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Colony Lysate Arrays for Proteomic Profiling of Drug-Tolerant Persisters of Cancer Cell

Analytical chemistry (Washington), 2017-09, Vol.89 (17), p.8626-8631

2017

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Individualized Mutation Detection in Circulating Tumor DNA for Monitoring Colorectal Tumor Burden Using a Cancer-Associated Gene Sequencing Panel

PloS one, 2016-01, Vol.11 (1), p.e0146275-e0146275

2016

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Identification and functional analysis of SVP ortholog in herbaceous perennial plant Gentiana triflora: Implication for its multifunctional roles

Plant science (Limerick), 2016-07, Vol.248, p.1-7

2016

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A compensatory role of NF-κB to p53 in response to 5-FU-based chemotherapy for gastric cancer cell lines

PloS one, 2014-02, Vol.9 (2), p.e90155

2014

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Inhibition of PI3K suppresses propagation of drug-tolerant cancer cell subpopulations enriched by 5-fluorouracil

Scientific reports, 2017-05, Vol.7 (1), p.2262-13, Article 2262

2017

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Systematic Protein Level Regulation via Degradation Machinery Induced by Genotoxic Drugs

Journal of proteome research, 2016-01, Vol.15 (1), p.205-215

2016

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TAS1 trans-Acting siRNA Targets Are Differentially Regulated at Low Temperature, and TAS1 trans-Acting siRNA Mediates Temperature-Controlled At1g51670 Expression

Bioscience, biotechnology, and biochemistry, 2010, Vol.74 (7), p.1435-1440

2010

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MYC to BCL6 State-Transitions Determine Cell Size and Metabolic Fluctuations and Define a Novel Biorhythm in B-Cell Malignancies

Blood, 2023-11, Vol.142 (Supplement 1), p.2769-2769

2023

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Molecular marker identification for relapse prediction in 5-FU-based adjuvant chemotherapy in gastric and colorectal cancers

PloS one, 2012-08, Vol.7 (8), p.e43236

2012

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Abstract 3951: Colony lysate arrays for proteomic profiling of drug-persistent cancer cell subpopulations

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.3951-3951

2017

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Evaluation of chemosensitivity prediction using quantitative dose-response curve classification for highly advanced/relapsed gastric cancer

World journal of surgical oncology, 2013-01, Vol.11 (1), p.11-11, Article 11

2013

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Immunoglobulin Light Chains Control Permissiveness to Malignant B-Cell Transformation By RAS-Pathway Lesions

Blood, 2023-11, Vol.142 (Supplement 1), p.2974-2974

2023

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Autonomous Ca2+ Oscillations Reflect Oncogenic Signaling in B-ALL Cells

Blood, 2019-11, Vol.134 (Supplement_1), p.1253-1253

2019

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SYK and ZAP70 kinases in autoimmunity and lymphoid malignancies

Cellular signalling, 2022-06, Vol.94, p.110331-110331, Article 110331

2022

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Autonomous Ca2+ Oscillations Reflect Oncogenic BCR-Signaling in Multiple B-Cell Malignancies and Are Essential for Survival and Proliferation

Blood, 2018-11, Vol.132 (Supplement 1), p.1373-1373

2018

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Abstract 4371: Store-operated Ca2+ entry and NFATC1 -activation control oncogenic signaling in B-cell transformation and leukemogenesis

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.4371-4371

2018

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Optogenetic Control of Oncogenic Signaling in B-Cell Malignancies

Blood, 2023-11, Vol.142 (Supplement 1), p.4138-4138

2023

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STAT5-Feedback Controls Distinct Metabolic States for Dynamic Transitions between Cellular Activation and Quiescence in Acute Lymphoblastic Leukemia

Blood, 2023-11, Vol.142 (Supplement 1), p.2977-2977

2023

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Computational Discovery and Validation of NAD+ Biosynthesis As Unique Vulnerability in B-Lymphoid Malignancies

Blood, 2023-11, Vol.142 (Supplement 1), p.418-418

2023

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Identification of BCL6 As Synthetic Lethality in RAS-Driven B-Cell Transformation

Blood, 2021-11, Vol.138 (Supplement 1), p.792-792

2021

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Pharmacological Targeting of PI3K-Dependent Central Tolerance Mechanisms in Refractory Pre-Germinal Center B-Cell Malignancies

Blood, 2021-11, Vol.138 (Supplement 1), p.2267-2267

2021

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Ca2+ Oscillations and NFATC1 -Activation Enable Oncogenic Signaling in Pre-B Cell Transformation and Leukemogenesis

Blood, 2017-12, Vol.130, p.2500-2500

2017

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Divergent MYC- and BCL6-Driven Metabolic Programs Enable Dynamic Regulation of Cell Biomass in B-Cell Malignancies

Blood, 2022-11, Vol.140 (Supplement 1), p.5900-5901

2022

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GSK3β Inhibition Is a Unique Vulnerability in Lymphoid Malignancies

Blood, 2022-11, Vol.140 (Supplement 1), p.3114-3115

2022

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BCL6-Mediated Escape from Negative Selection Enables RAS-Driven B-Cell Transformation

Blood, 2022-11, Vol.140 (Supplement 1), p.9177-9178

2022

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Colony Lysate Arrays for Proteomic Profiling of Drug-Tolerant Persisters of Cancer Cell

Individualized Mutation Detection in Circulating Tumor DNA for Monitoring Colorectal Tumor Burden Using a Cancer-Associated Gene Sequencing Panel

Identification and functional analysis of SVP ortholog in herbaceous perennial plant Gentiana triflora: Implication for its multifunctional roles

A compensatory role of NF-κB to p53 in response to 5-FU-based chemotherapy for gastric cancer cell lines

Inhibition of PI3K suppresses propagation of drug-tolerant cancer cell subpopulations enriched by 5-fluorouracil

Systematic Protein Level Regulation via Degradation Machinery Induced by Genotoxic Drugs

TAS1 trans-Acting siRNA Targets Are Differentially Regulated at Low Temperature, and TAS1 trans-Acting siRNA Mediates Temperature-Controlled At1g51670 Expression

MYC to BCL6 State-Transitions Determine Cell Size and Metabolic Fluctuations and Define a Novel Biorhythm in B-Cell Malignancies

Molecular marker identification for relapse prediction in 5-FU-based adjuvant chemotherapy in gastric and colorectal cancers

Abstract 3951: Colony lysate arrays for proteomic profiling of drug-persistent cancer cell subpopulations

Evaluation of chemosensitivity prediction using quantitative dose-response curve classification for highly advanced/relapsed gastric cancer

Immunoglobulin Light Chains Control Permissiveness to Malignant B-Cell Transformation By RAS-Pathway Lesions

Autonomous Ca2+ Oscillations Reflect Oncogenic Signaling in B-ALL Cells

SYK and ZAP70 kinases in autoimmunity and lymphoid malignancies

Autonomous Ca2+ Oscillations Reflect Oncogenic BCR-Signaling in Multiple B-Cell Malignancies and Are Essential for Survival and Proliferation

Abstract 4371: Store-operated Ca2+ entry and NFATC1 -activation control oncogenic signaling in B-cell transformation and leukemogenesis

Optogenetic Control of Oncogenic Signaling in B-Cell Malignancies

STAT5-Feedback Controls Distinct Metabolic States for Dynamic Transitions between Cellular Activation and Quiescence in Acute Lymphoblastic Leukemia

Computational Discovery and Validation of NAD+ Biosynthesis As Unique Vulnerability in B-Lymphoid Malignancies

Identification of BCL6 As Synthetic Lethality in RAS-Driven B-Cell Transformation

Pharmacological Targeting of PI3K-Dependent Central Tolerance Mechanisms in Refractory Pre-Germinal Center B-Cell Malignancies

Ca2+ Oscillations and NFATC1 -Activation Enable Oncogenic Signaling in Pre-B Cell Transformation and Leukemogenesis

Divergent MYC- and BCL6-Driven Metabolic Programs Enable Dynamic Regulation of Cell Biomass in B-Cell Malignancies

GSK3β Inhibition Is a Unique Vulnerability in Lymphoid Malignancies

BCL6-Mediated Escape from Negative Selection Enables RAS-Driven B-Cell Transformation

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