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Minocycline Reduces Proinflammatory Cytokine Expression, Microglial Activation, and Caspase-3 Activation in a Rodent Model of Diabetic Retinopathy

Diabetes (New York, N.Y.), 2005-05, Vol.54 (5), p.1559-1565

2005

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Interleukin-1: A master regulator of neuroinflammation

Journal of neuroscience research, 2004-10, Vol.78 (2), p.151-156

2004

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The PPAR-γ Agonist, Darglitazone, Restores Acute Inflammatory Responses to Cerebral Hypoxia–Ischemia in the Diabetic ob/ob Mouse

Journal of cerebral blood flow and metabolism, 2010-02, Vol.30 (2), p.352-360

2010

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Interleukin-1 and the interleukin-1 type 1 receptor are essential for the progressive neurodegeneration that ensues subsequent to a mild hypoxic ischemic injury

Journal of Cerebral Blood Flow & Metabolism, 2005-01, Vol.25 (1), p.17-29

2005

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Ciliary neurotrophic factor and interleukin-6 differentially activate microglia

Journal of neuroscience research, 2008-05, Vol.86 (7), p.1538-1547

2008

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Diabetic retinopathy : Seeing beyond glucose-induced microvascular disease

Diabetes (New York, N.Y.), 2006-09, Vol.55 (9), p.2401-2411

2006

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IGF-I and microglia/macrophage proliferation in the ischemic mouse brain

Glia, 2002-07, Vol.39 (1), p.85-97

2002

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Astrogliosis is delayed in type 1 interleukin-1 receptor-null mice following a penetrating brain injury

Journal of neuroinflammation, 2006-06, Vol.3 (1), p.15-15, Article 15

2006

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The Type 1 Interleukin-1 Receptor Is Essential for the Efficient Activation of Microglia and the Induction of Multiple Proinflammatory Mediators in Response to Brain Injury

The Journal of neuroscience, 2002-07, Vol.22 (14), p.6071-6082

2002

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Impaired wound healing after cerebral hypoxia-ischemia in the diabetic mouse

Journal of cerebral blood flow and metabolism, 2007-04, Vol.27 (4), p.710-718

2007

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Neuroinflammation and both cytotoxic and vasogenic edema are reduced in interleukin-1 type 1 receptor-deficient mice conferring neuroprotection

Stroke (1970), 2005-10, Vol.36 (10), p.2226-2231

2005

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Differential expression of protein tyrosine kinase genes during microglial activation

Glia, 2002-10, Vol.40 (1), p.11-24

2002

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Diabetic Retinopathy

Diabetes (New York, N.Y.), 2006-09, Vol.55 (9), p.2401-2411

2006

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The Ins2Akita Mouse as a Model of Early Retinal Complications in Diabetes

Investigative ophthalmology & visual science, 2005-06, Vol.46 (6), p.2210-2218

2005

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Localization and characterization of stannin: relationship to cellular sensitivity to organotin compounds

Neurochemistry international, 1997-12, Vol.31 (6), p.801-815

1997

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Transforming growth factor β1 prevents IL-1β-induced microglial activation, whereas TNFα- and IL-6-stimulated activation are not antagonized

Glia, 2002-10, Vol.40 (1), p.109-120

2002

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Seven cDNAs enriched following hippocampal lesion: possible roles in neuronal responses to injury

Brain research. Molecular brain research., 2003-09, Vol.117 (1), p.58-67

2003

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Astrocytic ceruloplasmin expression, which is induced by IL-1β and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor

Glia, 2003-10, Vol.44 (1), p.76-84

2003

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The PPAR-[gamma] agonist, darglitazone, restores acute inflammatory responses to cerebral hypoxia-ischemia in the diabetic ob/ob mouse

Journal of cerebral blood flow and metabolism, 2010-02, Vol.30 (2), p.352

2010

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Transcriptional activation by the parvoviral nonstructural protein NS-1 is mediated via a direct interaction with Sp1

Molecular and Cellular Biology, 1995-01, Vol.15 (1), p.524-533

1995

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Astrocytic ceruloplasmin expression, which is induced by IL-1beta and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor

Glia, 2003-10, Vol.44 (1), p.76-84

2003

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Transforming growth factor beta1 prevents IL-1beta-induced microglial activation, whereas TNFalpha- and IL-6-stimulated activation are not antagonized

Glia, 2002-10, Vol.40 (1), p.109

2002

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Molecular neurotoxicology of trimethyltin: identification of stannin, a novel protein expressed in trimethyltin-sensitive cells

Molecular pharmacology, 1992-07, Vol.42 (1), p.44-56

1992

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The Ins2 Akita Mouse as a Model of Early Retinal Complications in Diabetes

Investigative ophthalmology & visual science, 2005-06, Vol.46 (6), p.2210

2005

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Molecular Mechanisms of Selective Neurotoxicants: Studies on Organotin Compounds

Annals of the New York Academy of Sciences, 1993-05, Vol.679 (1), p.157-177

1993

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Minocycline Reduces Proinflammatory Cytokine Expression, Microglial Activation, and Caspase-3 Activation in a Rodent Model of Diabetic Retinopathy

Interleukin-1: A master regulator of neuroinflammation

The PPAR-γ Agonist, Darglitazone, Restores Acute Inflammatory Responses to Cerebral Hypoxia–Ischemia in the Diabetic ob/ob Mouse

Interleukin-1 and the interleukin-1 type 1 receptor are essential for the progressive neurodegeneration that ensues subsequent to a mild hypoxic ischemic injury

Ciliary neurotrophic factor and interleukin-6 differentially activate microglia

Diabetic retinopathy : Seeing beyond glucose-induced microvascular disease

IGF-I and microglia/macrophage proliferation in the ischemic mouse brain

Astrogliosis is delayed in type 1 interleukin-1 receptor-null mice following a penetrating brain injury

The Type 1 Interleukin-1 Receptor Is Essential for the Efficient Activation of Microglia and the Induction of Multiple Proinflammatory Mediators in Response to Brain Injury

Impaired wound healing after cerebral hypoxia-ischemia in the diabetic mouse

Neuroinflammation and both cytotoxic and vasogenic edema are reduced in interleukin-1 type 1 receptor-deficient mice conferring neuroprotection

Differential expression of protein tyrosine kinase genes during microglial activation

Diabetic Retinopathy

The Ins2Akita Mouse as a Model of Early Retinal Complications in Diabetes

Localization and characterization of stannin: relationship to cellular sensitivity to organotin compounds

Transforming growth factor β1 prevents IL-1β-induced microglial activation, whereas TNFα- and IL-6-stimulated activation are not antagonized

Seven cDNAs enriched following hippocampal lesion: possible roles in neuronal responses to injury

Astrocytic ceruloplasmin expression, which is induced by IL-1β and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor

The PPAR-[gamma] agonist, darglitazone, restores acute inflammatory responses to cerebral hypoxia-ischemia in the diabetic ob/ob mouse

Transcriptional activation by the parvoviral nonstructural protein NS-1 is mediated via a direct interaction with Sp1

Astrocytic ceruloplasmin expression, which is induced by IL-1beta and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor

Transforming growth factor beta1 prevents IL-1beta-induced microglial activation, whereas TNFalpha- and IL-6-stimulated activation are not antagonized

Molecular neurotoxicology of trimethyltin: identification of stannin, a novel protein expressed in trimethyltin-sensitive cells

The Ins2 Akita Mouse as a Model of Early Retinal Complications in Diabetes

Molecular Mechanisms of Selective Neurotoxicants: Studies on Organotin Compounds

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