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Two-stage electro–mechanical coupling of a KV channel in voltage-dependent activation

Nature communications, 2020-02, Vol.11 (1), p.676-676, Article 676

2020

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Structure and physiological function of the human KCNQ1 channel voltage sensor intermediate state

eLife, 2020-02, Vol.9

2020

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Towards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium Channels

Current molecular pharmacology, 2015-01, Vol.8 (2), p.188-205

2015

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Conformations of voltage-sensing domain III differentially define NaV channel closed- and open-state inactivation

The Journal of general physiology, 2021-09, Vol.153 (9), p.1

2021

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A Gain‐of‐Function Mutation in KCNMA1 Causes Dystonia Spells Controlled With Stimulant Therapy

Movement disorders, 2020-10, Vol.35 (10), p.1868-1873

2020

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Calmodulin acts as a state-dependent switch to control a cardiac potassium channel opening

Science advances, 2020-12, Vol.6 (50)

2020

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Dual activation of CFTR and CLCN2 by lubiprostone in murine nasal epithelia

American journal of physiology. Lung cellular and molecular physiology, 2013-03, Vol.304 (5), p.L324-L331

2013

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Aberrant QRS morphologies during atrial tachycardia: What is the mechanism?

Heart rhythm, 2023-07, Vol.20 (7), p.1061-1063

2023

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Elementary mechanisms of calmodulin regulation of NaV1.5 producing divergent arrhythmogenic phenotypes

Proceedings of the National Academy of Sciences - PNAS, 2021-05, Vol.118 (21), p.1-12

2021

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B-PO03-016 DISEASE-ASSOCIATED CALMODULIN VARIANTS INTERACTION WITH THE KCNQ1 POTASSIUM CHANNEL

Heart rhythm, 2021-08, Vol.18 (8), p.S195-S195

2021

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Aberrant Calmodulin Regulation of NaV1.5 Channels Linked to Inherited Cardiac Arrhythmia

Biophysical journal, 2020-02, Vol.118 (3), p.577a-577a

2020

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Disease associated CaM variants interact with KCNQ1 to confer aberrant membrane trafficking and function

Biophysical journal, 2022-02, Vol.121 (3), p.241a-241a

2022

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Mutations affecting electro-mechanical coupling in KCNQ channels alter kinetics and voltage dependence of activation

Biophysical journal, 2023-02, Vol.122 (3), p.386a-386a

2023

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Elementary mechanisms of calmodulin regulation of Na V 1.5 producing divergent arrhythmogenic phenotypes

Proceedings of the National Academy of Sciences - PNAS, 2021-05, Vol.118 (21)

2021

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Gating Mechanisms of Cardiac and Neuronal KCNQ Potassium Channels

Biophysical journal, 2020-02, Vol.118 (3), p.264a-264a

2020

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Mechanisms of KCNQ1-Calmodulin Channel Complex Activation to Dual Open States

Kang, Po wei

2023

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Structure and Physiological Function of the KCNQ1 Channel Voltage Sensor Intermediate State

Biophysical journal, 2020-02, Vol.118 (3), p.333a-333a

2020

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Calmodulin Functionally Couples the KCNQ1 Channel Voltage-Sensing and Pore Domains

Biophysical journal, 2020-02, Vol.118 (3), p.332a-333a

2020

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Modulating the voltage sensor of a cardiac potassium channel shows antiarrhythmic effects

Proceedings of the National Academy of Sciences - PNAS, 2021-05, Vol.118 (20), p.1-8

2021

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Robust, Automated Analysis of Electrophysiology in Induced Pluripotent Stem Cell-Derived Micro-Heart Muscle for Drug Toxicity

Tissue engineering. Part C, Methods, 2022-09, Vol.28 (9), p.457-468

2022

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Two-stage “Hand-and-Elbow” Gating Mechanism of a KV Channel

Biophysical journal, 2020-02, Vol.118 (3), p.113a-113a

2020

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Structure of the Intermediate State of the Human KCNQ1 Channel Voltage-Sensor Domain

Biophysical journal, 2019-02, Vol.116 (3), p.542a-542a

2019

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Prevention of ubiquitination at lysines in the N tail, R, ICL4, and NBD2 domains disrupts CFTR trafficking and immune response

The FASEB journal, 2013-04, Vol.27 (S1)

2013

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Mechanisms and models of cardiac sodium channel inactivation

Channels (Austin, Tex.), 2017-11, Vol.11 (6), p.517-533

2017

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Arrhythmia-associated calmodulin variants interact with KCNQ1 to confer aberrant membrane trafficking and function

PNAS nexus, 2023-11, Vol.2 (11), p.pgad335-pgad335

2023

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Two-stage electro–mechanical coupling of a KV channel in voltage-dependent activation

Structure and physiological function of the human KCNQ1 channel voltage sensor intermediate state

Towards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium Channels

Conformations of voltage-sensing domain III differentially define NaV channel closed- and open-state inactivation

A Gain‐of‐Function Mutation in KCNMA1 Causes Dystonia Spells Controlled With Stimulant Therapy

Calmodulin acts as a state-dependent switch to control a cardiac potassium channel opening

Dual activation of CFTR and CLCN2 by lubiprostone in murine nasal epithelia

Aberrant QRS morphologies during atrial tachycardia: What is the mechanism?

Elementary mechanisms of calmodulin regulation of NaV1.5 producing divergent arrhythmogenic phenotypes

B-PO03-016 DISEASE-ASSOCIATED CALMODULIN VARIANTS INTERACTION WITH THE KCNQ1 POTASSIUM CHANNEL

Aberrant Calmodulin Regulation of NaV1.5 Channels Linked to Inherited Cardiac Arrhythmia

Disease associated CaM variants interact with KCNQ1 to confer aberrant membrane trafficking and function

Mutations affecting electro-mechanical coupling in KCNQ channels alter kinetics and voltage dependence of activation

Elementary mechanisms of calmodulin regulation of Na V 1.5 producing divergent arrhythmogenic phenotypes

Gating Mechanisms of Cardiac and Neuronal KCNQ Potassium Channels

Mechanisms of KCNQ1-Calmodulin Channel Complex Activation to Dual Open States

Structure and Physiological Function of the KCNQ1 Channel Voltage Sensor Intermediate State

Calmodulin Functionally Couples the KCNQ1 Channel Voltage-Sensing and Pore Domains

Modulating the voltage sensor of a cardiac potassium channel shows antiarrhythmic effects

Robust, Automated Analysis of Electrophysiology in Induced Pluripotent Stem Cell-Derived Micro-Heart Muscle for Drug Toxicity

Two-stage “Hand-and-Elbow” Gating Mechanism of a KV Channel

Structure of the Intermediate State of the Human KCNQ1 Channel Voltage-Sensor Domain

Prevention of ubiquitination at lysines in the N tail, R, ICL4, and NBD2 domains disrupts CFTR trafficking and immune response

Mechanisms and models of cardiac sodium channel inactivation

Arrhythmia-associated calmodulin variants interact with KCNQ1 to confer aberrant membrane trafficking and function

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