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HPV upregulates MARCHF8 ubiquitin ligase and inhibits apoptosis by degrading the death receptors in head and neck cancer

PLoS pathogens, 2023-03, Vol.19 (3), p.e1011171-e1011171

2023

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Viral Interactions with PDZ Domain-Containing Proteins-An Oncogenic Trait?

Pathogens (Basel), 2016-01, Vol.5 (1), p.8

2016

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Werner Helicase Control of Human Papillomavirus 16 E1-E2 DNA Replication Is Regulated by SIRT1 Deacetylation

mBio, 2019-03, Vol.10 (2)

2019

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The Role of Protein Kinase A Regulation of the E6 PDZ-Binding Domain during the Differentiation-Dependent Life Cycle of Human Papillomavirus Type 18

Journal of Virology, 2013-09, Vol.87 (17), p.9463-9472

2013

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The Relationship between Estrogen-Related Signaling and Human Papillomavirus Positive Cancers

Pathogens (Basel), 2020-05, Vol.9 (5), p.403

2020

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Estrogen Attenuates the Growth of Human Papillomavirus-Positive Epithelial Cells

mSphere, 2020-03, Vol.5 (2)

2020

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Restoring the DREAM Complex Inhibits the Proliferation of High-Risk HPV Positive Human Cells

Cancers, 2021-01, Vol.13 (3), p.489

2021

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A Critical Role for p53 during the HPV16 Life Cycle

Microbiology spectrum, 2022-06, Vol.10 (3), p.e0068122-e0068122

2022

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Human Papillomavirus 16 E6 and E7 Synergistically Repress Innate Immune Gene Transcription

mSphere, 2020-01, Vol.5 (1)

2020

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Correction for Morgan et al., "Werner Helicase Control of Human Papillomavirus 16 E1-E2 DNA Replication Is Regulated by SIRT1 Deacetylation"

mBio, 2019-08, Vol.10 (4)

2019

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Activating the DNA Damage Response and Suppressing Innate Immunity: Human Papillomaviruses Walk the Line

Pathogens (Basel), 2020-06, Vol.9 (6), p.467

2020

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SAMHD1 Regulates Human Papillomavirus 16-Induced Cell Proliferation and Viral Replication during Differentiation of Keratinocytes

mSphere, 2019-08, Vol.4 (4)

2019

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Werner Syndrome Protein (WRN) Regulates Cell Proliferation and the Human Papillomavirus 16 Life Cycle during Epithelial Differentiation

mSphere, 2020-09, Vol.5 (5)

2020

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Human Papillomavirus 16 (HPV16) E2 Repression of TWIST1 Transcription Is a Potential Mediator of HPV16 Cancer Outcomes

mSphere, 2020-12, Vol.5 (6)

2020

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The membrane-associated ubiquitin ligase MARCHF8 stabilizes the human papillomavirus oncoprotein E7 by degrading CUL1 and UBE2L3 in head and neck cancer

Journal of virology, 2024-01, Vol.98 (2), p.e0172623

2024

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Interaction with TopBP1 Is Required for Human Papillomavirus 16 E2 Plasmid Segregation/Retention Function during Mitosis

Journal of virology, 2022-07, Vol.96 (16), p.e0083022-e0083022

2022

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Human Papillomavirus 16 E2 Interaction with TopBP1 Is Required for E2 and Viral Genome Stability during the Viral Life Cycle

Journal of virology, 2023-03, Vol.97 (3), p.e0006323-e0006323

2023

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Human papillomavirus 16 E2 blocks cellular senescence in response to activation of the DNA damage response

Virology (New York, N.Y.), 2022-10, Vol.575, p.54-62

2022

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Direct interaction with the BRD4 carboxyl-terminal motif (CTM) and TopBP1 is required for human papillomavirus 16 E2 association with mitotic chromatin and plasmid segregation function

Journal of virology, 2023-10, Vol.97 (10), p.e0078223-e0078223

2023

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Human Papillomavirus 16 E2 Regulates Keratinocyte Gene Expression Relevant to Cancer and the Viral Life Cycle

Journal of virology, 2019-02, Vol.93 (4)

2019

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A human papillomavirus 16 E2-TopBP1 dependent SIRT1-p300 acetylation switch regulates mitotic viral and human protein levels and activates the DNA damage response

mBio, 2024-05, Vol.15 (6), p.e0067624-e0067624

2024

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An oral keratinocyte life cycle model identifies novel host genome regulation by human papillomavirus 16 relevant to HPV positive head and neck cancer

Oncotarget, 2017-10, Vol.8 (47), p.81892-81909

2017

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The human papillomavirus type 16 L1 protein directly interacts with E2 and enhances E2-dependent replication and transcription activation

Journal of general virology, 2015-08, Vol.96 (8), p.2274-2285

2015

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Correction for Evans et al., "Human Papillomavirus 16 E2 Regulates Keratinocyte Gene Expression Relevant to Cancer and the Viral Life Cycle"

Journal of virology, 2019-09, Vol.93 (18)

2019

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HPV upregulates MARCHF8 ubiquitin ligase and inhibits apoptosis by degrading the death receptors in head and neck cancer

Viral Interactions with PDZ Domain-Containing Proteins-An Oncogenic Trait?

Werner Helicase Control of Human Papillomavirus 16 E1-E2 DNA Replication Is Regulated by SIRT1 Deacetylation

CK2 Phosphorylation of Human Papillomavirus 16 E2 on Serine 23 Promotes Interaction with TopBP1 and Is Critical for E2 Interaction with Mitotic Chromatin and the Viral Life Cycle

The Role of Protein Kinase A Regulation of the E6 PDZ-Binding Domain during the Differentiation-Dependent Life Cycle of Human Papillomavirus Type 18

The Relationship between Estrogen-Related Signaling and Human Papillomavirus Positive Cancers

Estrogen Attenuates the Growth of Human Papillomavirus-Positive Epithelial Cells

Restoring the DREAM Complex Inhibits the Proliferation of High-Risk HPV Positive Human Cells

A Critical Role for p53 during the HPV16 Life Cycle

Human Papillomavirus 16 E6 and E7 Synergistically Repress Innate Immune Gene Transcription

Correction for Morgan et al., "Werner Helicase Control of Human Papillomavirus 16 E1-E2 DNA Replication Is Regulated by SIRT1 Deacetylation"

Activating the DNA Damage Response and Suppressing Innate Immunity: Human Papillomaviruses Walk the Line

SAMHD1 Regulates Human Papillomavirus 16-Induced Cell Proliferation and Viral Replication during Differentiation of Keratinocytes

Werner Syndrome Protein (WRN) Regulates Cell Proliferation and the Human Papillomavirus 16 Life Cycle during Epithelial Differentiation

Human Papillomavirus 16 (HPV16) E2 Repression of TWIST1 Transcription Is a Potential Mediator of HPV16 Cancer Outcomes

The membrane-associated ubiquitin ligase MARCHF8 stabilizes the human papillomavirus oncoprotein E7 by degrading CUL1 and UBE2L3 in head and neck cancer

Interaction with TopBP1 Is Required for Human Papillomavirus 16 E2 Plasmid Segregation/Retention Function during Mitosis

Human Papillomavirus 16 E2 Interaction with TopBP1 Is Required for E2 and Viral Genome Stability during the Viral Life Cycle

Human papillomavirus 16 E2 blocks cellular senescence in response to activation of the DNA damage response

Direct interaction with the BRD4 carboxyl-terminal motif (CTM) and TopBP1 is required for human papillomavirus 16 E2 association with mitotic chromatin and plasmid segregation function

Human Papillomavirus 16 E2 Regulates Keratinocyte Gene Expression Relevant to Cancer and the Viral Life Cycle

A human papillomavirus 16 E2-TopBP1 dependent SIRT1-p300 acetylation switch regulates mitotic viral and human protein levels and activates the DNA damage response

An oral keratinocyte life cycle model identifies novel host genome regulation by human papillomavirus 16 relevant to HPV positive head and neck cancer

The human papillomavirus type 16 L1 protein directly interacts with E2 and enhances E2-dependent replication and transcription activation

Correction for Evans et al., "Human Papillomavirus 16 E2 Regulates Keratinocyte Gene Expression Relevant to Cancer and the Viral Life Cycle"

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