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Ciliary protein trafficking mediated by IFT and BBSome complexes with the aid of kinesin-2 and dynein-2 motors

Journal of biochemistry (Tokyo), 2018-03, Vol.163 (3), p.155-164

2018

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Architecture of the IFT ciliary trafficking machinery and interplay between its components

Critical reviews in biochemistry and molecular biology, 2020-03, Vol.55 (2), p.179-196

2020

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Intraflagellar transport-A complex mediates ciliary entry and retrograde trafficking of ciliary G protein-coupled receptors

Molecular biology of the cell, 2017-02, Vol.28 (3), p.429-439

2017

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Architectures of multisubunit complexes revealed by a visible immunoprecipitation assay using fluorescent fusion proteins

Journal of cell science, 2015-06, Vol.128 (12), p.2351-2362

2015

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Practical method for superresolution imaging of primary cilia and centrioles by expansion microscopy using an amplibody for fluorescence signal amplification

Molecular biology of the cell, 2020-09, Vol.31 (20), p.2195-2206

2020

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CEP19-RABL2-IFT-B axis controls BBSome-mediated ciliary GPCR export

Molecular biology of the cell, 2022-11, Vol.33 (13), p.ar126-ar126

2022

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Regulation of ciliary retrograde protein trafficking by the Joubert syndrome proteins ARL13B and INPP5E

Journal of cell science, 2017-02, Vol.130 (3), p.563-576

2017

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Overall Architecture of the Intraflagellar Transport (IFT)-B Complex Containing Cluap1/IFT38 as an Essential Component of the IFT-B Peripheral Subcomplex

The Journal of biological chemistry, 2016-05, Vol.291 (21), p.10962-10975

2016

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Combinations of deletion and missense variations of the dynein-2 DYNC2LI1 subunit found in skeletal ciliopathies cause ciliary defects

Scientific reports, 2022-01, Vol.12 (1), p.31-31, Article 31

2022

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Molecular basis of ciliary defects caused by compound heterozygous IFT144/WDR19 mutations found in cranioectodermal dysplasia

Human molecular genetics, 2021-04, Vol.30 (3-4), p.213-225

2021

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Impaired cooperation between IFT74/BBS22-IFT81 and IFT25-IFT27/BBS19 causes Bardet-Biedl syndrome

Human molecular genetics, 2022-05, Vol.31 (10), p.1681-1693

2022

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Practical method for targeted disruption of cilia-related genes by using CRISPR/Cas9-mediated, homology-independent knock-in system

Molecular biology of the cell, 2017-04, Vol.28 (7), p.898-906

2017

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Rab11 regulates exocytosis of recycling vesicles at the plasma membrane

Journal of cell science, 2012-09, Vol.125 (Pt 17), p.4049-4057

2012

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CCRK/CDK20 regulates ciliary retrograde protein trafficking via interacting with BROMI/TBC1D32

PloS one, 2021-10, Vol.16 (10), p.e0258497

2021

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Anterograde trafficking of ciliary MAP kinase–like ICK/CILK1 by the intraflagellar transport machinery is required for intraciliary retrograde protein trafficking

The Journal of biological chemistry, 2020-09, Vol.295 (38), p.13363-13376

2020

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Formation of the B9-domain protein complex MKS1-B9D2-B9D1 is essential as a diffusion barrier for ciliary membrane proteins

Molecular biology of the cell, 2020-09, Vol.31 (20), p.2259-2268

2020

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BBS1 is involved in retrograde trafficking of ciliary GPCRs in the context of the BBSome complex

PloS one, 2018-03, Vol.13 (3), p.e0195005-e0195005

2018

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Robust interaction of IFT70 with IFT52-IFT88 in the IFT-B complex is required for ciliogenesis

Biology open, 2018, Vol.7 (5), p.bio033241-bio033241

2018

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Ciliary entry of KIF17 is dependent on its binding to the IFT-B complex via IFT46-IFT56 as well as on its nuclear localization signal

Molecular biology of the cell, 2017-03, Vol.28 (5), p.624-633

2017

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Disease-associated mutations in WDR34 lead to diverse impacts on the assembly and function of dynein-2

Journal of cell science, 2023-03, Vol.136 (5)

2023

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Interaction of INPP5E with ARL13B is essential for its ciliary membrane retention but dispensable for its ciliary entry

Biology open, 2021-01, Vol.10 (1)

2021

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The phospholipid flippase ATP9A is required for the recycling pathway from the endosomes to the plasma membrane

Molecular biology of the cell, 2016-12, Vol.27 (24), p.3883-3893

2016

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Modulation of primary cilia length by melanin-concentrating hormone receptor 1

Cellular signalling, 2016-06, Vol.28 (6), p.572-584

2016

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Visible Immunoprecipitation (VIP) Assay: a Simple and Versatile Method for Visual Detection of Protein-protein Interactions

Bio-protocol, 2018, Vol.8 (1)

2018

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ARL3 and ARL13B GTPases participate in distinct steps of INPP5E targeting to the ciliary membrane

Biology open, 2021-09, Vol.10 (9)

2021

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Ciliary protein trafficking mediated by IFT and BBSome complexes with the aid of kinesin-2 and dynein-2 motors

Architecture of the IFT ciliary trafficking machinery and interplay between its components

Intraflagellar transport-A complex mediates ciliary entry and retrograde trafficking of ciliary G protein-coupled receptors

Architectures of multisubunit complexes revealed by a visible immunoprecipitation assay using fluorescent fusion proteins

Practical method for superresolution imaging of primary cilia and centrioles by expansion microscopy using an amplibody for fluorescence signal amplification

CEP19-RABL2-IFT-B axis controls BBSome-mediated ciliary GPCR export

Regulation of ciliary retrograde protein trafficking by the Joubert syndrome proteins ARL13B and INPP5E

Overall Architecture of the Intraflagellar Transport (IFT)-B Complex Containing Cluap1/IFT38 as an Essential Component of the IFT-B Peripheral Subcomplex

Combinations of deletion and missense variations of the dynein-2 DYNC2LI1 subunit found in skeletal ciliopathies cause ciliary defects

Molecular basis of ciliary defects caused by compound heterozygous IFT144/WDR19 mutations found in cranioectodermal dysplasia

Impaired cooperation between IFT74/BBS22-IFT81 and IFT25-IFT27/BBS19 causes Bardet-Biedl syndrome

Practical method for targeted disruption of cilia-related genes by using CRISPR/Cas9-mediated, homology-independent knock-in system

Rab11 regulates exocytosis of recycling vesicles at the plasma membrane

CCRK/CDK20 regulates ciliary retrograde protein trafficking via interacting with BROMI/TBC1D32

Anterograde trafficking of ciliary MAP kinase–like ICK/CILK1 by the intraflagellar transport machinery is required for intraciliary retrograde protein trafficking

Formation of the B9-domain protein complex MKS1-B9D2-B9D1 is essential as a diffusion barrier for ciliary membrane proteins

BBS1 is involved in retrograde trafficking of ciliary GPCRs in the context of the BBSome complex

Robust interaction of IFT70 with IFT52-IFT88 in the IFT-B complex is required for ciliogenesis

Ciliary entry of KIF17 is dependent on its binding to the IFT-B complex via IFT46-IFT56 as well as on its nuclear localization signal

Disease-associated mutations in WDR34 lead to diverse impacts on the assembly and function of dynein-2

Interaction of INPP5E with ARL13B is essential for its ciliary membrane retention but dispensable for its ciliary entry

The phospholipid flippase ATP9A is required for the recycling pathway from the endosomes to the plasma membrane

Modulation of primary cilia length by melanin-concentrating hormone receptor 1

Visible Immunoprecipitation (VIP) Assay: a Simple and Versatile Method for Visual Detection of Protein-protein Interactions

ARL3 and ARL13B GTPases participate in distinct steps of INPP5E targeting to the ciliary membrane

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