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Regulation of the latent-lytic switch in Epstein–Barr virus

Seminars in cancer biology, 2014-06, Vol.26, p.60-68

2014

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PD-1/CTLA-4 Blockade Inhibits Epstein-Barr Virus-Induced Lymphoma Growth in a Cord Blood Humanized-Mouse Model

PLoS pathogens, 2016-05, Vol.12 (5), p.e1005642-e1005642

2016

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A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infection

PLoS pathogens, 2018-07, Vol.14 (7), p.e1007179

2018

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Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells

PLoS pathogens, 2022-04, Vol.18 (4), p.e1010453-e1010453

2022

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Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

PLoS pathogens, 2022-10, Vol.18 (10), p.e1010868

2022

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Rta is the principal activator of Epstein-Barr virus epithelial lytic transcription

PLoS pathogens, 2022-09, Vol.18 (9), p.e1010886-e1010886

2022

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Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells

PLoS pathogens, 2015-10, Vol.11 (10), p.e1005195-e1005195

2015

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Epstein-Barr virus: Biology and clinical disease

Cell, 2022-09, Vol.185 (20), p.3652-3670

2022

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Transcription factor gene Pea3 regulates erectile function during copulation in mice

PloS one, 2022-10, Vol.17 (10), p.e0276069-e0276069

2022

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Orthogonal glycolytic pathway enables directed evolution of noncanonical cofactor oxidase

Nature communications, 2022-11, Vol.13 (1), p.7282-14, Article 7282

2022

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EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized mice

PLoS pathogens, 2020-06, Vol.16 (6), p.e1008590

2020

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Hippo signaling effectors YAP and TAZ induce Epstein-Barr Virus (EBV) lytic reactivation through TEADs in epithelial cells

PLoS pathogens, 2021-08, Vol.17 (8), p.e1009783-e1009783

2021

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B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection

PLoS pathogens, 2020-02, Vol.16 (2), p.e1008365-e1008365

2020

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A New Model of Epstein-Barr Virus Infection Reveals an Important Role for Early Lytic Viral Protein Expression in the Development of Lymphomas

Journal of Virology, 2011-01, Vol.85 (1), p.165-177

2011

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Differentiation-Dependent LMP1 Expression Is Required for Efficient Lytic Epstein-Barr Virus Reactivation in Epithelial Cells

Journal of virology, 2017-04, Vol.91 (8)

2017

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Cellular differentiation regulator BLIMP1 induces Epstein-Barr virus lytic reactivation in epithelial and B cells by activating transcription from both the R and Z promoters

Journal of virology, 2015-02, Vol.89 (3), p.1731-1743

2015

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ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells

PLoS pathogens, 2021-11, Vol.17 (11), p.e1010045-e1010045

2021

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Restricted TET2 Expression in Germinal Center Type B Cells Promotes Stringent Epstein-Barr Virus Latency

Journal of virology, 2017-03, Vol.91 (5)

2017

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Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in mice

PLoS pathogens, 2024-04, Vol.20 (4), p.e1012132-e1012132

2024

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GPi deep brain stimulation for Tourette syndrome improves tics and psychiatric comorbidities

Neurology, 2007-01, Vol.68 (2), p.159-160

2007

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Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter

PLoS pathogens, 2017-06, Vol.13 (6), p.e1006404

2017

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The Cellular Ataxia Telangiectasia-Mutated Kinase Promotes Epstein-Barr Virus Lytic Reactivation in Response to Multiple Different Types of Lytic Reactivation-Inducing Stimuli

Journal of Virology, 2012-12, Vol.86 (24), p.13360-13370

2012

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Botulinum toxin in the treatment of blepharospasm and hemifacial spasm

Journal of Neural Transmission, 2008-04, Vol.115 (4), p.585-591

2008

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An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model

PLoS pathogens, 2018-08, Vol.14 (8), p.e1007221

2018

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Regulation of the latent-lytic switch in Epstein–Barr virus

PD-1/CTLA-4 Blockade Inhibits Epstein-Barr Virus-Induced Lymphoma Growth in a Cord Blood Humanized-Mouse Model

A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infection

Optimal intereye difference thresholds by optical coherence tomography in multiple sclerosis: An international study

Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells

Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

Rta is the principal activator of Epstein-Barr virus epithelial lytic transcription

Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells

Epstein-Barr virus: Biology and clinical disease

Transcription factor gene Pea3 regulates erectile function during copulation in mice

Orthogonal glycolytic pathway enables directed evolution of noncanonical cofactor oxidase

EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized mice

Hippo signaling effectors YAP and TAZ induce Epstein-Barr Virus (EBV) lytic reactivation through TEADs in epithelial cells

B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection

A New Model of Epstein-Barr Virus Infection Reveals an Important Role for Early Lytic Viral Protein Expression in the Development of Lymphomas

Differentiation-Dependent LMP1 Expression Is Required for Efficient Lytic Epstein-Barr Virus Reactivation in Epithelial Cells

Cellular differentiation regulator BLIMP1 induces Epstein-Barr virus lytic reactivation in epithelial and B cells by activating transcription from both the R and Z promoters

ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells

Restricted TET2 Expression in Germinal Center Type B Cells Promotes Stringent Epstein-Barr Virus Latency

Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in mice

GPi deep brain stimulation for Tourette syndrome improves tics and psychiatric comorbidities

Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter

The Cellular Ataxia Telangiectasia-Mutated Kinase Promotes Epstein-Barr Virus Lytic Reactivation in Response to Multiple Different Types of Lytic Reactivation-Inducing Stimuli

Botulinum toxin in the treatment of blepharospasm and hemifacial spasm

An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model

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