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Isoflurane and Sevoflurane Induce Severe Hepatic Insulin Resistance in a Canine Model

PloS one, 2016-11, Vol.11 (11), p.e0163275

2016

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Abdominal Obesity: Role in the Pathophysiology of Metabolic Disease and Cardiovascular Risk

The American journal of medicine, 2007-02, Vol.120 (2), p.S3-S8

2007

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Why Visceral Fat is Bad: Mechanisms of the Metabolic Syndrome

Obesity (Silver Spring, Md.), 2006-02, Vol.14 (2S), p.16S-19S

2006

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Molecular evidence supporting the portal theory: a causative link between visceral adiposity and hepatic insulin resistance

American journal of physiology: endocrinology and metabolism, 2005-02, Vol.288 (2), p.E454-E461

2005

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Hepatic insulin clearance is the primary determinant of insulin sensitivity in the normal dog

Obesity (Silver Spring, Md.), 2014-05, Vol.22 (5), p.1238-1245

2014

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CB1R antagonist increases hepatic insulin clearance in fat-fed dogs likely via upregulation of liver adiponectin receptors

American journal of physiology: endocrinology and metabolism, 2015-10, Vol.309 (8), p.E747-E758

2015

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Metabolic Dysregulation With Atypical Antipsychotics Occurs in the Absence of Underlying Disease A Placebo-Controlled Study of Olanzapine and Risperidone in Dogs

Diabetes (New York, N.Y.), 2005-03, Vol.54 (3), p.862-871

2005

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Primacy of Hepatic Insulin Resistance in the Development of the Metabolic Syndrome Induced by an Isocaloric Moderate-Fat Diet in the Dog

Diabetes (New York, N.Y.), 2003-10, Vol.52 (10), p.2453-2460

2003

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Consistency of the disposition index in the face of diet induced insulin resistance: potential role of FFA

PloS one, 2011-03, Vol.6 (3), p.e18134-e18134

2011

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High-fat diet-induced insulin resistance does not increase plasma anandamide levels or potentiate anandamide insulinotropic effect in isolated canine islets

PloS one, 2015-04, Vol.10 (4), p.e0123558-e0123558

2015

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Corrections: High-Fat Diet-Induced Insulin Resistance does not Increase Plasma Anandamide Levels or Potentiate Anandamide Insulinotropic Effect in Isolated Canine Islets

PloS one, 2015-06, Vol.10 (6), p.e0131033-e0131033

2015

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Variability of Directly Measured First-Pass Hepatic Insulin Extraction and Its Association With Insulin Sensitivity and Plasma Insulin

Diabetes (New York, N.Y.), 2018-08, Vol.67 (8), p.1495-1503

2018

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Metabolic syndrome, hyperinsulinemia, and cancer

The American journal of clinical nutrition, 2007-09, Vol.86 (3), p.s867-871S

2007

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Systems analysis and the prediction and prevention of Type 2 diabetes mellitus

Current opinion in biotechnology, 2014-08, Vol.28, p.165-170

2014

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Assessment of hepatic insulin extraction from in vivo surrogate methods of insulin clearance measurement

American journal of physiology: endocrinology and metabolism, 2018-10, Vol.315 (4), p.E605-E612

2018

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Reduced Access to Insulin-Sensitive Tissues in Dogs With Obesity Secondary to Increased Fat Intake

Diabetes (New York, N.Y.), 2006-06, Vol.55 (6), p.1769-1775

2006

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Activation of NPRs and UCP1-independent pathway following CB1R antagonist treatment is associated with adipose tissue beiging in fat-fed male dogs

American journal of physiology: endocrinology and metabolism, 2019-09, Vol.317 (3), p.E535-E547

2019

Volltextzugriff (PDF)

Failure of Homeostatic Model Assessment of Insulin Resistance to Detect Marked Diet-Induced Insulin Resistance in Dogs

Diabetes (New York, N.Y.), 2014-06, Vol.63 (6), p.1914-1919

2014

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Central Role of the Adipocyte in the Metabolic Syndrome

Journal of investigative medicine, 2001-01, Vol.49 (1), p.119-126

2001

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beta-Cell "rest" accompanies reduced first-pass hepatic insulin extraction in the insulin-resistant, fat-fed canine model

American journal of physiology: endocrinology and metabolism, 2007-06, Vol.292 (6), p.E1581-E1589

2007

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Elevated nocturnal NEFA are an early signal for hyperinsulinaemic compensation during diet-induced insulin resistance in dogs

Diabetologia, 2015-11, Vol.58 (11), p.2663-2670

2015

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Nocturnal free fatty acids are uniquely elevated in the longitudinal development of diet-induced insulin resistance and hyperinsulinemia

American journal of physiology: endocrinology and metabolism, 2007-06, Vol.292 (6), p.E1590-E1598

2007

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Large Size Cells in the Visceral Adipose Depot Predict Insulin Resistance in the Canine Model

Obesity (Silver Spring, Md.), 2011-11, Vol.19 (11), p.2121-2129

2011

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Physiological Hyperinsulinemia in Dogs Augments Access of Macromolecules to Insulin-Sensitive Tissues

Diabetes (New York, N.Y.), 2004-11, Vol.53 (11), p.2741-2747

2004

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Increase in visceral fat per se does not induce insulin resistance in the canine model

Obesity (Silver Spring, Md.), 2015-01, Vol.23 (1), p.105-111

2015

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Isoflurane and Sevoflurane Induce Severe Hepatic Insulin Resistance in a Canine Model

Abdominal Obesity: Role in the Pathophysiology of Metabolic Disease and Cardiovascular Risk

Why Visceral Fat is Bad: Mechanisms of the Metabolic Syndrome

Molecular evidence supporting the portal theory: a causative link between visceral adiposity and hepatic insulin resistance

Hepatic insulin clearance is the primary determinant of insulin sensitivity in the normal dog

CB1R antagonist increases hepatic insulin clearance in fat-fed dogs likely via upregulation of liver adiponectin receptors

Metabolic Dysregulation With Atypical Antipsychotics Occurs in the Absence of Underlying Disease A Placebo-Controlled Study of Olanzapine and Risperidone in Dogs

Primacy of Hepatic Insulin Resistance in the Development of the Metabolic Syndrome Induced by an Isocaloric Moderate-Fat Diet in the Dog

Consistency of the disposition index in the face of diet induced insulin resistance: potential role of FFA

High-fat diet-induced insulin resistance does not increase plasma anandamide levels or potentiate anandamide insulinotropic effect in isolated canine islets

Corrections: High-Fat Diet-Induced Insulin Resistance does not Increase Plasma Anandamide Levels or Potentiate Anandamide Insulinotropic Effect in Isolated Canine Islets

Variability of Directly Measured First-Pass Hepatic Insulin Extraction and Its Association With Insulin Sensitivity and Plasma Insulin

Metabolic syndrome, hyperinsulinemia, and cancer

Systems analysis and the prediction and prevention of Type 2 diabetes mellitus

Assessment of hepatic insulin extraction from in vivo surrogate methods of insulin clearance measurement

Reduced Access to Insulin-Sensitive Tissues in Dogs With Obesity Secondary to Increased Fat Intake

Activation of NPRs and UCP1-independent pathway following CB1R antagonist treatment is associated with adipose tissue beiging in fat-fed male dogs

Failure of Homeostatic Model Assessment of Insulin Resistance to Detect Marked Diet-Induced Insulin Resistance in Dogs

Central Role of the Adipocyte in the Metabolic Syndrome

beta-Cell "rest" accompanies reduced first-pass hepatic insulin extraction in the insulin-resistant, fat-fed canine model

Elevated nocturnal NEFA are an early signal for hyperinsulinaemic compensation during diet-induced insulin resistance in dogs

Nocturnal free fatty acids are uniquely elevated in the longitudinal development of diet-induced insulin resistance and hyperinsulinemia

Large Size Cells in the Visceral Adipose Depot Predict Insulin Resistance in the Canine Model

Physiological Hyperinsulinemia in Dogs Augments Access of Macromolecules to Insulin-Sensitive Tissues

Increase in visceral fat per se does not induce insulin resistance in the canine model

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