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A carnosine analog mitigates metabolic disorders of obesity by reducing carbonyl stress

The Journal of clinical investigation, 2018-12, Vol.128 (12), p.5280-5293

2018

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Mitochondria as a source and target of lipid peroxidation products in healthy and diseased heart

Clinical and experimental pharmacology & physiology, 2012-02, Vol.39 (2), p.179-193

2012

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2-Chlorofatty acids: lipid mediators of neutrophil extracellular trap formation

Journal of lipid research, 2018-08, Vol.59 (8), p.1424-1432

2018

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Enhanced Catecholamine Flux and Impaired Carbonyl Metabolism Disrupt Cardiac Mitochondrial Oxidative Phosphorylation in Diabetes Patients

Antioxidants & redox signaling, 2021-08, Vol.35 (4), p.235

2021

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Novel role for thioredoxin reductase‐2 in mitochondrial redox adaptations to obesogenic diet and exercise in heart and skeletal muscle

The Journal of physiology, 2013-07, Vol.591 (14), p.3471-3486

2013

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Potential for improved glycemic control with dietary Momordica charantia in patients with insulin resistance and pre-diabetes

International journal of environmental research and public health, 2014-02, Vol.11 (2), p.2328-2345

2014

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Obesity in a model of gpx4 haploinsufficiency uncovers a causal role for lipid-derived aldehydes in human metabolic disease and cardiomyopathy

Molecular metabolism (Germany), 2015-06, Vol.4 (6), p.493-506

2015

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The "Goldilocks Zone" from a redox perspective-Adaptive vs. deleterious responses to oxidative stress in striated muscle

Frontiers in physiology, 2014-09, Vol.5, p.358

2014

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Acyl-CoA synthetase 1 deficiency alters cardiolipin species and impairs mitochondrial function

Journal of lipid research, 2015-08, Vol.56 (8), p.1572-1582

2015

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Smoking and dietary inadequacy among Inuvialuit women of child bearing age in the Northwest Territories, Canada

Nutrition journal, 2013-02, Vol.12 (1), p.27-27, Article 27

2013

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Monoamine Oxidase is a Major Determinant of Redox Balance in Human Atrial Myocardium and is Associated With Postoperative Atrial Fibrillation

Journal of the American Heart Association, 2014-02, Vol.3 (1), p.e000713-n/a

2014

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Corrigendum to “Obesity in a model of gpx4 haploinsufficiency uncovers a causal role for lipid-derived aldehydes in human metabolic disease and cardiomyopathy” [Mol Metab 4 (6) (2015) 493–506]

Molecular metabolism (Germany), 2015-10, Vol.4 (10), p.753-753

2015

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Myeloperoxidase‐Derived 2‐Chlorofatty Acids Make Neutrophils Go NETs

The FASEB journal, 2018-04, Vol.32 (S1), p.813.6-813.6

2018

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The â€œGoldilocks Zoneâ€ from a redox perspectiveâ€”Adaptive vs. deleterious responses to oxidative stress in striated muscle

Frontiers in physiology, 2014-09, Vol.5

2014

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Human Macrophage Derived Myeloperoxidase Exacerbates Nonalcoholic Steatohepatitis Disease (NASH) in Diet‐induced Obesity

The FASEB journal, 2018-04, Vol.32 (S1), p.873.16-873.16

2018

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A Translational Kinetic Analysis of Rodent and Human Hearts Shows Species‐specific Differences and Increases in Monoamine Oxidase Activity with Obesity/Diabetes

The FASEB journal, 2015-04, Vol.29 (S1)

2015

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Glutathione Peroxidase‐4 plays a critical role in protection from metabolic syndrome and cardiac remodeling caused by diet‐induced obesity

The FASEB journal, 2013-04, Vol.27 (S1)

2013

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Reactive aldehydes formed from peroxidation of n‐6 and n‐3 PUFAs have differential effects on mitochondrial function in heart

The FASEB journal, 2012-04, Vol.26 (S1)

2012

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Exploring the role of elevated lipid peroxidation and glutathione peroxidase 4 in metabolic syndrome and cardiac remodeling in obesity

Katunga, Lalage Adalaide

2016

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ProQuest

Differential Adaptations to High‐calorie, Western‐pattern Diet and Exercise Training in Redox Status and Mitochondria of Heart and Skeletal Muscle

The FASEB journal, 2012-04, Vol.26 (S1)

2012

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Corrigendum to “Obesity in a model of gpx 4 haploinsufficiency uncovers a causal role for lipid-derived aldehydes in human metabolic disease and cardiomyopathy” [Mol Metab 4 (6) (2015) 493–506]

Molecular metabolism (Germany), 2015, Vol.4 (10), p.753-753

2015

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The ‘Goldilocks Zone’ from a redox perspective - Adaptive versus deleterious responses to oxidative stress in striated muscle

Frontiers in physiology, 2014-09, Vol.5

2014

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A carnosine analog mitigates metabolic disorders of obesity by reducing carbonyl stress

Mitochondria as a source and target of lipid peroxidation products in healthy and diseased heart

2-Chlorofatty acids: lipid mediators of neutrophil extracellular trap formation

Enhanced Catecholamine Flux and Impaired Carbonyl Metabolism Disrupt Cardiac Mitochondrial Oxidative Phosphorylation in Diabetes Patients

Novel role for thioredoxin reductase‐2 in mitochondrial redox adaptations to obesogenic diet and exercise in heart and skeletal muscle

Potential for improved glycemic control with dietary Momordica charantia in patients with insulin resistance and pre-diabetes

Obesity in a model of gpx4 haploinsufficiency uncovers a causal role for lipid-derived aldehydes in human metabolic disease and cardiomyopathy

The "Goldilocks Zone" from a redox perspective-Adaptive vs. deleterious responses to oxidative stress in striated muscle

Acyl-CoA synthetase 1 deficiency alters cardiolipin species and impairs mitochondrial function

Smoking and dietary inadequacy among Inuvialuit women of child bearing age in the Northwest Territories, Canada

Monoamine Oxidase is a Major Determinant of Redox Balance in Human Atrial Myocardium and is Associated With Postoperative Atrial Fibrillation

Corrigendum to “Obesity in a model of gpx4 haploinsufficiency uncovers a causal role for lipid-derived aldehydes in human metabolic disease and cardiomyopathy” [Mol Metab 4 (6) (2015) 493–506]

Myeloperoxidase‐Derived 2‐Chlorofatty Acids Make Neutrophils Go NETs

The â€œGoldilocks Zoneâ€ from a redox perspectiveâ€”Adaptive vs. deleterious responses to oxidative stress in striated muscle

Human Macrophage Derived Myeloperoxidase Exacerbates Nonalcoholic Steatohepatitis Disease (NASH) in Diet‐induced Obesity

A Translational Kinetic Analysis of Rodent and Human Hearts Shows Species‐specific Differences and Increases in Monoamine Oxidase Activity with Obesity/Diabetes

Glutathione Peroxidase‐4 plays a critical role in protection from metabolic syndrome and cardiac remodeling caused by diet‐induced obesity

Reactive aldehydes formed from peroxidation of n‐6 and n‐3 PUFAs have differential effects on mitochondrial function in heart

Exploring the role of elevated lipid peroxidation and glutathione peroxidase 4 in metabolic syndrome and cardiac remodeling in obesity

Differential Adaptations to High‐calorie, Western‐pattern Diet and Exercise Training in Redox Status and Mitochondria of Heart and Skeletal Muscle

Corrigendum to “Obesity in a model of gpx 4 haploinsufficiency uncovers a causal role for lipid-derived aldehydes in human metabolic disease and cardiomyopathy” [Mol Metab 4 (6) (2015) 493–506]

The ‘Goldilocks Zone’ from a redox perspective - Adaptive versus deleterious responses to oxidative stress in striated muscle

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