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Enzymatic pathways in the pathogenesis of hereditary angioedema: The role of C1 inhibitor therapy

Journal of allergy and clinical immunology, 2010-11, Vol.126 (5), p.918-925

Kaplan, Allen P., MD

2010

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The bradykinin-forming cascade and its role in hereditary angioedema

Annals of allergy, asthma, & immunology, 2010-03, Vol.104 (3), p.193-204

2010

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Therapy of chronic urticaria: a simple, modern approach

Annals of allergy, asthma, & immunology, 2014-05, Vol.112 (5), p.419-425

Kaplan, Allen P., MD

2014

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Treatment of chronic autoimmune urticaria with omalizumab

Journal of allergy and clinical immunology, 2008-09, Vol.122 (3), p.569-573

2008

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Deficiency of plasminogen activator inhibitor 2 in plasma of patients with hereditary angioedema with normal C1 inhibitor levels

Journal of allergy and clinical immunology, 2016-06, Vol.137 (6), p.1822-1829.e1

2016

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Fibrinolysis and bradykinin formation: From in vitro observations to human disease in “just” 45 years

Journal of allergy and clinical immunology, 2016-11, Vol.138 (5), p.1424-1425

Kaplan, Allen P., MD

2016

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Factor XII–independent activation of the bradykinin-forming cascade: Implications for the pathogenesis of hereditary angioedema types I and II

Journal of allergy and clinical immunology, 2013-08, Vol.132 (2), p.470-475

2013

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Omalizumab is effective in nonautoimmune urticaria

Journal of allergy and clinical immunology, 2011-05, Vol.127 (5), p.1300-1302

2011

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Similar Efficacy with Omalizumab in Chronic Idiopathic/Spontaneous Urticaria Despite Different Background Therapy

The journal of allergy and clinical immunology in practice (Cambridge, MA), 2015-09, Vol.3 (5), p.743-750.e1

2015

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What the first 10,000 patients with chronic urticaria have taught me: A personal journey

Journal of allergy and clinical immunology, 2009-03, Vol.123 (3), p.713-717

Kaplan, Allen P., MD

2009

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Factor XII–independent cleavage of high-molecular-weight kininogen by prekallikrein and inhibition by C1 inhibitor

Journal of allergy and clinical immunology, 2009-07, Vol.124 (1), p.143-149

2009

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Cytokine and Estrogen Stimulation of Endothelial Cells Augments Activation of the Surface-Bound Prekallikrein-High Molecular Weight Kininogen Complex: Implications for Hereditary Angioedema (HAE)

Journal of allergy and clinical immunology, 2016-02, Vol.137 (2), p.AB249-AB249

2016

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Pathogenesis of Hereditary Angioedema with Normal C1 Inhibitor: Evidence for Abnormalities in Plasminogen Activator Inhibitors

Journal of allergy and clinical immunology, 2015-02, Vol.135 (2), p.AB277-AB277

2015

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Reply

Journal of allergy and clinical immunology, 2017

2017

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Response Patterns in Chronic Idiopathic/Spontaneous Urticaria (CIU/CSU) Patients Treated with Omalizumab for 24 Weeks in Two Randomized, Double-Blind, Placebo-Controlled Clinical Trials (ASTERIA I and GLACIAL)

Journal of allergy and clinical immunology, 2015-02, Vol.135 (2), p.AB127-AB127

2015

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Treatment of episodes of hereditary angioedema with C1 inhibitor: serial assessment of observed abnormalities of the plasma bradykinin-forming pathway and fibrinolysis

Annals of allergy, asthma, & immunology, 2010, Vol.104 (1), p.50-54

2010

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Studies of the mechanisms of bradykinin generation in hereditary angioedema plasma

Annals of allergy, asthma, & immunology, 2008-09, Vol.101 (3), p.279-286

2008

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Kinin formation in C1 inhibitor deficiency

Journal of allergy and clinical immunology, 2010-06, Vol.125 (6), p.1411-1412

2010

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Reply

Journal of allergy and clinical immunology, 2009, Vol.123 (4), p.970-971

Kaplan, Allen P., MD

2009

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Basophil secretion in chronic urticaria: Autoantibody-dependent or not?

Journal of allergy and clinical immunology, 2007-09, Vol.120 (3), p.729-730

2007

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How omalizumab came to be studied as a therapy for chronic spontaneous/idiopathic urticaria

The journal of allergy and clinical immunology in practice (Cambridge, MA), 2015-07, Vol.3 (4), p.648-648

2015

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History of the World Allergy Organization: 1989 to 2006, the XVIII World Allergy Congress, Journal Development, Reorganization, and New Programs

The World Allergy Organization journal, 2011-08, Vol.4 (8), p.135-137

Kaplan, Allen P

2011

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Diagnosis and treatment of urticaria and angioedema: a worldwide perspective

The World Allergy Organization journal, 2012-11, Vol.5 (11), p.125-147

2012

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Reply

Journal of allergy and clinical immunology, 2009, Vol.123 (2), p.511-511

2009

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Diagnosis, pathogenesis, and treatment of chronic spontaneous urticaria

Allergy and asthma proceedings, 2018-05, Vol.39 (3), p.184

Kaplan, Allen P

2018

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Enzymatic pathways in the pathogenesis of hereditary angioedema: The role of C1 inhibitor therapy

The bradykinin-forming cascade and its role in hereditary angioedema

Therapy of chronic urticaria: a simple, modern approach

Treatment of chronic autoimmune urticaria with omalizumab

Deficiency of plasminogen activator inhibitor 2 in plasma of patients with hereditary angioedema with normal C1 inhibitor levels

Fibrinolysis and bradykinin formation: From in vitro observations to human disease in “just” 45 years

Factor XII–independent activation of the bradykinin-forming cascade: Implications for the pathogenesis of hereditary angioedema types I and II

Omalizumab is effective in nonautoimmune urticaria

Similar Efficacy with Omalizumab in Chronic Idiopathic/Spontaneous Urticaria Despite Different Background Therapy

What the first 10,000 patients with chronic urticaria have taught me: A personal journey

Factor XII–independent cleavage of high-molecular-weight kininogen by prekallikrein and inhibition by C1 inhibitor

Cytokine and Estrogen Stimulation of Endothelial Cells Augments Activation of the Surface-Bound Prekallikrein-High Molecular Weight Kininogen Complex: Implications for Hereditary Angioedema (HAE)

Pathogenesis of Hereditary Angioedema with Normal C1 Inhibitor: Evidence for Abnormalities in Plasminogen Activator Inhibitors

Reply

Response Patterns in Chronic Idiopathic/Spontaneous Urticaria (CIU/CSU) Patients Treated with Omalizumab for 24 Weeks in Two Randomized, Double-Blind, Placebo-Controlled Clinical Trials (ASTERIA I and GLACIAL)

Treatment of episodes of hereditary angioedema with C1 inhibitor: serial assessment of observed abnormalities of the plasma bradykinin-forming pathway and fibrinolysis

Studies of the mechanisms of bradykinin generation in hereditary angioedema plasma

Kinin formation in C1 inhibitor deficiency

Reply

Basophil secretion in chronic urticaria: Autoantibody-dependent or not?

How omalizumab came to be studied as a therapy for chronic spontaneous/idiopathic urticaria

History of the World Allergy Organization: 1989 to 2006, the XVIII World Allergy Congress, Journal Development, Reorganization, and New Programs

Diagnosis and treatment of urticaria and angioedema: a worldwide perspective

Reply

Diagnosis, pathogenesis, and treatment of chronic spontaneous urticaria

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