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Omalizumab in patients with symptomatic chronic idiopathic/spontaneous urticaria despite standard combination therapy

Journal of allergy and clinical immunology, 2013-07, Vol.132 (1), p.101-109

2013

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A randomized, placebo-controlled, dose-ranging study of single-dose omalizumab in patients with H1 -antihistamine–refractory chronic idiopathic urticaria

Journal of allergy and clinical immunology, 2011-09, Vol.128 (3), p.567-573.e1

2011

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Timing and duration of omalizumab response in patients with chronic idiopathic/spontaneous urticaria

Journal of allergy and clinical immunology, 2016-02, Vol.137 (2), p.474-481

2016

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Enzymatic pathways in the pathogenesis of hereditary angioedema: The role of C1 inhibitor therapy

Journal of allergy and clinical immunology, 2010-11, Vol.126 (5), p.918-925

Kaplan, Allen P., MD

2010

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Angioedema in the omalizumab chronic idiopathic/spontaneous urticaria pivotal studies

Annals of allergy, asthma, & immunology, 2016-10, Vol.117 (4), p.370-377.e1

2016

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Therapy of chronic urticaria: a simple, modern approach

Annals of allergy, asthma, & immunology, 2014-05, Vol.112 (5), p.419-425

Kaplan, Allen P., MD

2014

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The bradykinin-forming cascade and its role in hereditary angioedema

Annals of allergy, asthma, & immunology, 2010-03, Vol.104 (3), p.193-204

2010

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Type III hereditary angioedema: defined, but not understood

Annals of allergy, asthma, & immunology, 2012, Vol.109 (3), p.153-154

Kaplan, Allen, MD

2012

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Treatment of chronic autoimmune urticaria with omalizumab

Journal of allergy and clinical immunology, 2008-09, Vol.122 (3), p.569-573

2008

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Deficiency of plasminogen activator inhibitor 2 in plasma of patients with hereditary angioedema with normal C1 inhibitor levels

Journal of allergy and clinical immunology, 2016-06, Vol.137 (6), p.1822-1829.e1

2016

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A Review of Childhood and Adolescent Craniopharyngiomas With Particular Attention to Hypothalamic Obesity

Pediatric neurology, 2014, Vol.50 (1), p.4-10

2014

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Development of a daily diary for patients with chronic idiopathic urticaria

Annals of allergy, asthma, & immunology, 2010-08, Vol.105 (2), p.142-148

2010

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Factor XII–independent activation of the bradykinin-forming cascade: Implications for the pathogenesis of hereditary angioedema types I and II

Journal of allergy and clinical immunology, 2013-08, Vol.132 (2), p.470-475

2013

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Fibrinolysis and bradykinin formation: From in vitro observations to human disease in “just” 45 years

Journal of allergy and clinical immunology, 2016-11, Vol.138 (5), p.1424-1425

Kaplan, Allen P., MD

2016

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Omalizumab is effective in nonautoimmune urticaria

Journal of allergy and clinical immunology, 2011-05, Vol.127 (5), p.1300-1302

2011

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Importance of angioedema-free days in patients with chronic idiopathic or spontaneous urticaria

Annals of allergy, asthma, & immunology, 2017-04, Vol.118 (4), p.524-524

2017

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Similar Efficacy with Omalizumab in Chronic Idiopathic/Spontaneous Urticaria Despite Different Background Therapy

The journal of allergy and clinical immunology in practice (Cambridge, MA), 2015-09, Vol.3 (5), p.743-750.e1

2015

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What the first 10,000 patients with chronic urticaria have taught me: A personal journey

Journal of allergy and clinical immunology, 2009-03, Vol.123 (3), p.713-717

Kaplan, Allen P., MD

2009

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Factor XII–independent cleavage of high-molecular-weight kininogen by prekallikrein and inhibition by C1 inhibitor

Journal of allergy and clinical immunology, 2009-07, Vol.124 (1), p.143-149

2009

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Cytokine and Estrogen Stimulation of Endothelial Cells Augments Activation of the Surface-Bound Prekallikrein-High Molecular Weight Kininogen Complex: Implications for Hereditary Angioedema (HAE)

Journal of allergy and clinical immunology, 2016-02, Vol.137 (2), p.AB249-AB249

2016

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Rapid oral desensitization to sirolimus in a patient with lymphangioleiomyomatosis

The journal of allergy and clinical immunology in practice (Cambridge, MA), 2016-03, Vol.4 (2), p.352-353

2016

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Pathogenesis of Hereditary Angioedema with Normal C1 Inhibitor: Evidence for Abnormalities in Plasminogen Activator Inhibitors

Journal of allergy and clinical immunology, 2015-02, Vol.135 (2), p.AB277-AB277

2015

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Reply

Journal of allergy and clinical immunology, 2017

2017

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Response Patterns in Chronic Idiopathic/Spontaneous Urticaria (CIU/CSU) Patients Treated with Omalizumab for 24 Weeks in Two Randomized, Double-Blind, Placebo-Controlled Clinical Trials (ASTERIA I and GLACIAL)

Journal of allergy and clinical immunology, 2015-02, Vol.135 (2), p.AB127-AB127

2015

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Treatment of episodes of hereditary angioedema with C1 inhibitor: serial assessment of observed abnormalities of the plasma bradykinin-forming pathway and fibrinolysis

Annals of allergy, asthma, & immunology, 2010, Vol.104 (1), p.50-54

2010

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Omalizumab in patients with symptomatic chronic idiopathic/spontaneous urticaria despite standard combination therapy

A randomized, placebo-controlled, dose-ranging study of single-dose omalizumab in patients with H1 -antihistamine–refractory chronic idiopathic urticaria

Timing and duration of omalizumab response in patients with chronic idiopathic/spontaneous urticaria

Enzymatic pathways in the pathogenesis of hereditary angioedema: The role of C1 inhibitor therapy

Angioedema in the omalizumab chronic idiopathic/spontaneous urticaria pivotal studies

Therapy of chronic urticaria: a simple, modern approach

The bradykinin-forming cascade and its role in hereditary angioedema

Type III hereditary angioedema: defined, but not understood

Treatment of chronic autoimmune urticaria with omalizumab

Deficiency of plasminogen activator inhibitor 2 in plasma of patients with hereditary angioedema with normal C1 inhibitor levels

A Review of Childhood and Adolescent Craniopharyngiomas With Particular Attention to Hypothalamic Obesity

Development of a daily diary for patients with chronic idiopathic urticaria

Factor XII–independent activation of the bradykinin-forming cascade: Implications for the pathogenesis of hereditary angioedema types I and II

Fibrinolysis and bradykinin formation: From in vitro observations to human disease in “just” 45 years

Omalizumab is effective in nonautoimmune urticaria

Importance of angioedema-free days in patients with chronic idiopathic or spontaneous urticaria

Similar Efficacy with Omalizumab in Chronic Idiopathic/Spontaneous Urticaria Despite Different Background Therapy

What the first 10,000 patients with chronic urticaria have taught me: A personal journey

Factor XII–independent cleavage of high-molecular-weight kininogen by prekallikrein and inhibition by C1 inhibitor

Cytokine and Estrogen Stimulation of Endothelial Cells Augments Activation of the Surface-Bound Prekallikrein-High Molecular Weight Kininogen Complex: Implications for Hereditary Angioedema (HAE)

Rapid oral desensitization to sirolimus in a patient with lymphangioleiomyomatosis

Pathogenesis of Hereditary Angioedema with Normal C1 Inhibitor: Evidence for Abnormalities in Plasminogen Activator Inhibitors

Reply

Response Patterns in Chronic Idiopathic/Spontaneous Urticaria (CIU/CSU) Patients Treated with Omalizumab for 24 Weeks in Two Randomized, Double-Blind, Placebo-Controlled Clinical Trials (ASTERIA I and GLACIAL)

Treatment of episodes of hereditary angioedema with C1 inhibitor: serial assessment of observed abnormalities of the plasma bradykinin-forming pathway and fibrinolysis

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