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Bone marrow stromal cells use TGF-β to suppress allergic responses in a mouse model of ragweed-induced asthma

Proceedings of the National Academy of Sciences - PNAS, 2010-03, Vol.107 (12), p.5652-5657

2010

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TLR3-specific double-stranded RNA oligonucleotide adjuvants induce dendritic cell cross-presentation, CTL responses, and antiviral protection

The Journal of immunology (1950), 2011-02, Vol.186 (4), p.2422-2429

2011

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Discovery of Novel Human Histamine H4 Receptor Ligands by Large-Scale Structure-Based Virtual Screening

Journal of medicinal chemistry, 2008-06, Vol.51 (11), p.3145-3153

2008

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Histamine modulates multiple functional activities of monocyte-derived dendritic cell subsets via histamine receptor 2

International immunology, 2012-02, Vol.24 (2), p.107-116

2012

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Bone marrow stromal cells attenuate sepsis via prostaglandin E 2 -dependent reprogramming of host macrophages to increase their interleukin-10 production

Nature medicine

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Increased antigen presentation and Th1 polarization in genetically histamine-free mice

International immunology, 2007-01, Vol.19 (1), p.51

2007

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Extramedullary hematopoiesis is dysregulated in histamine-free histidine decarboxylase knockout (HDC⁻/⁻) mice

Inflammation research, 2010-06, Vol.59 (6), p.429-436

2010

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CCL17-expressing dendritic cells drive atherosclerosis by restraining regulatory T cell homeostasis in mice

The Journal of clinical investigation, 2011-07, Vol.121 (7), p.2898-2910

2011

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New directions in biomarker research, drug development and personalized medicine

Magyar onkologia, 2013-03, Vol.57 (1), p.5

2013

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Carbohydrate recognition systems in autoimmunity

Autoimmunity (Chur, Switzerland), 2006-12, Vol.39 (8), p.691-704

2006

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Toll-Like Receptor 3-Specific dsRNA Oligonucleotide Adjuvants Induce Dendritic Cell Cross-presentation, CTL Responses and Antiviral Protection

The Journal of immunology (1950), 2011-02, Vol.186 (4), p.2422-2429

2011

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Bone marrow stromal cells use TGF-[beta] to suppress allergic responses in a mouse model of ragweed-induced asthma

Proceedings of the National Academy of Sciences - PNAS, 2010-03, Vol.107 (12), p.5652

2010

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Bone marrow stromal cells use TGF-Î² to suppress allergic responses in a mouse model of ragweed-induced asthma

Proceedings of the National Academy of Sciences - PNAS, 2010, Vol.107 (12), p.5652-5657

2010

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CCL17-expressing dendritic cells drive atherosclerosis by restraining regulatory T cell homeostasis in mice

The Journal of clinical investigation, 2011-07, Vol.121 (7), p.2898

2011

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Increased antigen presentation and T(h)1 polarization in genetically histamine-free mice

International immunology, 2007-01, Vol.19 (1), p.51-58

2007

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Corrigendum: Bone marrow stromal cells attenuate sepsis via prostaglandin E 2 -dependent reprogramming of host macrophages to increase their interleukin-10 production

Nature medicine, 2009-04, Vol.15 (4), p.462-462

2009

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Corrigendum: Bone marrow stromal cells attenuate sepsis via prostaglandin E2-dependent reprogramming of host macrophages to increase their interleukin-10 production

Nature medicine, 2009-04, Vol.15 (4), p.462

2009

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Erratum: Corrigendum: Bone marrow stromal cells attenuate sepsis via prostaglandin E2–dependent reprogramming of host macrophages to increase their interleukin-10 production

Nature medicine, 2009-04, Vol.15 (4), p.462-462

2009

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Bone marrow stromal cells attenuate sepsis via prostaglandin E sub(2)- dependent reprogramming of host macrophages to increase their interleukin-10 production

Nature medicine, 2009-01, Vol.15 (1), p.42-49

2009

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Bone marrow stromal cells attenuate sepsis via prostaglandin [E.sub.2]-dependent reprogramming of host macrophages to increase their interleukin-10 production

Nature medicine, 2009-01, Vol.15 (1), p.42

2009

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Bone marrow stromal cells attenuate sepsis via prostaglandin E2–dependent reprogramming of host macrophages to increase their interleukin-10 production

Nature medicine, 2009, Vol.15 (1), p.42-49

2009

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Correction: Corrigendum: Bone marrow stromal cells attenuate sepsis via prostaglandin E2–dependent reprogramming of host macrophages to increase their interleukin-10 production

Nature medicine, 2009, Vol.15 (4), p.462-462

2009

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Increased antigen presentation and T sub(h)1 polarization in genetically histamine-free mice

International immunology, 2007-01, Vol.19 (1), p.51-58

2007

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Reply to 'Mesenchymal stem cells: another anti-inflammatory treatment for sepsis?'

Nature medicine, 2009-06, Vol.15 (6), p.602-602

2009

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A hisztamin és hisztamin H4 receptor hiányának hatása a dendritikus sejtek mu ködésére

Jelinek, Ivett

2007

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Bone marrow stromal cells use TGF-β to suppress allergic responses in a mouse model of ragweed-induced asthma

TLR3-specific double-stranded RNA oligonucleotide adjuvants induce dendritic cell cross-presentation, CTL responses, and antiviral protection

Discovery of Novel Human Histamine H4 Receptor Ligands by Large-Scale Structure-Based Virtual Screening

Histamine modulates multiple functional activities of monocyte-derived dendritic cell subsets via histamine receptor 2

Bone marrow stromal cells attenuate sepsis via prostaglandin E 2 -dependent reprogramming of host macrophages to increase their interleukin-10 production

Increased antigen presentation and Th1 polarization in genetically histamine-free mice

Extramedullary hematopoiesis is dysregulated in histamine-free histidine decarboxylase knockout (HDC⁻/⁻) mice

CCL17-expressing dendritic cells drive atherosclerosis by restraining regulatory T cell homeostasis in mice

New directions in biomarker research, drug development and personalized medicine

Carbohydrate recognition systems in autoimmunity

Toll-Like Receptor 3-Specific dsRNA Oligonucleotide Adjuvants Induce Dendritic Cell Cross-presentation, CTL Responses and Antiviral Protection

Bone marrow stromal cells use TGF-[beta] to suppress allergic responses in a mouse model of ragweed-induced asthma

Bone marrow stromal cells use TGF-Î² to suppress allergic responses in a mouse model of ragweed-induced asthma

CCL17-expressing dendritic cells drive atherosclerosis by restraining regulatory T cell homeostasis in mice

Increased antigen presentation and T(h)1 polarization in genetically histamine-free mice

Corrigendum: Bone marrow stromal cells attenuate sepsis via prostaglandin E 2 -dependent reprogramming of host macrophages to increase their interleukin-10 production

Corrigendum: Bone marrow stromal cells attenuate sepsis via prostaglandin E2-dependent reprogramming of host macrophages to increase their interleukin-10 production

Erratum: Corrigendum: Bone marrow stromal cells attenuate sepsis via prostaglandin E2–dependent reprogramming of host macrophages to increase their interleukin-10 production

Bone marrow stromal cells attenuate sepsis via prostaglandin E sub(2)- dependent reprogramming of host macrophages to increase their interleukin-10 production

Bone marrow stromal cells attenuate sepsis via prostaglandin [E.sub.2]-dependent reprogramming of host macrophages to increase their interleukin-10 production

Bone marrow stromal cells attenuate sepsis via prostaglandin E2–dependent reprogramming of host macrophages to increase their interleukin-10 production

Correction: Corrigendum: Bone marrow stromal cells attenuate sepsis via prostaglandin E2–dependent reprogramming of host macrophages to increase their interleukin-10 production

Increased antigen presentation and T sub(h)1 polarization in genetically histamine-free mice

Reply to 'Mesenchymal stem cells: another anti-inflammatory treatment for sepsis?'

A hisztamin és hisztamin H4 receptor hiányának hatása a dendritikus sejtek mu ködésére

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