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PPARγ and TGFβ—Major Regulators of Metabolism, Inflammation, and Fibrosis in the Lungs and Kidneys

International journal of molecular sciences, 2021-10, Vol.22 (19), p.10431

2021

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The PPARγ agonist pioglitazone prevents TGF-β induced renal fibrosis by repressing EGR-1 and STAT3

BMC nephrology, 2019-07, Vol.20 (1), p.245-245, Article 245

2019

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Pioglitazone Protects Tubular Epithelial Cells during Kidney Fibrosis by Attenuating miRNA Dysregulation and Autophagy Dysfunction Induced by TGF-β

International journal of molecular sciences, 2023-11, Vol.24 (21), p.15520

2023

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HK-2 cell response to TGF-β highly depends on cell culture medium formulations

Histochemistry and cell biology, 2024-01, Vol.161 (1), p.69-79

2024

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PPARγ is a gatekeeper for extracellular matrix and vascular cell homeostasis: beneficial role in pulmonary hypertension and renal/cardiac/pulmonary fibrosis

Current opinion in nephrology and hypertension, 2020-03, Vol.29 (2), p.171-179

2020

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Editorial: New Advances in Cardiorenal Syndrome

Frontiers in cardiovascular medicine, 2022-07, Vol.9, p.976846-976846

2022

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Susceptibility to kidney fibrosis in mice is associated with early growth response-2 protein and tissue inhibitor of metalloproteinase-1 expression

Kidney international, 2022-08, Vol.102 (2), p.337-354

2022

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Selective phosphodiesterase-5 (PDE-5) inhibitor vardenafil ameliorates renal damage in type 1 diabetic rats by restoring cyclic 3′,5′ guanosine monophosphate (cGMP) level in podocytes

Nephrology, dialysis, transplantation, 2013-07, Vol.28 (7), p.1751-1761

2013

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Cinaciguat ameliorates glomerular damage by reducing ERK1/2 activity and TGF-ß expression in type-1 diabetic rats

Scientific reports, 2017-09, Vol.7 (1), p.11218-15, Article 11218

2017

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Oxidative/Nitrative Stress and Inflammation Drive Progression of Doxorubicin-Induced Renal Fibrosis in Rats as Revealed by Comparing a Normal and a Fibrosis-Resistant Rat Strain

PloS one, 2015-06, Vol.10 (6), p.e0127090-e0127090

2015

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Sustained hyperosmolarity increses TGF-ß1 and Egr-1 expression in the rat renal medulla

BMC nephrology, 2017-07, Vol.18 (1), p.209-209, Article 209

2017

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Characterization and Role of SCAI during Renal Fibrosis and Epithelial-to-Mesenchymal Transition

The American journal of pathology, 2013-02, Vol.182 (2), p.388-400

2013

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Cinaciguat prevents the development of pathologic hypertrophy in a rat model of left ventricular pressure overload

Scientific reports, 2016-11, Vol.6 (1), p.37166-37166, Article 37166

2016

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The Effect of Combined Treatment with the (Pro)Renin Receptor Blocker HRP and Quinapril in Type 1 Diabetic Rats

Kidney & blood pressure research, 2017-01, Vol.42 (1), p.109-122

2017

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2222 Suppression of miR-199a-3p ameliorates TGF-β-induced EMT in renal tubular cells

Nephrology, dialysis, transplantation, 2024-05, Vol.39 (Supplement_1)

2024

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Simple, readily available clinical indices predict early and late mortality among patients with ANCA-associated vasculitis

BMC nephrology, 2017-02, Vol.18 (1), p.76-76, Article 76

2017

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Alterations in SCAI Expression during Cell Plasticity, Fibrosis and Cancer

Pathology oncology research, 2018-07, Vol.24 (3), p.641-651

2018

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Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice

American journal of physiology. Heart and circulatory physiology, 2010-08, Vol.299 (2), p.H386-H395

2010

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GENETIC SUSCEPTIBILITY TO EARLY ONSET OF TRANSFORMING GROWTH FACTOR-BETA DRIVEN CARDIAC REMODELING IN MICE IS ASSOCIATED WITH EGR1 AND STAT3

Journal of hypertension, 2021-04, Vol.39 (Supplement 1), p.e262

2021

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QUINAPRIL AMELIORATES CARDIAC REMODELING IN DIABETIC RATS VIA SUPPRESSION OF EGR1 AND STAT3 ACTIVATION

Journal of hypertension, 2021-04, Vol.39 (Supplement 1), p.e210

2021

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Prevention of the development of heart failure with preserved ejection fraction by the phosphodiesterase‐5A inhibitor vardenafil in rats with type 2 diabetes

European journal of heart failure, 2017-03, Vol.19 (3), p.326-336

2017

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Susceptibility to kidney fibrosis in mice is associated with early growth response-2 protein and tissue inhibitor of metaloproteinase-1 expression

Kidney international, 2022-05, Vol.102 (2), p.337-354

2022

Volltextzugriff (PDF)

Selective phosphodiesterase-5 (PDE-5) inhibitor vardenafil ameliorates renal damage in type 1 diabetic rats by restoring cyclic 3',5' guanosine monophosphate (cGMP) level in podocytes

Nephrology, dialysis, transplantation, 2013-07, Vol.28 (7), p.1751-1761

2013

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SP320OXIDATIVE/NITRATIVE STRESS AND INFLAMMATION DRIVE PROGRESSION OF DOXORUBICIN-INDUCED RENAL FIBROSIS IN RATS AS REVEALED BY COMPARING A NORMAL AND A FIBROSIS-RESISTANT RAT STRAIN

Nephrology, dialysis, transplantation, 2015-05, Vol.30 (suppl_3), p.iii485-iii485

2015

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Mechanisms behind flare of renal lupus during murine pregnancy

Reproductive biomedicine online, 2008, Vol.17 (1), p.114-126

2008

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PPARγ and TGFβ—Major Regulators of Metabolism, Inflammation, and Fibrosis in the Lungs and Kidneys

The PPARγ agonist pioglitazone prevents TGF-β induced renal fibrosis by repressing EGR-1 and STAT3

Pioglitazone Protects Tubular Epithelial Cells during Kidney Fibrosis by Attenuating miRNA Dysregulation and Autophagy Dysfunction Induced by TGF-β

HK-2 cell response to TGF-β highly depends on cell culture medium formulations

PPARγ is a gatekeeper for extracellular matrix and vascular cell homeostasis: beneficial role in pulmonary hypertension and renal/cardiac/pulmonary fibrosis

Editorial: New Advances in Cardiorenal Syndrome

Susceptibility to kidney fibrosis in mice is associated with early growth response-2 protein and tissue inhibitor of metalloproteinase-1 expression

Selective phosphodiesterase-5 (PDE-5) inhibitor vardenafil ameliorates renal damage in type 1 diabetic rats by restoring cyclic 3′,5′ guanosine monophosphate (cGMP) level in podocytes

Cinaciguat ameliorates glomerular damage by reducing ERK1/2 activity and TGF-ß expression in type-1 diabetic rats

Oxidative/Nitrative Stress and Inflammation Drive Progression of Doxorubicin-Induced Renal Fibrosis in Rats as Revealed by Comparing a Normal and a Fibrosis-Resistant Rat Strain

Sustained hyperosmolarity increses TGF-ß1 and Egr-1 expression in the rat renal medulla

Characterization and Role of SCAI during Renal Fibrosis and Epithelial-to-Mesenchymal Transition

Cinaciguat prevents the development of pathologic hypertrophy in a rat model of left ventricular pressure overload

The Effect of Combined Treatment with the (Pro)Renin Receptor Blocker HRP and Quinapril in Type 1 Diabetic Rats

2222 Suppression of miR-199a-3p ameliorates TGF-β-induced EMT in renal tubular cells

Simple, readily available clinical indices predict early and late mortality among patients with ANCA-associated vasculitis

Alterations in SCAI Expression during Cell Plasticity, Fibrosis and Cancer

Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice

GENETIC SUSCEPTIBILITY TO EARLY ONSET OF TRANSFORMING GROWTH FACTOR-BETA DRIVEN CARDIAC REMODELING IN MICE IS ASSOCIATED WITH EGR1 AND STAT3

QUINAPRIL AMELIORATES CARDIAC REMODELING IN DIABETIC RATS VIA SUPPRESSION OF EGR1 AND STAT3 ACTIVATION

Prevention of the development of heart failure with preserved ejection fraction by the phosphodiesterase‐5A inhibitor vardenafil in rats with type 2 diabetes

Susceptibility to kidney fibrosis in mice is associated with early growth response-2 protein and tissue inhibitor of metaloproteinase-1 expression

Selective phosphodiesterase-5 (PDE-5) inhibitor vardenafil ameliorates renal damage in type 1 diabetic rats by restoring cyclic 3',5' guanosine monophosphate (cGMP) level in podocytes

SP320OXIDATIVE/NITRATIVE STRESS AND INFLAMMATION DRIVE PROGRESSION OF DOXORUBICIN-INDUCED RENAL FIBROSIS IN RATS AS REVEALED BY COMPARING A NORMAL AND A FIBROSIS-RESISTANT RAT STRAIN

Mechanisms behind flare of renal lupus during murine pregnancy

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