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Context-Dependent and Disease-Specific Diversity in Protein Interactions within Stress Granules

Cell, 2018-01, Vol.172 (3), p.590-604.e13

2018

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Antibody Therapy Targeting RAN Proteins Rescues C9 ALS/FTD Phenotypes in C9orf72 Mouse Model

Neuron (Cambridge, Mass.), 2020-02, Vol.105 (4), p.645-662.e11

2020

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Production of poly(GA) in C9ORF72 patient motor neurons derived from induced pluripotent stem cells

Acta neuropathologica, 2019-12, Vol.138 (6), p.1099-1101

2019

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PINK1/Parkin Influences Cell Cycle by Sequestering TBK1 at Damaged Mitochondria, Inhibiting Mitosis

Cell reports (Cambridge), 2019-10, Vol.29 (1), p.225-235.e5

2019

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Poly(GR) and poly(GA) in cerebrospinal fluid as potential biomarkers for C9ORF72-ALS/FTD

Nature communications, 2022-05, Vol.13 (1), p.2799-2799, Article 2799

2022

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A C. elegans model of C9orf72-associated ALS/FTD uncovers a conserved role for eIF2D in RAN translation

Nature communications, 2021-10, Vol.12 (1), p.6025-17, Article 6025

2021

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Regulation of protein quality control by UBE4B and LSD1 through p53-mediated transcription

PLoS biology, 2015-04, Vol.13 (4), p.e1002114

2015

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The Notch signalling system: recent insights into the complexity of a conserved pathway

Nature reviews. Genetics, 2012-09, Vol.13 (9), p.654-666

2012

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Modeling spinal muscular atrophy in Drosophila

PloS one, 2008-09, Vol.3 (9), p.e3209-e3209

2008

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Arabidopsis MET1 Cytosine Methyltransferase Mutants

Genetics (Austin), 2003-03, Vol.163 (3), p.1109-1122

2003

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Exploring the genetics and non-cell autonomous mechanisms underlying ALS/FTLD

Cell death and differentiation, 2018-03, Vol.25 (4), p.646-662

2018

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Genetic circuitry of Survival motor neuron, the gene underlying spinal muscular atrophy

Proceedings of the National Academy of Sciences - PNAS, 2013-06, Vol.110 (26), p.E2371-E2380

2013

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Nodal points and complexity of Notch-Ras signal integration

Proceedings of the National Academy of Sciences - PNAS, 2009-02, Vol.106 (7), p.2218-2223

2009

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Investigating the Genetic Circuitry of Mastermind in Drosophila, a Notch Signal Effector

Genetics (Austin), 2007-12, Vol.177 (4), p.2493-2505

2007

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CRISPR deletion of the C9ORF72 promoter in ALS/FTD patient motor neurons abolishes production of dipeptide repeat proteins and rescues neurodegeneration

Acta neuropathologica, 2020-07, Vol.140 (1), p.81-84

2020

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Amyotrophic Lateral Sclerosis Modifiers in Drosophila Reveal the Phospholipase D Pathway as a Potential Therapeutic Target

Genetics (Austin), 2020-07, Vol.215 (3), p.747-766

2020

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Crossing paths with Notch in the hyper-network

Current opinion in cell biology, 2007-04, Vol.19 (2), p.166-175

2007

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Comprehensive evaluation of human-derived anti-poly-GA antibodies in cellular and animal models of C9orf72 disease

Nevada RNformation, 2022-12, Vol.119 (49), p.e2123487119

2022

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Modeling spinal muscular atrophy in Drosophila links Smn to FGF signaling

The Journal of cell biology, 2011-02, Vol.192 (3), p.481-495

2011

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Discovery of Phospholipase D Inhibitors with Improved Drug-like Properties and Central Nervous System Penetrance

ACS medicinal chemistry letters, 2022-04, Vol.13 (4), p.665-673

2022

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screen for genes that interact with the Drosophila pair-rule segmentation gene fushi tarazu

Genetics (Austin), 2004-09, Vol.168 (1), p.161-180

2004

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Regulation of Protein Quality Control by UBE4B and LSD1 through p53-Mediated Transcription: e1002114

PLoS biology, 2015-04, Vol.13 (4)

2015

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Context‐dependent and Disease‐specific Diversity in Stress Granules Formed from Pre‐existing Protein Interactions

The FASEB journal, 2018-04, Vol.32 (S1), p.252.3-252.3

2018

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Genetic circuitry of Survival motor neuron, the gene underlying spinal muscular atrophy

Proceedings of the National Academy of Sciences - PNAS, 2013-06, Vol.110 (26), p.10477-10477

2013

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Context-Dependent and Disease-Specific Diversity in Protein Interactions within Stress Granules

Antibody Therapy Targeting RAN Proteins Rescues C9 ALS/FTD Phenotypes in C9orf72 Mouse Model

Production of poly(GA) in C9ORF72 patient motor neurons derived from induced pluripotent stem cells

PINK1/Parkin Influences Cell Cycle by Sequestering TBK1 at Damaged Mitochondria, Inhibiting Mitosis

Poly(GR) and poly(GA) in cerebrospinal fluid as potential biomarkers for C9ORF72-ALS/FTD

A C. elegans model of C9orf72-associated ALS/FTD uncovers a conserved role for eIF2D in RAN translation

In vivo genome editing using novel AAV-PHP variants rescues motor function deficits and extends survival in a SOD1-ALS mouse model

Regulation of protein quality control by UBE4B and LSD1 through p53-mediated transcription

The Notch signalling system: recent insights into the complexity of a conserved pathway

Modeling spinal muscular atrophy in Drosophila

Arabidopsis MET1 Cytosine Methyltransferase Mutants

Exploring the genetics and non-cell autonomous mechanisms underlying ALS/FTLD

Genetic circuitry of Survival motor neuron, the gene underlying spinal muscular atrophy

Nodal points and complexity of Notch-Ras signal integration

Investigating the Genetic Circuitry of Mastermind in Drosophila, a Notch Signal Effector

CRISPR deletion of the C9ORF72 promoter in ALS/FTD patient motor neurons abolishes production of dipeptide repeat proteins and rescues neurodegeneration

Amyotrophic Lateral Sclerosis Modifiers in Drosophila Reveal the Phospholipase D Pathway as a Potential Therapeutic Target

Crossing paths with Notch in the hyper-network

Comprehensive evaluation of human-derived anti-poly-GA antibodies in cellular and animal models of C9orf72 disease

Modeling spinal muscular atrophy in Drosophila links Smn to FGF signaling

Discovery of Phospholipase D Inhibitors with Improved Drug-like Properties and Central Nervous System Penetrance

screen for genes that interact with the Drosophila pair-rule segmentation gene fushi tarazu

Regulation of Protein Quality Control by UBE4B and LSD1 through p53-Mediated Transcription: e1002114

Context‐dependent and Disease‐specific Diversity in Stress Granules Formed from Pre‐existing Protein Interactions

Genetic circuitry of Survival motor neuron, the gene underlying spinal muscular atrophy

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