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The β3‐adrenoceptor agonist mirabegron increases human atrial force through β1‐adrenoceptors: an indirect mechanism?

British journal of pharmacology, 2017-08, Vol.174 (16), p.2706-2715

2017

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5-hydroxytryptamine receptors in the human cardiovascular system

Pharmacology & therapeutics (Oxford), 2006-09, Vol.111 (3), p.674-706

2006

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In permanent atrial fibrillation, PDE3 reduces force responses to 5‐HT, but PDE3 and PDE4 do not cause the blunting of atrial arrhythmias

British journal of pharmacology, 2016-08, Vol.173 (16), p.2478-2489

2016

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Phosphodiesterase PDE2 activity, increased by isoprenaline, does not reduce β-adrenoceptor-mediated chronotropic and inotropic effects in rat heart

Naunyn-Schmiedeberg's archives of pharmacology, 2018-06, Vol.391 (6), p.571-585

2018

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Phosphodiesterases reduce spontaneous sinoatrial beating but not the ‘fight or flight’ tachycardia elicited by agonists through Gs-protein-coupled receptors

Trends in pharmacological sciences (Regular ed.), 2011-07, Vol.32 (7), p.377-383

Kaumann, Alberto J

2011

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The low-affinity site of the β1-adrenoceptor and its relevance to cardiovascular pharmacology

Pharmacology & therapeutics (Oxford), 2008-06, Vol.118 (3), p.303-336

2008

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Impact of phosphodiesterases PDE3 and PDE4 on 5-hydroxytryptamine receptor4-mediated increase of cAMP in human atrial fibrillation

Naunyn-Schmiedeberg's archives of pharmacology, 2021-02, Vol.394 (2), p.291-298

2021

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Inhibitors of phosphodiesterases PDE2, PDE3, and PDE4 do not increase the sinoatrial tachycardia of noradrenaline and prostaglandin PGE1 in mice

Naunyn-Schmiedeberg's archives of pharmacology, 2016-02, Vol.389 (2), p.177-186

2016

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PDE3, but not PDE4, reduces β1‐ and β2‐adrenoceptor‐mediated inotropic and lusitropic effects in failing ventricle from metoprolol‐treated patients

British journal of pharmacology, 2013-06, Vol.169 (3), p.528-538

2013

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Mucosa of human detrusor impairs contraction and β‐adrenoceptor‐mediated relaxation

BJU international, 2013-12, Vol.112 (8), p.1215-1222

2013

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Mucosa of murine detrusor impairs β2-adrenoceptor-mediated relaxation

Neurourology and urodynamics, 2015-08, Vol.34 (6), p.592-597

2015

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Ontogenic changes of the control by phosphodiesterase‐3 and ‐4 of 5‐HT responses in porcine heart and relevance to human atrial 5‐HT4 receptors

British journal of pharmacology, 2009-01, Vol.156 (2), p.237-249

2009

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Fading of 5‐HT 4 receptor‐mediated inotropic responses to 5‐hydroxytryptamine is caused by phosphodiesterase activity in porcine atrium

British journal of pharmacology, 2009-01, Vol.147 (2), p.128-130

2009

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Function of cardiac β1- and β2-adrenoceptors of newborn piglets: Role of phosphodiesterases PDE3 and PDE4

European journal of pharmacology, 2010-07, Vol.638 (1-3), p.99-107

2010

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Phosphodiesterases PDE3 and PDE4 jointly control the inotropic effects but not chronotropic effects of (−)-CGP12177 despite PDE4-evoked sinoatrial bradycardia in rat atrium

Naunyn-Schmiedeberg's archives of pharmacology, 2009-04, Vol.379 (4), p.379-384

2009

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Appearance of a ventricular 5-HT4 receptor-mediated inotropic response to serotonin in heart failure

Cardiovascular research, 2005-03, Vol.65 (4), p.869-878

2005

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BIBN4096BS is a potent competitive antagonist of the relaxant effects of α‐CGRP on human temporal artery: comparison with CGRP(8‐37)

British journal of pharmacology, 2002-05, Vol.136 (1), p.120-126

2002

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Phosphodiesterase PDE3 blunts the positive inotropic and cyclic AMP enhancing effects of CGP12177 but not of noradrenaline in rat ventricle

British journal of pharmacology, 2006-01, Vol.147 (2), p.158-163

2006

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Cloning, pharmacological characterisation and tissue distribution of a novel 5-HT4 receptor splice variant, 5-HT4(i)

Naunyn-Schmiedeberg's archives of pharmacology, 2004-06, Vol.369 (6), p.616-628

2004

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Functional serotonin 5-HT4 receptors in porcine and human ventricular myocardium with increased 5-HT4 mRNA in heart failure

Naunyn-Schmiedeberg's archives of pharmacology, 2004-09, Vol.370 (3), p.157-166

2004

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Inotropy and L‐type Ca2+ current, activated by β1‐ and β2‐adrenoceptors, are differently controlled by phosphodiesterases 3 and 4 in rat heart

British journal of pharmacology, 2009-01, Vol.156 (1), p.62-83

2009

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Homology model and docking studies on porcine β2 adrenoceptor: description of two binding sites

Journal of molecular modeling, 2011-10, Vol.17 (10), p.2525-2538

2011

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Comparison of the affinity of β‐blockers for two states of the β1‐adrenoceptor in ferret ventricular myocardium

British journal of pharmacology, 2002-01, Vol.135 (2), p.451-461

2002

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Catecholamines relax detrusor through beta 2-adrenoceptors in mouse and beta 3-adrenoceptors in man

The Journal of pharmacology and experimental therapeutics, 2009-01, Vol.328 (1), p.213

2009

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Physiological antagonism between ventricular β1‐adrenoceptors and α1‐adrenoceptors but no evidence for β2‐ and β3‐adrenoceptor function in murine heart

British journal of pharmacology, 2002-05, Vol.136 (2), p.217-229

2002

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The β3‐adrenoceptor agonist mirabegron increases human atrial force through β1‐adrenoceptors: an indirect mechanism?

5-hydroxytryptamine receptors in the human cardiovascular system

In permanent atrial fibrillation, PDE3 reduces force responses to 5‐HT, but PDE3 and PDE4 do not cause the blunting of atrial arrhythmias

Phosphodiesterase PDE2 activity, increased by isoprenaline, does not reduce β-adrenoceptor-mediated chronotropic and inotropic effects in rat heart

Phosphodiesterases reduce spontaneous sinoatrial beating but not the ‘fight or flight’ tachycardia elicited by agonists through Gs-protein-coupled receptors

The low-affinity site of the β1-adrenoceptor and its relevance to cardiovascular pharmacology

Impact of phosphodiesterases PDE3 and PDE4 on 5-hydroxytryptamine receptor4-mediated increase of cAMP in human atrial fibrillation

Inhibitors of phosphodiesterases PDE2, PDE3, and PDE4 do not increase the sinoatrial tachycardia of noradrenaline and prostaglandin PGE1 in mice

PDE3, but not PDE4, reduces β1‐ and β2‐adrenoceptor‐mediated inotropic and lusitropic effects in failing ventricle from metoprolol‐treated patients

Mucosa of human detrusor impairs contraction and β‐adrenoceptor‐mediated relaxation

Mucosa of murine detrusor impairs β2-adrenoceptor-mediated relaxation

Ontogenic changes of the control by phosphodiesterase‐3 and ‐4 of 5‐HT responses in porcine heart and relevance to human atrial 5‐HT4 receptors

Fading of 5‐HT 4 receptor‐mediated inotropic responses to 5‐hydroxytryptamine is caused by phosphodiesterase activity in porcine atrium

Function of cardiac β1- and β2-adrenoceptors of newborn piglets: Role of phosphodiesterases PDE3 and PDE4

Phosphodiesterases PDE3 and PDE4 jointly control the inotropic effects but not chronotropic effects of (−)-CGP12177 despite PDE4-evoked sinoatrial bradycardia in rat atrium

Appearance of a ventricular 5-HT4 receptor-mediated inotropic response to serotonin in heart failure

BIBN4096BS is a potent competitive antagonist of the relaxant effects of α‐CGRP on human temporal artery: comparison with CGRP(8‐37)

Phosphodiesterase PDE3 blunts the positive inotropic and cyclic AMP enhancing effects of CGP12177 but not of noradrenaline in rat ventricle

Cloning, pharmacological characterisation and tissue distribution of a novel 5-HT4 receptor splice variant, 5-HT4(i)

Functional serotonin 5-HT4 receptors in porcine and human ventricular myocardium with increased 5-HT4 mRNA in heart failure

Inotropy and L‐type Ca2+ current, activated by β1‐ and β2‐adrenoceptors, are differently controlled by phosphodiesterases 3 and 4 in rat heart

Homology model and docking studies on porcine β2 adrenoceptor: description of two binding sites

Comparison of the affinity of β‐blockers for two states of the β1‐adrenoceptor in ferret ventricular myocardium

Catecholamines relax detrusor through beta 2-adrenoceptors in mouse and beta 3-adrenoceptors in man

Physiological antagonism between ventricular β1‐adrenoceptors and α1‐adrenoceptors but no evidence for β2‐ and β3‐adrenoceptor function in murine heart

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