanisms as a possible cause of remission of rheumatoid arthritis in hemiparetic limbs

Annals of the rheumatic diseases, 2004-10, Vol.63 (10), p.1349-1351

2004

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Artificial tissues in perfusion culture

International journal of artificial organs, 1997-01, Vol.20 (1), p.57

1997

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Efficacy and safety of a combination therapy of methotrexate, chloroquine and cyclophosphamide in patients with refractory rheumatoid arthritis: results of an observational study with matched-pair analysis

Clinical rheumatology, 1999-01, Vol.18 (2), p.145-151

1999

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Matrix metalloproteinases, but not cathepsins B, H, and L or their inhibitors in peripheral blood of patients with rheumatoid arthritis are potentially useful markers of disease activity

Zeitschrift für Rheumatologie, 1998-12, Vol.57 (6), p.392-398

1998

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Comparison of cathepsins K and S expression within the rheumatoid and osteoarthritic synovium

Arthritis and rheumatism, 2002-03, Vol.46 (3), p.663-674

2002

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Differential expression of cathepsins B and L compared with matrix metalloproteinases and their respective inhibitors in rheumatoid arthritis and osteoarthritis: A parallel investigation by semiquantitative reverse transcriptase‐polymerase chain reaction and immunohistochemistry

Arthritis and rheumatism, 1998-08, Vol.41 (8), p.1378-1387

1998

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Development of in vitro model systems for destructive joint diseases. Novel strategies for establishing inflammatory pannus

Arthritis and rheumatism, 1997-08, Vol.40 (8), p.1420-1428

1997

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Neurovascular mechanisms as a possible cause of remission of rheumatoid arthritis in hemiparetic limbs

Annals of the rheumatic diseases, 2004, Vol.63 (10), p.1349-1351

2004

Volltextzugriff (PDF)

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Cathepsin B and its endogenous inhibitor cystatin C in rheumatoid arthritis synovium

Expression of vascular cell adhesion molecule-1 mRNA and protein in rheumatoid synovium demonstrated by in situ hybridization and immunohistochemistry

Detection of insulin-like growth factor I and II in synovial tissue specimens of patients with rheumatoid arthritis and osteoarthritis by in situ hybridization

Inhibition of RNA polymerases by digoxigenin-labeled UTP

Expression of Cysteine Proteinases Cathepsins B and K and of Cysteine Proteinase Inhibitor Cystatin C in Giant Cell Tumor of Tendon Sheath

Cytokines and oncogenes in cellular interactions of rheumatoid arthritis

A new model for rheumatoid arthritis generated by engraftment of rheumatoid synovial tissue and normal human cartilage into scid mice

Comparative analysis of cathepsin l, cathepsin d, and collagenase messenger rna expression in synovial tissues of patients with rheumatoid arthritis and osteoarthritis, by in situ hybridization

Expression of e‐selectin messenger RNA and protein in rheumatoid arthritis

Cathepsin K Is a Critical Protease in Synovial Fibroblast-Mediated Collagen Degradation

Circulating levels of matrix metalloproteinases MMP-3 and MMP-1, tissue inhibitor of metalloproteinases 1 (TIMP-1), and MMP-1/TIMP-1 complex in rheumatic disease. Correlation with clinical activity of rheumatoid arthritis versus other surrogate markers

Neurovascular mechanisms as a possible cause of remission of rheumatoid arthritis in hemiparetic limbs

Artificial tissues in perfusion culture

Efficacy and safety of a combination therapy of methotrexate, chloroquine and cyclophosphamide in patients with refractory rheumatoid arthritis: results of an observational study with matched-pair analysis

Matrix metalloproteinases, but not cathepsins B, H, and L or their inhibitors in peripheral blood of patients with rheumatoid arthritis are potentially useful markers of disease activity

Comparison of cathepsins K and S expression within the rheumatoid and osteoarthritic synovium

Differential expression of cathepsins B and L compared with matrix metalloproteinases and their respective inhibitors in rheumatoid arthritis and osteoarthritis: A parallel investigation by semiquantitative reverse transcriptase‐polymerase chain reaction and immunohistochemistry

Development of in vitro model systems for destructive joint diseases. Novel strategies for establishing inflammatory pannus

Neurovascular mechanisms as a possible cause of remission of rheumatoid arthritis in hemiparetic limbs

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