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Increased myofilament Ca2+ sensitivity and diastolic dysfunction as early consequences of Mybpc3 mutation in heterozygous knock-in mice

Journal of molecular and cellular cardiology, 2012-06, Vol.52 (6), p.1299-1307

2012

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Oxidant-induced Activation of Type I Protein Kinase A Is Mediated by RI Subunit Interprotein Disulfide Bond Formation

The Journal of biological chemistry, 2006-08, Vol.281 (31), p.21827-21836

2006

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Distinct Sarcomeric Substrates Are Responsible for Protein Kinase D-mediated Regulation of Cardiac Myofilament Ca2+ Sensitivity and Cross-bridge Cycling

The Journal of biological chemistry, 2010-02, Vol.285 (8), p.5674-5682

2010

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Right Ventricle Has Normal Myofilament Function But Shows Perturbations in the Expression of Extracellular Matrix Genes in Patients With Tetralogy of Fallot Undergoing Pulmonary Valve Replacement

Journal of the American Heart Association, 2020-08, Vol.9 (16), p.e015342-e015342

2020

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Protein Kinase D Selectively Targets Cardiac Troponin I and Regulates Myofilament Ca2+ Sensitivity in Ventricular Myocytes

Circulation research, 2007-03, Vol.100 (6), p.864-873

2007

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Phosphorylation of Troponin I by Protein Kinase A Accelerates Relaxation and Crossbridge Cycle Kinetics in Mouse Ventricular Muscle

Circulation research, 2001-05, Vol.88 (10), p.1059-1065

2001

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A model of cardiac contraction based on novel measurements of tension development in human cardiomyocytes

Journal of molecular and cellular cardiology, 2017-05, Vol.106, p.68-83

2017

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Reduced First-Phase Ejection Fraction and Sustained Myocardial Wall Stress in Hypertensive Patients With Diastolic Dysfunction: A Manifestation of Impaired Shortening Deactivation That Links Systolic to Diastolic Dysfunction and Preserves Systolic Ejection Fraction

Hypertension (Dallas, Tex. 1979), 2017-04, Vol.69 (4), p.633-640

2017

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Changes in force and cytosolic Ca2+ concentration after length changes in isolated rat ventricular trabeculae

The Journal of physiology, 1998-01, Vol.506 (2), p.431-444

1998

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Enhanced length-dependent Ca2+ activation in fish cardiomyocytes permits a large operating range of sarcomere lengths

Journal of molecular and cellular cardiology, 2010-05, Vol.48 (5), p.917-924

2010

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Esmolol cardioplegia: the cellular mechanism of diastolic arrest

Cardiovascular research, 2010-08, Vol.87 (3), p.552-560

2010

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Passive Stiffness of Myocardium From Congenital Heart Disease and Implications for Diastole

Circulation (New York, N.Y.), 2010-03, Vol.121 (8), p.979-988

2010

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Monophosphorylation of cardiac troponin-I at Ser-23/24 is sufficient to regulate cardiac myofibrillar Ca2+ sensitivity and calpain-induced proteolysis

The Journal of biological chemistry, 2018-06, Vol.293 (22), p.8588-8599

2018

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Novel Role for p90 Ribosomal S6 Kinase in the Regulation of Cardiac Myofilament Phosphorylation

The Journal of biological chemistry, 2011-02, Vol.286 (7), p.5300-5310

2011

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cMyBP-C as a promiscuous substrate: phosphorylation by non-PKA kinases and its potential significance

Journal of muscle research and cell motility, 2012-05, Vol.33 (1), p.53-60

2012

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Activation of Myocardial Contraction by the N-Terminal Domains of Myosin Binding Protein-C

Circulation research, 2006-05, Vol.98 (10), p.1290-1298

2006

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Normal passive viscoelasticity but abnormal myofibrillar force generation in human hypertrophic cardiomyopathy

Journal of molecular and cellular cardiology, 2010-11, Vol.49 (5), p.737-745

2010

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Protein Kinase D Is a Novel Mediator of Cardiac Troponin I Phosphorylation and Regulates Myofilament Function

Circulation research, 2004-11, Vol.95 (11), p.1091-1099

2004

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Myofilament-based relaxant effect of isoprenaline revealed during work-loop contractions in rat cardiac trabeculae

The Journal of physiology, 2002-10, Vol.544 (1), p.171-182

2002

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A Role for the Sarcolemmal Na+/H+ Exchanger in the Slow Force Response to Myocardial Stretch

Circulation research, 1999-10, Vol.85 (8), p.658-660

Kentish, Jonathan C

1999

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Protection against endotoxemia-induced contractile dysfunction in mice with cardiac-specific expression of slow skeletal troponin I

The FASEB journal, 2005-07, Vol.19 (9), p.1137-1139

2005

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Targeting for cardioplegia: arresting agents and their safety

Current opinion in pharmacology, 2009-04, Vol.9 (2), p.220-226

2009

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Functional analysis of a unique troponin c mutation, GLY159ASP, that causes familial dilated cardiomyopathy, studied in explanted heart muscle

Circulation. Heart failure, 2009-09, Vol.2 (5), p.456-464

2009

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Protein phosphatase 2A contributes to the cardiac dysfunction induced by endotoxemia

Cardiovascular research, 2009-04, Vol.82 (1), p.67-76

2009

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From genotype to phenotype: a longitudinal study of a patient with hypertrophic cardiomyopathy due to a mutation in the MYBPC3 gene

Journal of muscle research and cell motility, 2008-12, Vol.29 (6-8), p.239-246

2008

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Increased myofilament Ca2+ sensitivity and diastolic dysfunction as early consequences of Mybpc3 mutation in heterozygous knock-in mice

Oxidant-induced Activation of Type I Protein Kinase A Is Mediated by RI Subunit Interprotein Disulfide Bond Formation

Distinct Sarcomeric Substrates Are Responsible for Protein Kinase D-mediated Regulation of Cardiac Myofilament Ca2+ Sensitivity and Cross-bridge Cycling

Right Ventricle Has Normal Myofilament Function But Shows Perturbations in the Expression of Extracellular Matrix Genes in Patients With Tetralogy of Fallot Undergoing Pulmonary Valve Replacement

Protein Kinase D Selectively Targets Cardiac Troponin I and Regulates Myofilament Ca2+ Sensitivity in Ventricular Myocytes

Phosphorylation of Troponin I by Protein Kinase A Accelerates Relaxation and Crossbridge Cycle Kinetics in Mouse Ventricular Muscle

A model of cardiac contraction based on novel measurements of tension development in human cardiomyocytes

Reduced First-Phase Ejection Fraction and Sustained Myocardial Wall Stress in Hypertensive Patients With Diastolic Dysfunction: A Manifestation of Impaired Shortening Deactivation That Links Systolic to Diastolic Dysfunction and Preserves Systolic Ejection Fraction

Changes in force and cytosolic Ca2+ concentration after length changes in isolated rat ventricular trabeculae

Enhanced length-dependent Ca2+ activation in fish cardiomyocytes permits a large operating range of sarcomere lengths

Esmolol cardioplegia: the cellular mechanism of diastolic arrest

Passive Stiffness of Myocardium From Congenital Heart Disease and Implications for Diastole

Monophosphorylation of cardiac troponin-I at Ser-23/24 is sufficient to regulate cardiac myofibrillar Ca2+ sensitivity and calpain-induced proteolysis

Novel Role for p90 Ribosomal S6 Kinase in the Regulation of Cardiac Myofilament Phosphorylation

cMyBP-C as a promiscuous substrate: phosphorylation by non-PKA kinases and its potential significance

Activation of Myocardial Contraction by the N-Terminal Domains of Myosin Binding Protein-C

Normal passive viscoelasticity but abnormal myofibrillar force generation in human hypertrophic cardiomyopathy

Protein Kinase D Is a Novel Mediator of Cardiac Troponin I Phosphorylation and Regulates Myofilament Function

Myofilament-based relaxant effect of isoprenaline revealed during work-loop contractions in rat cardiac trabeculae

A Role for the Sarcolemmal Na+/H+ Exchanger in the Slow Force Response to Myocardial Stretch

Protection against endotoxemia-induced contractile dysfunction in mice with cardiac-specific expression of slow skeletal troponin I

Targeting for cardioplegia: arresting agents and their safety

Functional analysis of a unique troponin c mutation, GLY159ASP, that causes familial dilated cardiomyopathy, studied in explanted heart muscle

Protein phosphatase 2A contributes to the cardiac dysfunction induced by endotoxemia

From genotype to phenotype: a longitudinal study of a patient with hypertrophic cardiomyopathy due to a mutation in the MYBPC3 gene

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