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Glial connexins and gap junctions in CNS inflammation and disease

Journal of neurochemistry, 2008-08, Vol.106 (3), p.1000-1016

Kielian, Tammy

2008

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Toll-like receptors in central nervous system glial inflammation and homeostasis

Journal of neuroscience research, 2006-04, Vol.83 (5), p.711-730

Kielian, Tammy

2006

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Toll-like receptors in health and disease in the brain: mechanisms and therapeutic potential

Clinical science (1979), 2011-11, Vol.121 (9), p.367-387

2011

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Tumor necrosis factor regulates leukocyte recruitment but not bacterial persistence during Staphylococcus aureus craniotomy infection

Journal of neuroinflammation, 2024-07, Vol.21 (1), p.179-15, Article 179

2024

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Metabolism Shapes Immune Responses to Staphylococcus aureus

Journal of innate immunity, 2024-01, Vol.16 (1), p.12-30

2024

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Microglia in juvenile neuronal ceroid lipofuscinosis are primed toward a pro‐inflammatory phenotype

Journal of neurochemistry, 2013-10, Vol.127 (2), p.245-258

2013

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Toll-like receptor (TLR) and inflammasome actions in the central nervous system

Trends in immunology, 2012-07, Vol.33 (7), p.333-342

2012

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Critical role for the AIM2 inflammasome during acute CNS bacterial infection

Journal of neurochemistry, 2014-05, Vol.129 (4), p.704-711

2014

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Crosstalk Between Staphylococcus aureus and Innate Immunity: Focus on Immunometabolism

Frontiers in immunology, 2021-02, Vol.11, p.621750-621750

2021

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Neuroinflammatory paradigms in lysosomal storage diseases

Frontiers in neuroscience, 2015-10, Vol.9, p.417

2015

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Neuroinflammation leads to region-dependent alterations in astrocyte gap junction communication and hemichannel activity

The Journal of neuroscience, 2011-01, Vol.31 (2), p.414-425

2011

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Immunopathogenesis of brain abscess

Journal of neuroinflammation, 2004-08, Vol.1 (1), p.16-16

Kielian, Tammy

2004

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Myeloid-derived suppressor cells contribute to Staphylococcus aureus orthopedic biofilm infection

The Journal of immunology (1950), 2014-04, Vol.192 (8), p.3778-3792

2014

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Tissue niche influences immune and metabolic profiles to Staphylococcus aureus biofilm infection

Nature communications, 2024-10, Vol.15 (1), p.8965-20, Article 8965

2024

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Urease is an essential component of the acid response network of Staphylococcus aureus and is required for a persistent murine kidney infection

PLoS pathogens, 2019-01, Vol.15 (1), p.e1007538-e1007538

2019

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Monocyte metabolic reprogramming promotes pro-inflammatory activity and Staphylococcus aureus biofilm clearance

PLoS pathogens, 2020-03, Vol.16 (3), p.e1008354

2020

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Lactate production by Staphylococcus aureus biofilm inhibits HDAC11 to reprogramme the host immune response during persistent infection

Nature microbiology, 2020-10, Vol.5 (10), p.1271-1284

2020

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The YIN and YANG of lipoproteins in developing and preventing infectious arthritis by Staphylococcus aureus

PLoS pathogens, 2019-06, Vol.15 (6), p.e1007877-e1007877

2019

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C1q is elevated during chronic Staphylococcus epidermidis central nervous system catheter infection

Frontiers in immunology, 2024-05, Vol.15, p.1342467

2024

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Neutrophils are mediators of metastatic prostate cancer progression in bone

Cancer Immunology, Immunotherapy, 2020-06, Vol.69 (6), p.1113-1130

2020

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Induction of type I interferon signaling determines the relative pathogenicity of Staphylococcus aureus strains

PLoS pathogens, 2014-02, Vol.10 (2), p.e1003951-e1003951

2014

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MyD88-dependent signaling influences fibrosis and alternative macrophage activation during Staphylococcus aureus biofilm infection

PloS one, 2012-08, Vol.7 (8), p.e42476-e42476

2012

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Age-dependent alterations in neuronal activity in the hippocampus and visual cortex in a mouse model of Juvenile Neuronal Ceroid Lipofuscinosis (CLN3)

Neurobiology of disease, 2017-04, Vol.100, p.19-29

2017

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Lysosomal storage disorders: pathology within the lysosome and beyond

Journal of neurochemistry, 2019-03, Vol.148 (5), p.568-572

Kielian, Tammy

2019

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Staphylococcus aureus Biofilms Induce Macrophage Dysfunction Through Leukocidin AB and Alpha-Toxin

mBio, 2015-08, Vol.6 (4)

2015

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Glial connexins and gap junctions in CNS inflammation and disease

Toll-like receptors in central nervous system glial inflammation and homeostasis

Toll-like receptors in health and disease in the brain: mechanisms and therapeutic potential

Tumor necrosis factor regulates leukocyte recruitment but not bacterial persistence during Staphylococcus aureus craniotomy infection

Metabolism Shapes Immune Responses to Staphylococcus aureus

Microglia in juvenile neuronal ceroid lipofuscinosis are primed toward a pro‐inflammatory phenotype

Toll-like receptor (TLR) and inflammasome actions in the central nervous system

Critical role for the AIM2 inflammasome during acute CNS bacterial infection

Crosstalk Between Staphylococcus aureus and Innate Immunity: Focus on Immunometabolism

Neuroinflammatory paradigms in lysosomal storage diseases

Neuroinflammation leads to region-dependent alterations in astrocyte gap junction communication and hemichannel activity

Immunopathogenesis of brain abscess

Myeloid-derived suppressor cells contribute to Staphylococcus aureus orthopedic biofilm infection

Tissue niche influences immune and metabolic profiles to Staphylococcus aureus biofilm infection

Urease is an essential component of the acid response network of Staphylococcus aureus and is required for a persistent murine kidney infection

Monocyte metabolic reprogramming promotes pro-inflammatory activity and Staphylococcus aureus biofilm clearance

Lactate production by Staphylococcus aureus biofilm inhibits HDAC11 to reprogramme the host immune response during persistent infection

The YIN and YANG of lipoproteins in developing and preventing infectious arthritis by Staphylococcus aureus

C1q is elevated during chronic Staphylococcus epidermidis central nervous system catheter infection

Neutrophils are mediators of metastatic prostate cancer progression in bone

Induction of type I interferon signaling determines the relative pathogenicity of Staphylococcus aureus strains

MyD88-dependent signaling influences fibrosis and alternative macrophage activation during Staphylococcus aureus biofilm infection

Age-dependent alterations in neuronal activity in the hippocampus and visual cortex in a mouse model of Juvenile Neuronal Ceroid Lipofuscinosis (CLN3)

Lysosomal storage disorders: pathology within the lysosome and beyond

Staphylococcus aureus Biofilms Induce Macrophage Dysfunction Through Leukocidin AB and Alpha-Toxin

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