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Cancer and altered metabolism: potential importance of hypoxia-inducible factor and 2-oxoglutarate-dependent dioxygenases

Cold Spring Harbor Symposia on Quantitative Biology, 2011, Vol.76, p.335-345

Kaelin, Jr, W G

2011

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A phase I study of cabozantinib (XL184) in patients with renal cell cancer

Annals of oncology, 2014-08, Vol.25 (8), p.1603-1608

2014

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Rbx1, a Component of the VHL Tumor Suppressor Complex and SCF Ubiquitin Ligase

Science (American Association for the Advancement of Science), 1999-04, Vol.284 (5414), p.657-661

1999

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Structure of the VHL-ElonginC-ElonginB Complex: Implications for VHL Tumor Suppressor Function

Science (American Association for the Advancement of Science), 1999-04, Vol.284 (5413), p.455-461

1999

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The tyrosine kinase c-Abl regulates p73 in apoptotic response to cisplatin-induced DNA damage

Nature (London), 1999-06, Vol.399 (6738), p.806-809

1999

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Molecular basis of the VHL hereditary cancer syndrome

Nature reviews. Cancer, 2002-09, Vol.2 (9), p.673-682

Kaelin, William G

2002

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Role for the p53 homologue p73 in E2F-1-induced apoptosis

Nature (London), 2000-10, Vol.407 (6804), p.645-648

2000

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A common polymorphism acts as an intragenic modifier of mutant p53 behaviour

Nature genetics, 2000-05, Vol.25 (1), p.47-54

2000

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Negative Regulation of Hypoxia-Inducible Genes by the von Hippel--Lindau Protein

Proceedings of the National Academy of Sciences - PNAS, 1996-10, Vol.93 (20), p.10595-10599

1996

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The Emerging p53 Gene Family

JNCI : Journal of the National Cancer Institute, 1999-04, Vol.91 (7), p.594-598

KAELIN, W. G

1999

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The p53 gene family: p53

Oncogene, 1999-12, Vol.18 (53), p.7701-7705

KAELIN, W. G

1999

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The von Hippel–Lindau tumor suppressor protein

Current Opinion in Genetics & Development, 2001-02, Vol.11 (1), p.27-34

2001

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The von Hippel–Lindau protein, HIF hydroxylation, and oxygen sensing

Biochemical and biophysical research communications, 2005-12, Vol.338 (1), p.627-638

Kaelin, William G.

2005

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Role of the retinoblastoma protein in the pathogenesis of human cancer

Journal of clinical oncology, 1997-11, Vol.15 (11), p.3301-3312

1997

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Suppression of tumor growth through disruption of hypoxia-inducible transcription

Nature medicine, 2000-12, Vol.6 (12), p.1335-1340

2000

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p73 is a human p53-related protein that can induce apoptosis

Nature (London), 1997-09, Vol.389 (6647), p.191-194

1997

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Binding of the von Hippel-Lindau Tumor Suppressor Protein to Elongin B and C

Science (American Association for the Advancement of Science), 1995-09, Vol.269 (5229), p.1444-1446

1995

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The Elongin BC complex interacts with the conserved SOCS-box motif present in members of the SOCS, ras, WD-40 repeat, and ankyrin repeat families

Genes & development, 1998-12, Vol.12 (24), p.3872-3881

1998

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The VHL tumour-suppressor gene paradigm

Trends in genetics, 1998-10, Vol.14 (10), p.423-426

1998

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von Hippel-Lindau protein mutants linked to type 2C VHL disease preserve the ability to downregulate HIF

Human molecular genetics, 2001-05, Vol.10 (10), p.1019-1027

2001

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The von Hippel–Lindau Tumor Suppressor Gene

Experimental cell research, 2001-03, Vol.264 (1), p.117-125

2001

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Tumour suppression by the human von Hippel-Lindau gene product

Nature medicine, 1995-08, Vol.1 (8), p.822-826

1995

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Regulation of Hypoxia-Inducible mRNAs by the von Hippel-Lindau Tumor Suppressor Protein Requires Binding to Complexes Containing Elongins B/C and Cul2

Molecular and Cellular Biology, 1998-02, Vol.18 (2), p.732-741

1998

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Deregulated Transcription Factor E2F-1 Expression Leads to S-Phase Entry and p53-Mediated Apoptosis

Proceedings of the National Academy of Sciences - PNAS, 1994-11, Vol.91 (23), p.10918-10922

1994

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A Potent Transrepression Domain in the Retinoblastoma Protein Induces a Cell Cycle Arrest when Bound to E2F Sites

Proceedings of the National Academy of Sciences - PNAS, 1995-12, Vol.92 (25), p.11544-11548

1995

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Cancer and altered metabolism: potential importance of hypoxia-inducible factor and 2-oxoglutarate-dependent dioxygenases

A phase I study of cabozantinib (XL184) in patients with renal cell cancer

Rbx1, a Component of the VHL Tumor Suppressor Complex and SCF Ubiquitin Ligase

Structure of the VHL-ElonginC-ElonginB Complex: Implications for VHL Tumor Suppressor Function

The tyrosine kinase c-Abl regulates p73 in apoptotic response to cisplatin-induced DNA damage

Molecular basis of the VHL hereditary cancer syndrome

Role for the p53 homologue p73 in E2F-1-induced apoptosis

A common polymorphism acts as an intragenic modifier of mutant p53 behaviour

Negative Regulation of Hypoxia-Inducible Genes by the von Hippel--Lindau Protein

The Emerging p53 Gene Family

The p53 gene family: p53

The von Hippel–Lindau tumor suppressor protein

The von Hippel–Lindau protein, HIF hydroxylation, and oxygen sensing

Role of the retinoblastoma protein in the pathogenesis of human cancer

Suppression of tumor growth through disruption of hypoxia-inducible transcription

p73 is a human p53-related protein that can induce apoptosis

Binding of the von Hippel-Lindau Tumor Suppressor Protein to Elongin B and C

The Elongin BC complex interacts with the conserved SOCS-box motif present in members of the SOCS, ras, WD-40 repeat, and ankyrin repeat families

The VHL tumour-suppressor gene paradigm

von Hippel-Lindau protein mutants linked to type 2C VHL disease preserve the ability to downregulate HIF

The von Hippel–Lindau Tumor Suppressor Gene

Tumour suppression by the human von Hippel-Lindau gene product

Regulation of Hypoxia-Inducible mRNAs by the von Hippel-Lindau Tumor Suppressor Protein Requires Binding to Complexes Containing Elongins B/C and Cul2

Deregulated Transcription Factor E2F-1 Expression Leads to S-Phase Entry and p53-Mediated Apoptosis

A Potent Transrepression Domain in the Retinoblastoma Protein Induces a Cell Cycle Arrest when Bound to E2F Sites

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