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SmcHD1, containing a structural-maintenance-of-chromosomes hinge domain, has a critical role in X inactivation

Nature genetics, 2008-05, Vol.40 (5), p.663-669

2008

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Smchd1 regulates long-range chromatin interactions on the inactive X chromosome and at Hox clusters

Nature structural & molecular biology, 2018-09, Vol.25 (9), p.766-777

2018

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Aggregation chimeras provide evidence of in vivo intercellular correction in ovine CLN6 neuronal ceroid lipofuscinosis (Batten disease)

PloS one, 2022-04, Vol.17 (4), p.e0261544-e0261544

2022

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Smchd1 Targeting to the Inactive X Is Dependent on the Xist-HnrnpK-PRC1 Pathway

Cell reports (Cambridge), 2018-11, Vol.25 (7), p.1912-1923.e9

2018

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Genome-wide binding and mechanistic analyses of Smchd1-mediated epigenetic regulation

Proceedings of the National Academy of Sciences - PNAS, 2015-07, Vol.112 (27), p.E3535-E3544

2015

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Chronic oral administration of minocycline to sheep with ovine CLN6 neuronal ceroid lipofuscinosis maintains pharmacological concentrations in the brain but does not suppress neuroinflammation or disease progression

Journal of neuroinflammation, 2013-07, Vol.10 (1), p.97-97, Article 900

2013

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Smchd1 regulates a subset of autosomal genes subject to monoallelic expression in addition to being critical for X inactivation

Epigenetics & chromatin, 2013-07, Vol.6 (1), p.19-19, Article 19

2013

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Glycosylphosphatidylinositol-Anchored Serine Protease PRSS21 (Testisin) Imparts Murine Epididymal Sperm Cell Maturation and Fertilizing Ability

Biology of reproduction, 2009-11, Vol.81 (5), p.921-932

2009

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Menin Associates with a Trithorax Family Histone Methyltransferase Complex and with the Hoxc8 Locus

Molecular cell, 2004-02, Vol.13 (4), p.587-597

2004

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Using a Fall-Prevention Expo To Educate the Community, Students, and Professionals

The American journal of occupational therapy, 2020-08, Vol.74 (4_Supplement_1), p.7411520473-7411520473p1

Graham, Kay

2020

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The A-repeat links ASF/SF2-dependent Xist RNA processing with random choice during X inactivation

Nature structural & molecular biology, 2010-08, Vol.17 (8), p.948-954

2010

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‘Being an Indian. . .’: An Interpretative Phenomenological Study of the Experiences of Indian-Gujarati Women During the Confinement Period

The American journal of occupational therapy, 2024-08, Vol.78 (Supplement_2), p.7811500154-7811500154p1

2024

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Glial activation spreads from specific cerebral foci and precedes neurodegeneration in presymptomatic ovine neuronal ceroid lipofuscinosis (CLN6)

Neurobiology of disease, 2005-10, Vol.20 (1), p.49-63

2005

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Lentiviral-mediated gene transfer to the sheep brain: implications for gene therapy in Batten disease

Human gene therapy, 2011-08, Vol.22 (8), p.1011-1020

2011

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Menin and p53 have non-synergistic effects on tumorigenesis in mice

BMC cancer, 2012-06, Vol.12 (1), p.252-252, Article 252

2012

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Conditional Inactivation of the Men1 Gene Leads to Pancreatic and Pituitary Tumorigenesis but Does Not Affect Normal Development of These Tissues

Molecular and Cellular Biology, 2004-04, Vol.24 (8), p.3125-3131

2004

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The specific loss of GnRH-positive neurons from the hypothalamus of sheep with CLN6 neuronal ceroid lipofuscinosis occurs without glial activation and has only minor effects on reproduction

Neurobiology of disease, 2011-03, Vol.41 (3), p.614-623

2011

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A new large animal model of CLN5 neuronal ceroid lipofuscinosis in Borderdale sheep is caused by a nucleotide substitution at a consensus splice site (c.571 + 1G >>> A) leading to excision of exon 3

Neurobiology of disease, 2008-02, Vol.29 (2), p.306-315

2008

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Requirement for Xist in X chromosome inactivation

Nature (London), 1996-01, Vol.379 (6561), p.131-137

1996

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Location and connectivity determine GABAergic interneuron survival in the brains of South Hampshire sheep with CLN6 neuronal ceroid lipofuscinosis

Neurobiology of disease, 2008-10, Vol.32 (1), p.50-65

2008

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Older Adult Perceptions of Health Management Based on Social Determinants of Health

The American journal of occupational therapy, 2024-08, Vol.78 (Supplement_2), p.7811500156-7811500156p1

2024

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Patched 1 conditional null allele in mice

Genesis (New York, N.Y. : 2000), 2003-07, Vol.36 (3), p.158-161

2003

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Tissue-Specific Ablation of Prkar1a Causes Schwannomas by Suppressing Neurofibromatosis Protein Production

Neoplasia (New York, N.Y.), 2008-11, Vol.10 (11), p.1213,IN8-1221,IN9

2008

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Mice Lacking the Vascular Endothelial Growth Factor-B Gene (Vegfb) Have Smaller Hearts, Dysfunctional Coronary Vasculature, and Impaired Recovery From Cardiac Ischemia

Circulation research, 2000-02, Vol.86 (2), p.e29-e35

2000

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Pex13 Inactivation in the Mouse Disrupts Peroxisome Biogenesis and Leads to a Zellweger Syndrome Phenotype

Molecular and Cellular Biology, 2003-08, Vol.23 (16), p.5947-5957

2003

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SmcHD1, containing a structural-maintenance-of-chromosomes hinge domain, has a critical role in X inactivation

Smchd1 regulates long-range chromatin interactions on the inactive X chromosome and at Hox clusters

Aggregation chimeras provide evidence of in vivo intercellular correction in ovine CLN6 neuronal ceroid lipofuscinosis (Batten disease)

Smchd1 Targeting to the Inactive X Is Dependent on the Xist-HnrnpK-PRC1 Pathway

Genome-wide binding and mechanistic analyses of Smchd1-mediated epigenetic regulation

Chronic oral administration of minocycline to sheep with ovine CLN6 neuronal ceroid lipofuscinosis maintains pharmacological concentrations in the brain but does not suppress neuroinflammation or disease progression

Smchd1 regulates a subset of autosomal genes subject to monoallelic expression in addition to being critical for X inactivation

Glycosylphosphatidylinositol-Anchored Serine Protease PRSS21 (Testisin) Imparts Murine Epididymal Sperm Cell Maturation and Fertilizing Ability

Menin Associates with a Trithorax Family Histone Methyltransferase Complex and with the Hoxc8 Locus

Using a Fall-Prevention Expo To Educate the Community, Students, and Professionals

The A-repeat links ASF/SF2-dependent Xist RNA processing with random choice during X inactivation

‘Being an Indian. . .’: An Interpretative Phenomenological Study of the Experiences of Indian-Gujarati Women During the Confinement Period

Glial activation spreads from specific cerebral foci and precedes neurodegeneration in presymptomatic ovine neuronal ceroid lipofuscinosis (CLN6)

Lentiviral-mediated gene transfer to the sheep brain: implications for gene therapy in Batten disease

Menin and p53 have non-synergistic effects on tumorigenesis in mice

Conditional Inactivation of the Men1 Gene Leads to Pancreatic and Pituitary Tumorigenesis but Does Not Affect Normal Development of These Tissues

The specific loss of GnRH-positive neurons from the hypothalamus of sheep with CLN6 neuronal ceroid lipofuscinosis occurs without glial activation and has only minor effects on reproduction

A new large animal model of CLN5 neuronal ceroid lipofuscinosis in Borderdale sheep is caused by a nucleotide substitution at a consensus splice site (c.571 + 1G >>> A) leading to excision of exon 3

Requirement for Xist in X chromosome inactivation

Location and connectivity determine GABAergic interneuron survival in the brains of South Hampshire sheep with CLN6 neuronal ceroid lipofuscinosis

Older Adult Perceptions of Health Management Based on Social Determinants of Health

Patched 1 conditional null allele in mice

Tissue-Specific Ablation of Prkar1a Causes Schwannomas by Suppressing Neurofibromatosis Protein Production

Mice Lacking the Vascular Endothelial Growth Factor-B Gene (Vegfb) Have Smaller Hearts, Dysfunctional Coronary Vasculature, and Impaired Recovery From Cardiac Ischemia

Pex13 Inactivation in the Mouse Disrupts Peroxisome Biogenesis and Leads to a Zellweger Syndrome Phenotype

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