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Subcellular optogenetic activation of Cdc42 controls local and distal signaling to drive immune cell migration

Molecular biology of the cell, 2016-05, Vol.27 (9), p.1442-1450

2016

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A photoswitchable GPCR-based opsin for presynaptic inhibition

Neuron (Cambridge, Mass.), 2021-06, Vol.109 (11), p.1791-1809.e11

2021

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Optically triggering spatiotemporally confined GPCR activity in a cell and programming neurite initiation and extension

Proceedings of the National Academy of Sciences - PNAS, 2013-04, Vol.110 (17), p.E1565-E1574

2013

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Shuttling of G Protein Subunits between the Plasma Membrane and Intracellular Membranes

The Journal of biological chemistry, 2007-08, Vol.282 (33), p.24092-24098

2007

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Parvalbumin expression does not account for discrete electrophysiological profiles of glutamatergic ventral pallidal subpopulations

Addiction neuroscience, 2024-09, Vol.12, p.100170, Article 100170

2024

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G-protein signaling leverages subunit-dependent membrane affinity to differentially control βγ translocation to intracellular membranes

Proceedings of the National Academy of Sciences - PNAS, 2012-12, Vol.109 (51), p.E3568-E3577

2012

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Regulation of Golgi structure and secretion by receptor-induced G protein βγ complex translocation

Proceedings of the National Academy of Sciences - PNAS, 2010-06, Vol.107 (25), p.11417-11422

2010

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Receptor-mediated Reversible Translocation of the G Protein βγ Complex from the Plasma Membrane to the Golgi Complex[boxs]

The Journal of biological chemistry, 2004-12, Vol.279 (49), p.51541-51544

2004

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G protein subunit dissociation and translocation regulate cellular response to receptor stimulation

PloS one, 2009-11, Vol.4 (11), p.e7797-e7797

2009

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Mu Opioid Receptor Knockout in Rostral Ventromedial Medulla Neurons Versus Presynaptic Terminals Has Divergent Effects on Acute Pain Phenotypes in Mice

The journal of pain, 2024-04, Vol.25 (4), p.17-17

2024

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Membrane Flow Drives an Adhesion-Independent Amoeboid Cell Migration Mode

Developmental cell, 2018-07, Vol.46 (1), p.9-22.e4

2018

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A Family of G Protein βγ Subunits Translocate Reversibly from the Plasma Membrane to Endomembranes on Receptor Activation

The Journal of biological chemistry, 2007-08, Vol.282 (33), p.24099-24108

2007

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G protein βγ complex translocation from plasma membrane to Golgi complex is influenced by receptor γ subunit interaction

Cellular signalling, 2006-10, Vol.18 (10), p.1758-1768

2006

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Optically triggering spatiotemporally confined GPCR activity in a cell and programming neurite initiation and extension

Proceedings of the National Academy of Sciences - PNAS, 2013-04, Vol.110 (17), p.6622-6622

2013

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Cell-Specific Single Viral Vector CRISPR/Cas9 Editing and Genetically Encoded Tool Delivery in the Central and Peripheral Nervous Systems

eNeuro, 2024-07, Vol.11 (7), p.ENEURO.0438-23.2024

2024

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All G protein βγ complexes are capable of translocation on receptor activation

Biochemical and biophysical research communications, 2012-05, Vol.421 (3), p.605-611

2012

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A G-Protein Subunit Translocation Embedded Network Motif Underlies GPCR Regulation of Calcium Oscillations

Biophysical journal, 2014-07, Vol.107 (1), p.242-254

2014

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RINm5f Cells Express Inactivating BK Channels Whereas HIT Cells Express Noninactivating BK Channels

Journal of neurophysiology, 1999-02, Vol.81 (2), p.611-624

1999

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G-protein signaling leverages subunit-dependent membrane affinity to differentially control [Beta][gamma] translocation to intracellular membranes

Proceedings of the National Academy of Sciences - PNAS, 2012-12, Vol.109 (51), p.E3568

2012

Volltextzugriff (PDF)

G-protein signaling leverages subunit-dependent membrane affinity to differentially control βγ translocation to intracellular membranes

Proceedings of the National Academy of Sciences - PNAS, 2012-12, Vol.109 (51), p.20784-20785

2012

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G protein βγ11 complex translocation is induced by Gi, Gq and Gs coupling receptors and is regulated by the α subunit type

Cellular signalling, 2006-08, Vol.18 (8), p.1190-1200

2006

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Alteration of Golgi structure in senescent cells and its regulation by a G protein γ subunit

Cellular signalling, 2011-05, Vol.23 (5), p.785-793

2011

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Alteration of Golgi structure in senescent cells and its regulation by a G protein [gamma] subunit

Cellular signalling, 2011-05, Vol.23 (5), p.785-793

2011

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Regulation of Golgi structure and secretion by receptor-induced G protein [Beta][gamma] complex translocation

Proceedings of the National Academy of Sciences - PNAS, 2010-06, Vol.107 (25), p.11417

2010

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Differential Ability to Form the G Protein βγ Complex among Members of the β and γ Subunit Families

The Journal of biological chemistry, 1996-03, Vol.271 (12), p.7141-7146

1996

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Subcellular optogenetic activation of Cdc42 controls local and distal signaling to drive immune cell migration

A photoswitchable GPCR-based opsin for presynaptic inhibition

Optically triggering spatiotemporally confined GPCR activity in a cell and programming neurite initiation and extension

Shuttling of G Protein Subunits between the Plasma Membrane and Intracellular Membranes

Parvalbumin expression does not account for discrete electrophysiological profiles of glutamatergic ventral pallidal subpopulations

G-protein signaling leverages subunit-dependent membrane affinity to differentially control βγ translocation to intracellular membranes

Regulation of Golgi structure and secretion by receptor-induced G protein βγ complex translocation

Receptor-mediated Reversible Translocation of the G Protein βγ Complex from the Plasma Membrane to the Golgi Complex[boxs]

G protein subunit dissociation and translocation regulate cellular response to receptor stimulation

Mu Opioid Receptor Knockout in Rostral Ventromedial Medulla Neurons Versus Presynaptic Terminals Has Divergent Effects on Acute Pain Phenotypes in Mice

Membrane Flow Drives an Adhesion-Independent Amoeboid Cell Migration Mode

A Family of G Protein βγ Subunits Translocate Reversibly from the Plasma Membrane to Endomembranes on Receptor Activation

G protein βγ complex translocation from plasma membrane to Golgi complex is influenced by receptor γ subunit interaction

Optically triggering spatiotemporally confined GPCR activity in a cell and programming neurite initiation and extension

Cell-Specific Single Viral Vector CRISPR/Cas9 Editing and Genetically Encoded Tool Delivery in the Central and Peripheral Nervous Systems

All G protein βγ complexes are capable of translocation on receptor activation

A G-Protein Subunit Translocation Embedded Network Motif Underlies GPCR Regulation of Calcium Oscillations

RINm5f Cells Express Inactivating BK Channels Whereas HIT Cells Express Noninactivating BK Channels

G-protein signaling leverages subunit-dependent membrane affinity to differentially control [Beta][gamma] translocation to intracellular membranes

G-protein signaling leverages subunit-dependent membrane affinity to differentially control βγ translocation to intracellular membranes

G protein βγ11 complex translocation is induced by Gi, Gq and Gs coupling receptors and is regulated by the α subunit type

Alteration of Golgi structure in senescent cells and its regulation by a G protein γ subunit

Alteration of Golgi structure in senescent cells and its regulation by a G protein [gamma] subunit

Regulation of Golgi structure and secretion by receptor-induced G protein [Beta][gamma] complex translocation

Differential Ability to Form the G Protein βγ Complex among Members of the β and γ Subunit Families

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