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Cardiac-Specific Elevations in Thyroid Hormone Enhance Contractility and Prevent Pressure Overload-Induced Cardiac Dysfunction

Proceedings of the National Academy of Sciences - PNAS, 2006-04, Vol.103 (15), p.6043-6048

2006

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The role of phosphoinositide-3 kinase and PTEN in cardiovascular physiology and disease

Journal of Molecular and Cellular Cardiology, 2004-08, Vol.37 (2), p.449-471

2004

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PI3Kγ Is Required for PDE4, not PDE3, Activity in Subcellular Microdomains Containing the Sarcoplasmic Reticular Calcium ATPase in Cardiomyocytes

Circulation research, 2007-08, Vol.101 (4), p.400-408

2007

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Enhanced Late Na+Currents in Atrial Fibrillation: New Drug Target or Just an Epiphenomenon?

Journal of the American College of Cardiology, 2010-05, Vol.55 (21), p.2343-2345

2010

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Mineralocorticoid receptor antagonism prevents the electrical remodeling that precedes cellular hypertrophy after myocardial infarction

Circulation (New York, N.Y.), 2004-08, Vol.110 (7), p.776-783

2004

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Leaky ryanodine receptors in the failing heart: the root of all evil?

Cardiovascular research, 2011-06, Vol.90 (3), p.399-401

2011

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Enhanced Late Na+ Currents in Atrial Fibrillation

Journal of the American College of Cardiology, 2010-05, Vol.55 (21), p.2343-2345

2010

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Tachycardia-induced silencing of subcellular Ca2+ signaling in atrial myocytes

The Journal of clinical investigation, 2014-11, Vol.124 (11), p.4759-4772

2014

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Phosphoinositide 3-kinase γ Regulates Cardiac Contractility by Locally Controlling Cyclic Adenosine Monophosphate Levels

Trends in cardiovascular medicine, 2006-10, Vol.16 (7), p.250-256

2006

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Cardiac sarcoplasmic reticulum calcium release and load are enhanced by subcellular cAMP elevations in PI3Kγ-deficient mice

Circulation research, 2005-05, Vol.96 (10), p.1079-1086

2005

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Rabbit, a relevant model for the study of cardiac β3‐adrenoceptors

Experimental physiology, 2009-04, Vol.94 (4), p.400-411

2009

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Rabbit, a relevant model for the study of cardiac beta 3-adrenoceptors

Experimental physiology, 2009-04, Vol.94 (4), p.400-411

2009

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Autonomic regulation of calcium and potassium channels is oppositely modulated by microtubules in cardiac myocytes

American journal of physiology. Heart and circulatory physiology, 2004-06, Vol.55 (6), p.2065-2071

2004

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Autonomic regulation of calcium and potassium channels is oppositely modulated by microtubules in cardiac myocytes

American journal of physiology. Heart and circulatory physiology, 2004-06, Vol.286 (6), p.H2065-H2071

2004

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Enhanced Late Na+ Currents in Atrial Fibrillation: New Drug Target or Just an Epiphenomenon?Editorials published in the Journal of the American College of Cardiology reflect the views of the authors and do not necessarily represent the views of JACC or the American College of Cardiology

Journal of the American College of Cardiology, 2010-05, Vol.55 (21), p.2343

2010

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Tachycardia-induced silencing of subcellular [Ca.sup.2+] signaling in atrial myocytes

The Journal of clinical investigation, 2014-11, Vol.124 (11), p.4759

2014

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Tachycardia-induced silencing of subcellular Ca super( 2+) signaling in atrial myocytes

The Journal of clinical investigation, 2014-11, Vol.124 (11), p.4759-4759

2014

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Altered Nuclear Calcium Signaling in Tachycardia-Induced Remodeling in Rabbit Atria: A Mechanism of Altered Excitation-Transcription Coupling in Atrial Fibrillation?

Biophysical journal, 2011-02, Vol.100 (3), p.517a-517a

2011

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Insulin-Like Growth Factor-1 and PTEN Deletion Enhance Cardiac L-Type Ca2+ Currents via Increased PI3Kα/PKB Signaling

Circulation research, 2006-06, Vol.98 (11), p.1390-1397

2006

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Cardiac Sarcoplasmic Reticulum Calcium Release and Load Are Enhanced by Subcellular cAMP Elevations in PI3Kγ-Deficient Mice

Circulation research, 2005-05, Vol.96 (10), p.1079-1086

2005

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PS9 - 41. Translocation of substrate transporters glut4 and cd36 to the sarcolemma and subsequent activation to increase substrate uptake are separate events

Nederlands Tijdschrift voor Diabetolog, 2012-11, Vol.10 (3), p.127-127

2012

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Insulin-Like Growth Factor-1 and PTEN Deletion Enhance Cardiac L-Type Ca 2+ Currents via Increased PI3Kα/PKB Signaling

Circulation research, 2006-06, Vol.98 (11), p.1390-1397

2006

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Insulin-Like Growth Factor-1 and PTEN Deletion Enhance Cardiac L-Type Ca super(2+) Currents via Increased PI3K alpha /PKB Signaling

Circulation research, 2006-06, Vol.98 (11), p.1390-1397

2006

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Phosphoinositide 3-kinase [gamma] Regulates Cardiac Contractility by Locally Controlling Cyclic Adenosine Monophosphate Levels

Trends in cardiovascular medicine, 2006-10, Vol.16 (7), p.250

2006

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Tachycardia-induced silencing of subcellular Ca^sup 2+^ signaling in atrial myocytes

The Journal of clinical investigation, 2014-11, Vol.124 (11), p.4759

2014

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Cardiac-Specific Elevations in Thyroid Hormone Enhance Contractility and Prevent Pressure Overload-Induced Cardiac Dysfunction

The role of phosphoinositide-3 kinase and PTEN in cardiovascular physiology and disease

PI3Kγ Is Required for PDE4, not PDE3, Activity in Subcellular Microdomains Containing the Sarcoplasmic Reticular Calcium ATPase in Cardiomyocytes

Enhanced Late Na+Currents in Atrial Fibrillation: New Drug Target or Just an Epiphenomenon?

Mineralocorticoid receptor antagonism prevents the electrical remodeling that precedes cellular hypertrophy after myocardial infarction

Leaky ryanodine receptors in the failing heart: the root of all evil?

Enhanced Late Na+ Currents in Atrial Fibrillation

Tachycardia-induced silencing of subcellular Ca2+ signaling in atrial myocytes

Phosphoinositide 3-kinase γ Regulates Cardiac Contractility by Locally Controlling Cyclic Adenosine Monophosphate Levels

Cardiac sarcoplasmic reticulum calcium release and load are enhanced by subcellular cAMP elevations in PI3Kγ-deficient mice

Rabbit, a relevant model for the study of cardiac β3‐adrenoceptors

Rabbit, a relevant model for the study of cardiac beta 3-adrenoceptors

Autonomic regulation of calcium and potassium channels is oppositely modulated by microtubules in cardiac myocytes

Autonomic regulation of calcium and potassium channels is oppositely modulated by microtubules in cardiac myocytes

Enhanced Late Na+ Currents in Atrial Fibrillation: New Drug Target or Just an Epiphenomenon?Editorials published in the Journal of the American College of Cardiology reflect the views of the authors and do not necessarily represent the views of JACC or the American College of Cardiology

Tachycardia-induced silencing of subcellular [Ca.sup.2+] signaling in atrial myocytes

Tachycardia-induced silencing of subcellular Ca super( 2+) signaling in atrial myocytes

Altered Nuclear Calcium Signaling in Tachycardia-Induced Remodeling in Rabbit Atria: A Mechanism of Altered Excitation-Transcription Coupling in Atrial Fibrillation?

Insulin-Like Growth Factor-1 and PTEN Deletion Enhance Cardiac L-Type Ca2+ Currents via Increased PI3Kα/PKB Signaling

Cardiac Sarcoplasmic Reticulum Calcium Release and Load Are Enhanced by Subcellular cAMP Elevations in PI3Kγ-Deficient Mice

PS9 - 41. Translocation of substrate transporters glut4 and cd36 to the sarcolemma and subsequent activation to increase substrate uptake are separate events

Insulin-Like Growth Factor-1 and PTEN Deletion Enhance Cardiac L-Type Ca 2+ Currents via Increased PI3Kα/PKB Signaling

Insulin-Like Growth Factor-1 and PTEN Deletion Enhance Cardiac L-Type Ca super(2+) Currents via Increased PI3K alpha /PKB Signaling

Phosphoinositide 3-kinase [gamma] Regulates Cardiac Contractility by Locally Controlling Cyclic Adenosine Monophosphate Levels

Tachycardia-induced silencing of subcellular Ca^sup 2+^ signaling in atrial myocytes

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