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Tumor-induced immune dysfunction: the macrophage connection

Journal of leukocyte biology, 1998-09, Vol.64 (3), p.275-290

1998

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Tumor-induced regulation of suppressor macrophage nitric oxide and TNF-alpha production. Role of tumor-derived IL-10, TGF-beta, and prostaglandin E2

The Journal of immunology (1950), 1994-08, Vol.153 (4), p.1674-1686

1994

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Paclitaxel Enhances Macrophage IL-12 Production in Tumor-Bearing Hosts Through Nitric Oxide

The Journal of immunology (1950), 1999-06, Vol.162 (11), p.6811-6818

1999

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Fibrosarcoma-induced increase in macrophage tumor necrosis factor alpha synthesis suppresses T cell responses

Journal of leukocyte biology, 1993-08, Vol.54 (2), p.152-160

1993

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Tumor growth increases Ia- macrophage synthesis of tumor necrosis factor-alpha and prostaglandin E2: changes in macrophage suppressor activity

Journal of leukocyte biology, 1993-05, Vol.53 (5), p.550-558

1993

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Taxol-mediated changes in fibrosarcoma-induced immune cell function : modulation of antitumor activities

Cancer Immunology, Immunotherapy, 1997-10, Vol.45 (1), p.20-28

1997

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Induction of macrophage suppressor activity by fibrosarcoma-derived transforming growth factor-beta 1: contrasting effects on resting and activated macrophages

Journal of leukocyte biology, 1995-06, Vol.57 (6), p.919-928

1995

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Tumour Growth Causes Suppression of Autoreactive T‐Cell Proliferation by Disrupting Macrophage Responsiveness to Interferon‐γ

Scandinavian journal of immunology, 1994-01, Vol.39 (1), p.31-38

1994

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Interleukin-12 Overcomes Paclitaxel-Mediated Suppression of T-Cell Proliferation

Immunopharmacology and immunotoxicology, 1998-01, Vol.20 (4), p.473-492

1998

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Cytokines and suppressor macrophages cause tumor-bearing host CD8+ T cells to suppress recognition of allogeneic and syngeneic MHC class II molecules

Journal of leukocyte biology, 1992-12, Vol.52 (6), p.661-669

1992

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Tumor growth modulates macrophage nitric oxide production following paclitaxel administration

International journal of immunopharmacology, 1998-10, Vol.20 (10), p.537-551

1998

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Variations in macrophage antigen phenotype: a correlation between Ia antigen reduction and immune dysfunction during tumor growth

Journal of leukocyte biology, 1986-11, Vol.40 (5), p.561-574

1986

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Prostaglandin E2 production by Mac-2+ macrophages: tumor-induced population shift

Journal of leukocyte biology, 1987-12, Vol.42 (6), p.673-681

1987

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Tumor Growth Alters T Cell and Macrophage Production of and Responsiveness to Granulocyte-Macrophage Colony-Stimulating Factor: Partial Dysregulation through Interleukin-10

Cellular immunology, 1994-04, Vol.154 (2), p.342-357

1994

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Tumor cell-derived TGF-β and IL-10 dysregulate paclitaxel-induced macrophage activation

Journal of leukocyte biology, 2001-01, Vol.69 (1), p.129-137

2001

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Changes in splenic macrophage Mac antigen expression during tumor growth: a kinetic study of accessory cell function and antigen-defined phenotypes

Journal of leukocyte biology, 1986-12, Vol.40 (6), p.709-724

1986

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Tumor growth and adherence change the expression of macrophage Mac-2

Cancer letters, 1993-04, Vol.69 (1), p.67-74

1993

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Tumor-Induced Macrophage Tumor Necrosis Factor-α Production Suppresses Autoreactive T Cell Proliferation

Immunobiology (1979), 1993-08, Vol.188 (4), p.430-445

1993

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Tumor modulation of autoreactivity: Decreased macrophage and autoreactive T cell interactions

Cellular immunology, 1990-04, Vol.127 (1), p.105-119

1990

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Tumor-induced alteration in macrophage accessory cell activity on autoreactive T cells

Cancer Immunology Immunotherapy, 1989-05, Vol.30 (3), p.170-176

1989

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Changes in macrophage populations: phenotypic differences between normal and tumor-bearing host macrophages

Immunobiology (1979), 1989-02, Vol.178 (4-5), p.416-435

1989

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Tumor growth changes the contribution of granulocyte-macrophage colony-stimulating factor during macrophage-mediated suppression of allorecognition

Immunobiology (1979), 1992-09, Vol.185 (5), p.427-439

1992

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Is celiac disease due to molecular mimicry between gliadin peptide-HLA class II molecule-T cell interactions and those of some unidentified superantigen?

Molecular immunology, 1997-05, Vol.34 (7), p.535-541

1997

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Shifts in macrophage (Mφ) surface phenotypes during tumor growth: Association of Mac-2 + and Mac-3 + Mφ with immunosuppressive activity

Cellular immunology, 1987-09, Vol.108 (2), p.255-268

1987

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Tumor Growth Alters Macrophage Responsiveness to Macrophage Colony-Stimulating Factor During Reactivity Against Allogeneic and Syngeneic Mhc Class II Molecules

Immunological investigations, 1993-01, Vol.22 (6-7), p.463-476

1993

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Tumor-induced immune dysfunction: the macrophage connection

Tumor-induced regulation of suppressor macrophage nitric oxide and TNF-alpha production. Role of tumor-derived IL-10, TGF-beta, and prostaglandin E2

Paclitaxel Enhances Macrophage IL-12 Production in Tumor-Bearing Hosts Through Nitric Oxide

Fibrosarcoma-induced increase in macrophage tumor necrosis factor alpha synthesis suppresses T cell responses

Tumor growth increases Ia- macrophage synthesis of tumor necrosis factor-alpha and prostaglandin E2: changes in macrophage suppressor activity

Taxol-mediated changes in fibrosarcoma-induced immune cell function : modulation of antitumor activities

Induction of macrophage suppressor activity by fibrosarcoma-derived transforming growth factor-beta 1: contrasting effects on resting and activated macrophages

Tumour Growth Causes Suppression of Autoreactive T‐Cell Proliferation by Disrupting Macrophage Responsiveness to Interferon‐γ

Interleukin-12 Overcomes Paclitaxel-Mediated Suppression of T-Cell Proliferation

Cytokines and suppressor macrophages cause tumor-bearing host CD8+ T cells to suppress recognition of allogeneic and syngeneic MHC class II molecules

Tumor growth modulates macrophage nitric oxide production following paclitaxel administration

Variations in macrophage antigen phenotype: a correlation between Ia antigen reduction and immune dysfunction during tumor growth

Prostaglandin E2 production by Mac-2+ macrophages: tumor-induced population shift

Tumor Growth Alters T Cell and Macrophage Production of and Responsiveness to Granulocyte-Macrophage Colony-Stimulating Factor: Partial Dysregulation through Interleukin-10

Tumor cell-derived TGF-β and IL-10 dysregulate paclitaxel-induced macrophage activation

Changes in splenic macrophage Mac antigen expression during tumor growth: a kinetic study of accessory cell function and antigen-defined phenotypes

Tumor growth and adherence change the expression of macrophage Mac-2

Tumor-Induced Macrophage Tumor Necrosis Factor-α Production Suppresses Autoreactive T Cell Proliferation

Tumor modulation of autoreactivity: Decreased macrophage and autoreactive T cell interactions

Tumor-induced alteration in macrophage accessory cell activity on autoreactive T cells

Changes in macrophage populations: phenotypic differences between normal and tumor-bearing host macrophages

Tumor growth changes the contribution of granulocyte-macrophage colony-stimulating factor during macrophage-mediated suppression of allorecognition

Is celiac disease due to molecular mimicry between gliadin peptide-HLA class II molecule-T cell interactions and those of some unidentified superantigen?

Shifts in macrophage (Mφ) surface phenotypes during tumor growth: Association of Mac-2 + and Mac-3 + Mφ with immunosuppressive activity

Tumor Growth Alters Macrophage Responsiveness to Macrophage Colony-Stimulating Factor During Reactivity Against Allogeneic and Syngeneic Mhc Class II Molecules

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