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Distinct merkel cell polyomavirus molecular features in tumour and non tumour specimens from patients with merkel cell carcinoma

PLoS pathogens, 2010-08, Vol.6 (8), p.e1001076-e1001076

2010

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Regulation of senescence escape by the cdk4-EZH2-AP2M1 pathway in response to chemotherapy

Cell death & disease, 2018-02, Vol.9 (2), p.199-15, Article 199

2018

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Phenyl‐Glutarimides: Alternative Cereblon Binders for the Design of PROTACs

Angewandte Chemie International Edition, 2021-12, Vol.60 (51), p.26663-26670

International ed. in English, 2021

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Patient-derived models recapitulate heterogeneity of molecular signatures and drug response in pediatric high-grade glioma

Nature communications, 2021-07, Vol.12 (1), p.1-17, Article 4089

2021

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Akt inhibition improves irinotecan treatment and prevents cell emergence by switching the senescence response to apoptosis

Oncotarget, 2015-12, Vol.6 (41), p.43342-43362

2015

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Irinotecan treatment and senescence failure promote the emergence of more transformed and invasive cells that depend on anti-apoptotic Mcl-1

Oncotarget, 2015-01, Vol.6 (1), p.409-426

2015

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Contribution to tumor escape and chemotherapy response: A choice between senescence and apoptosis in heterogeneous tumors

Bulletin du cancer, 2016-01, Vol.103 (1), p.73

2016

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Échappement tumoral lors de la chimiothérapie : un choix entre sénescence et apoptose dans des tumeurs hétérogènes

Bulletin du cancer, 2016-01, Vol.103 (1), p.73-86

2016

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STAT3 as a new autophagy regulator

JAK-STAT, 2013-07, Vol.2 (3), p.e24353-e24353

2013

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Identification of Potent, Selective, and Orally Bioavailable Small-Molecule GSPT1/2 Degraders from a Focused Library of Cereblon Modulators

Journal of medicinal chemistry, 2021-06, Vol.64 (11), p.7296-7311

2021

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Distinct Merkel Cell Polyomavirus Molecular Features in Tumour and Non Tumour Specimens from Patients with Merkel Cell Carcinoma: e1001076

PLoS pathogens, 2010-08, Vol.6 (8)

2010

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Combination of Ribociclib with BET-Bromodomain and PI3K/mTOR Inhibitors for Medulloblastoma Treatment In Vitro and In Vivo

Molecular cancer therapeutics, 2023-01, Vol.22 (1), p.37-51

2023

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INSP-09. Using genetically engineered mouse models and patient-derived orthotopic xenografts to develop new therapies for pediatric brain tumors

Neuro-oncology (Charlottesville, Va.), 2022-06, Vol.24 (Supplement_1), p.i188-i188

2022

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Combination of Ribociclib and Gemcitabine for the Treatment of Medulloblastoma

Molecular cancer therapeutics, 2022-08, Vol.21 (8), p.1306-1317

2022

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From PROTAC to inhibitor: Structure-guided discovery of potent and orally bioavailable BET inhibitors

European journal of medicinal chemistry, 2023-05, Vol.251 (C), p.115246-115246, Article 115246

2023

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Bromodomain-Selective BET Inhibitors Are Potent Antitumor Agents against MYC-Driven Pediatric Cancer

Cancer research (Chicago, Ill.), 2020-09, Vol.80 (17), p.3507-3518

2020

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PDTM-02. TARGETING THE RB PATHWAY IN MEDULLOBLASTOMA

Neuro-oncology (Charlottesville, Va.), 2019-11, Vol.21 (Supplement_6), p.vi187-vi187

2019

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MDB-23. ELP1 GERMLINE DEFICIENCY SENSITIZES THE GRANULE NEURON LINEAGE TO SHH MEDULLOBLASTOMA AND EXPOSES NOVEL THERAPEUTIC VULNERABILITIES

Neuro-oncology (Charlottesville, Va.), 2023-06, Vol.25 (Supplement_1), p.i67-i67

2023

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TARGETING THE RB PATHWAY IN MEDULLOBLASTOMA

Neuro-oncology (Charlottesville, Va.), 2019, Vol.21 (6)

2019

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MEDB-42. GermlineElp1 deficiency promotes genomic instability and survival of granule neuron progenitors primed for SHH medulloblastoma pathogenesis

Neuro-oncology (Charlottesville, Va.), 2022-06, Vol.24 (Suppl 1), p.i115-i115

2022

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MEDB-42. Germline Elp1 deficiency promotes genomic instability and survival of granule neuron progenitors primed for SHH medulloblastoma pathogenesis

Neuro-oncology (Charlottesville, Va.), 2022-06, Vol.24 (Supplement_1), p.i115-i115

2022

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Abstract PR005: Targeting EP300 and CBP for therapeutic benefit in pediatric solid tumors

Cancer research (Chicago, Ill.), 2022-12, Vol.82 (23_Supplement_2), p.PR005-PR005

2022

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Phenyl‐Glutarimides: Alternative Cereblon Binders for the Design of PROTACs

Angewandte Chemie, 2021-12, Vol.133 (51), p.26867-26874

2021

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Distinct merkel cell polyomavirus molecular features in tumour and non tumour specimens from patients with merkel cell carcinoma

Regulation of senescence escape by the cdk4-EZH2-AP2M1 pathway in response to chemotherapy

Phenyl‐Glutarimides: Alternative Cereblon Binders for the Design of PROTACs

Patient-derived models recapitulate heterogeneity of molecular signatures and drug response in pediatric high-grade glioma

Akt inhibition improves irinotecan treatment and prevents cell emergence by switching the senescence response to apoptosis

Irinotecan treatment and senescence failure promote the emergence of more transformed and invasive cells that depend on anti-apoptotic Mcl-1

Contribution to tumor escape and chemotherapy response: A choice between senescence and apoptosis in heterogeneous tumors

Échappement tumoral lors de la chimiothérapie : un choix entre sénescence et apoptose dans des tumeurs hétérogènes

STAT3 as a new autophagy regulator

Identification of Potent, Selective, and Orally Bioavailable Small-Molecule GSPT1/2 Degraders from a Focused Library of Cereblon Modulators

Distinct Merkel Cell Polyomavirus Molecular Features in Tumour and Non Tumour Specimens from Patients with Merkel Cell Carcinoma: e1001076

Combination of Ribociclib with BET-Bromodomain and PI3K/mTOR Inhibitors for Medulloblastoma Treatment In Vitro and In Vivo

INSP-09. Using genetically engineered mouse models and patient-derived orthotopic xenografts to develop new therapies for pediatric brain tumors

Combination of Ribociclib and Gemcitabine for the Treatment of Medulloblastoma

From PROTAC to inhibitor: Structure-guided discovery of potent and orally bioavailable BET inhibitors

Bromodomain-Selective BET Inhibitors Are Potent Antitumor Agents against MYC-Driven Pediatric Cancer

PDTM-02. TARGETING THE RB PATHWAY IN MEDULLOBLASTOMA

MDB-23. ELP1 GERMLINE DEFICIENCY SENSITIZES THE GRANULE NEURON LINEAGE TO SHH MEDULLOBLASTOMA AND EXPOSES NOVEL THERAPEUTIC VULNERABILITIES

TARGETING THE RB PATHWAY IN MEDULLOBLASTOMA

MEDB-42. GermlineElp1 deficiency promotes genomic instability and survival of granule neuron progenitors primed for SHH medulloblastoma pathogenesis

MEDB-42. Germline Elp1 deficiency promotes genomic instability and survival of granule neuron progenitors primed for SHH medulloblastoma pathogenesis

Abstract PR005: Targeting EP300 and CBP for therapeutic benefit in pediatric solid tumors

Phenyl‐Glutarimides: Alternative Cereblon Binders for the Design of PROTACs

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