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The role of MET in chemotherapy resistance

Oncogene, 2021-03, Vol.40 (11), p.1927-1941

2021

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Receptor trafficking controls weak signal delivery: a strategy used by c-Met for STAT3 nuclear accumulation

The Journal of cell biology, 2008-09, Vol.182 (5), p.855-863

2008

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Rationale for Cotargeting Hepatocyte Growth Factor and Epidermal Growth Factor Receptor in Recurrent/Metastatic Head and Neck Cancer

Journal of clinical oncology, 2023-08, Vol.41 (22), p.3863-3866

Kermorgant, Stéphanie

2023

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Met endosomal signalling: In the right place, at the right time

The international journal of biochemistry & cell biology, 2014-04, Vol.49, p.69-74

2014

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c-Met-integrin cooperation: Mechanisms, tumorigenic effects, and therapeutic relevance

Frontiers in cell and developmental biology, 2022-10, Vol.10, p.994528-994528

2022

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The Role of PI3K in Met Driven Cancer: A Recap

Frontiers in molecular biosciences, 2018-10, Vol.5, p.86-86

2018

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Receptor tyrosine kinase c-Met controls the cytoskeleton from different endosomes via different pathways

Nature communications, 2014-05, Vol.5 (1), p.3907-3907, Article 3907

2014

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PKC and the control of localized signal dynamics

Nature reviews. Molecular cell biology, 2010-02, Vol.11 (2), p.103-112

2010

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Hepatocyte growth factor induces colonic cancer cell invasiveness via enhanced motility and protease overproduction. Evidence for PI3 kinase and PKC involvement

Carcinogenesis (New York), 2001-07, Vol.22 (7), p.1035-1042

2001

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Beta 1-integrin-c-Met cooperation reveals an inside-in survival signalling on autophagy-related endomembranes

Nature communications, 2016-06, Vol.7 (1), p.11942-18, Article 11942

2016

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A direct role for Met endocytosis in tumorigenesis

Nature cell biology, 2011-07, Vol.13 (7), p.827-837

2011

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Unconventional role of RAC1 in MET-driven anchorage-independent tumor growth

Molecular & cellular oncology, 2020-11, Vol.7 (6), p.1803029-1803029

2020

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Correction: Corrigendum: Beta 1-integrin–c-Met cooperation reveals an inside-in survival signalling on autophagy-related endomembranes

Nature communications, 2016-07, Vol.7 (1), p.1-1, Article 12392

2016

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A PI3K- and GTPase-independent Rac1-mTOR mechanism mediates MET-driven anchorage-independent cell growth but not migration

Science signaling, 2020-06, Vol.13 (637)

2020

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Leptin and Ob-Rb Receptor Isoform in the Human Digestive Tract during Fetal Development

The journal of clinical endocrinology and metabolism, 2005-11, Vol.90 (11), p.6177-6184

2005

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ERK2 but not ERK1 mediates HGF-induced motility in non-small cell lung carcinoma cell lines

Journal of cell science, 2013-06, Vol.126 (Pt 11), p.2381-2391

2013

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PKC controls HGF-dependent c-Met traffic, signalling and cell migration

The EMBO journal, 2004-09, Vol.23 (19), p.3721-3734

2004

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C‐Met in invasive breast cancer

Cancer, 2014-01, Vol.120 (2), p.163-171

2014

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The stomach is a source of leptin

Nature (London), 1998-08, Vol.394 (6695), p.790-793

1998

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Matrix metalloproteinase inhibition prevents colon cancer peritoneal carcinomatosis development and prolongs survival in rats

Carcinogenesis (New York), 1999-08, Vol.20 (8), p.1445-1452

1999

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Protein Kinase C Controls Microtubule-based Traffic but Not Proteasomal Degradation of c-Met

The Journal of biological chemistry, 2003-08, Vol.278 (31), p.28921-28929

2003

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Anomalous inhibition of c-Met by the kinesin inhibitor aurintricarboxylic acid

International journal of cancer, 2012-03, Vol.130 (5), p.1060-1070

2012

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The clinical and functional significance of c-Met in breast cancer: a review

Breast cancer research : BCR, 2015-04, Vol.17 (1), p.52, Article 52

2015

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Abstract B26: PI3K class I and mTOR regulate distinct steps in Met dependent tumorigenesis

Molecular cancer therapeutics, 2015-07, Vol.14 (7_Supplement), p.B26-B26

2015

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The role of MET in chemotherapy resistance

Receptor trafficking controls weak signal delivery: a strategy used by c-Met for STAT3 nuclear accumulation

Rationale for Cotargeting Hepatocyte Growth Factor and Epidermal Growth Factor Receptor in Recurrent/Metastatic Head and Neck Cancer

Met endosomal signalling: In the right place, at the right time

c-Met-integrin cooperation: Mechanisms, tumorigenic effects, and therapeutic relevance

The Role of PI3K in Met Driven Cancer: A Recap

Receptor tyrosine kinase c-Met controls the cytoskeleton from different endosomes via different pathways

PKC and the control of localized signal dynamics

Hepatocyte growth factor induces colonic cancer cell invasiveness via enhanced motility and protease overproduction. Evidence for PI3 kinase and PKC involvement

Beta 1-integrin-c-Met cooperation reveals an inside-in survival signalling on autophagy-related endomembranes

A direct role for Met endocytosis in tumorigenesis

Unconventional role of RAC1 in MET-driven anchorage-independent tumor growth

Correction: Corrigendum: Beta 1-integrin–c-Met cooperation reveals an inside-in survival signalling on autophagy-related endomembranes

A PI3K- and GTPase-independent Rac1-mTOR mechanism mediates MET-driven anchorage-independent cell growth but not migration

Leptin and Ob-Rb Receptor Isoform in the Human Digestive Tract during Fetal Development

ERK2 but not ERK1 mediates HGF-induced motility in non-small cell lung carcinoma cell lines

PKC controls HGF-dependent c-Met traffic, signalling and cell migration

C‐Met in invasive breast cancer

The stomach is a source of leptin

Matrix metalloproteinase inhibition prevents colon cancer peritoneal carcinomatosis development and prolongs survival in rats

MET exon 14 skipping mutation is a hepatocyte growth factor (HGF)-dependent oncogenic driver in vitro and in humanised HGF knock-in mice

Protein Kinase C Controls Microtubule-based Traffic but Not Proteasomal Degradation of c-Met

Anomalous inhibition of c-Met by the kinesin inhibitor aurintricarboxylic acid

The clinical and functional significance of c-Met in breast cancer: a review

Abstract B26: PI3K class I and mTOR regulate distinct steps in Met dependent tumorigenesis

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