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Differential regulation of BIRC2 and BIRC3 expression by inflammatory cytokines and glucocorticoids in pulmonary epithelial cells

PloS one, 2023-06, Vol.18 (6), p.e0286783-e0286783

2023

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Chemokine release from human rhinovirus–infected airway epithelial cells promotes fibroblast migration

Journal of allergy and clinical immunology, 2016-07, Vol.138 (1), p.114-122.e4

2016

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Human β-defensin-2 production upon viral and bacterial co-infection is attenuated in COPD

PloS one, 2017-05, Vol.12 (5), p.e0175963-e0175963

2017

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Rhinovirus Load Is High despite Preserved Interferon-β Response in Cystic Fibrosis Bronchial Epithelial Cells

PloS one, 2015-11, Vol.10 (11), p.e0143129-e0143129

2015

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Cigarette smoke modulates expression of human rhinovirus-induced airway epithelial host defense genes

PloS one, 2012-07, Vol.7 (7), p.e40762-e40762

2012

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A Complete Lipopolysaccharide Inner Core Oligosaccharide Is Required for Resistance of Burkholderia cenocepacia to Antimicrobial Peptides and Bacterial Survival In Vivo

Journal of Bacteriology, 2006-03, Vol.188 (6), p.2073-2080

2006

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Burkholderia cenocepacia Requires a Periplasmic HtrA Protease for Growth under Thermal and Osmotic Stress and for Survival In Vivo

Infection and Immunity, 2007-04, Vol.75 (4), p.1679-1689

2007

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An inhaled dose of budesonide induces genes involved in transcription and signaling in the human airways: enhancement of anti‐ and proinflammatory effector genes

Pharmacology research & perspectives, 2016-08, Vol.4 (4), p.e00243-n/a

2016

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Identification of Burkholderia cenocepacia Genes Required for Bacterial Survival In Vivo

Infection and Immunity, 2004-07, Vol.72 (7), p.4010-4022

2004

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NF‐κB and Glucocorticoid Receptor Synergize to Enable Pro‐inflammatory Signaling via Toll‐like Receptor 2 in Pulmonary Epithelial Cells

The FASEB journal, 2021-05, Vol.35 (S1), p.n/a, Article fasebj.2021.35.S1.05502

2021

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Synergy between Interleukin-1 β , Interferon- γ , and Glucocorticoids to Induce TLR2 Expression Involves NF- κ B, STAT1, and the Glucocorticoid Receptor

Molecular pharmacology, 2023-12, Vol.105 (1), p.23

2023

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β 2 -Adrenoceptor Agonists Promote Extracellular Signal-Regulated Kinase 1/2 Dephosphorylation in Human Airway Epithelial Cells by Canonical, cAMP-Driven Signaling Independently of β -Arrestin 2

Molecular pharmacology, 2021-10, Vol.100 (4), p.388-405

2021

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Interplay between nuclear factor-κB, p38 MAPK, and glucocorticoid receptor signaling synergistically induces functional TLR2 in lung epithelial cells

The Journal of biological chemistry, 2022-04, Vol.298 (4), p.101747-101747, Article 101747

2022

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Interplay between the Glucocorticoid Receptor and Inflammatory Signaling leads to Enhanced TLR2 Expression in Pulmonary Epithelial Cells

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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Human Rhinovirus Infection of Epithelial Cells Modulates Airway Smooth Muscle Migration

American journal of respiratory cell and molecular biology, 2017-06, Vol.56 (6), p.796-803

2017

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Inducible gene expression of IκB-kinase ε is dependent on nuclear factor-κB in human pulmonary epithelial cells

Biochemical journal, 2024-07, Vol.481 (14), p.959-980

2024

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Burkholderia cenocepacia zinc metalloproteases influence resistance to antimicrobial peptides

Microbiology (Society for General Microbiology), 2009-09, Vol.155 (Pt 9), p.2818

2009

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Rhinovirus and Bacteria Synergistically Induce IL-17C Release from Human Airway Epithelial Cells To Promote Neutrophil Recruitment

The Journal of immunology (1950), 2019-01, Vol.202 (1), p.160-170

2019

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Rhinovirus-bacteria coexposure synergistically induces CCL20 production from human bronchial epithelial cells

American journal of physiology. Lung cellular and molecular physiology, 2017-05, Vol.312 (5), p.L731-L740

2017

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Differential regulation of BIRC2 and BIRC3 expression by inflammatory cytokines and glucocorticoids in pulmonary epithelial cells

PloS one, 2023-01, Vol.18 (6), p.e0286783

2023

Volltextzugriff (PDF)

A complete lipopolysaccharide inner core oligosaccharide is required for resistance of Burkholderia cenocepacia to antimicrobial peptides and bacterial survival in vivo

Journal of bacteriology, 2006-03, Vol.188 (6), p.2073-2080

2006

Volltextzugriff (PDF)

Human [beta]-defensin-2 production upon viral and bacterial co-infection is attenuated in COPD.(Research Article)(chronic obstructive pulmonary disease)

PloS one, 2017-05, Vol.12 (5), p.e0175963

2017

Volltextzugriff (PDF)

Rhinovirus Load Is High despite Preserved Interferon-[beta] Response in Cystic Fibrosis Bronchial Epithelial Cells

PloS one, 2015-11, Vol.10 (11)

2015

Volltextzugriff (PDF)

Cyclic stretch augments human rhinovirus induced inflammatory responses in airway epithelial cells

Allergy, asthma, and clinical immunology, 2014-03, Vol.10 (S1), p.A71-A71, Article A71

2014

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Pre-existing human rhinovirus infection modulates host response to secondary bacterial infections

Allergy, asthma, and clinical immunology, 2014-03, Vol.10 (S1), p.A57-A57, Article A57

2014

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Differential regulation of BIRC2 and BIRC3 expression by inflammatory cytokines and glucocorticoids in pulmonary epithelial cells

Chemokine release from human rhinovirus–infected airway epithelial cells promotes fibroblast migration

Human β-defensin-2 production upon viral and bacterial co-infection is attenuated in COPD

Rhinovirus Load Is High despite Preserved Interferon-β Response in Cystic Fibrosis Bronchial Epithelial Cells

Cigarette smoke modulates expression of human rhinovirus-induced airway epithelial host defense genes

A Complete Lipopolysaccharide Inner Core Oligosaccharide Is Required for Resistance of Burkholderia cenocepacia to Antimicrobial Peptides and Bacterial Survival In Vivo

Burkholderia cenocepacia Requires a Periplasmic HtrA Protease for Growth under Thermal and Osmotic Stress and for Survival In Vivo

An inhaled dose of budesonide induces genes involved in transcription and signaling in the human airways: enhancement of anti‐ and proinflammatory effector genes

Identification of Burkholderia cenocepacia Genes Required for Bacterial Survival In Vivo

NF‐κB and Glucocorticoid Receptor Synergize to Enable Pro‐inflammatory Signaling via Toll‐like Receptor 2 in Pulmonary Epithelial Cells

Synergy between Interleukin-1 β , Interferon- γ , and Glucocorticoids to Induce TLR2 Expression Involves NF- κ B, STAT1, and the Glucocorticoid Receptor

β 2 -Adrenoceptor Agonists Promote Extracellular Signal-Regulated Kinase 1/2 Dephosphorylation in Human Airway Epithelial Cells by Canonical, cAMP-Driven Signaling Independently of β -Arrestin 2

Interplay between nuclear factor-κB, p38 MAPK, and glucocorticoid receptor signaling synergistically induces functional TLR2 in lung epithelial cells

Interplay between the Glucocorticoid Receptor and Inflammatory Signaling leads to Enhanced TLR2 Expression in Pulmonary Epithelial Cells

Human Rhinovirus Infection of Epithelial Cells Modulates Airway Smooth Muscle Migration

Inducible gene expression of IκB-kinase ε is dependent on nuclear factor-κB in human pulmonary epithelial cells

Burkholderia cenocepacia zinc metalloproteases influence resistance to antimicrobial peptides

Rhinovirus and Bacteria Synergistically Induce IL-17C Release from Human Airway Epithelial Cells To Promote Neutrophil Recruitment

Rhinovirus-bacteria coexposure synergistically induces CCL20 production from human bronchial epithelial cells

Differential regulation of BIRC2 and BIRC3 expression by inflammatory cytokines and glucocorticoids in pulmonary epithelial cells

A complete lipopolysaccharide inner core oligosaccharide is required for resistance of Burkholderia cenocepacia to antimicrobial peptides and bacterial survival in vivo

Human [beta]-defensin-2 production upon viral and bacterial co-infection is attenuated in COPD.(Research Article)(chronic obstructive pulmonary disease)

Rhinovirus Load Is High despite Preserved Interferon-[beta] Response in Cystic Fibrosis Bronchial Epithelial Cells

Cyclic stretch augments human rhinovirus induced inflammatory responses in airway epithelial cells

Pre-existing human rhinovirus infection modulates host response to secondary bacterial infections

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