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Rictor Undergoes Glycogen Synthase Kinase 3 (GSK3)-dependent, FBXW7-mediated Ubiquitination and Proteasomal Degradation

The Journal of biological chemistry, 2015-05, Vol.290 (22), p.14120-14129

2015

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Maintaining Glycogen Synthase Kinase-3 Activity Is Critical for mTOR Kinase Inhibitors to Inhibit Cancer Cell Growth

Cancer research (Chicago, Ill.), 2014-05, Vol.74 (9), p.2555-2568

2014

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Elevated expression of eukaryotic translation initiation factor 4E is associated with proliferation, invasion and acquired resistance to erlotinib in lung cancer

Cancer biology & therapy, 2012-03, Vol.13 (5), p.272-280

2012

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GSK3 is required for rapalogs to induce degradation of some oncogenic proteins and to suppress cancer cell growth

Oncotarget, 2015-04, Vol.6 (11), p.8974-8987

2015

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mTOR Complex 2 Stabilizes Mcl-1 Protein by Suppressing Its Glycogen Synthase Kinase 3-Dependent and SCF-FBXW7-Mediated Degradation

Molecular and cellular biology, 2015-07, Vol.35 (13), p.2344-2355

2015

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Live-Cell Invasive Phenotyping Uncovers ALK2 as a Therapeutic Target in LKB1-Mutant Lung Cancer

Cancer research (Chicago, Ill.), 2024-08

2024

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Correction: GSK3 is required for rapalogs to induce degradation of some oncogenic proteins and to suppress cancer cell growth

Oncotarget, 2021-02, Vol.12 (3), p.251-252

2021

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Abstract 865: Isolation of circulating tumors cells from genetically engineered mouse models of lung adenocarcinoma

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.865-865

2019

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The E3 ubiquitin ligases β-TrCP and FBXW7 cooperatively mediates GSK3-dependent Mcl-1 degradation induced by the Akt inhibitor API-1, resulting in apoptosis

Molecular cancer, 2013-11, Vol.12 (1), p.146-146, Article 146

2013

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Abstract A027: Defining the role of PER1 and circadian rhythm dysregulation in KRAS/LKB1-mutant lung adenocarcinoma

Molecular cancer research, 2023-05, Vol.21 (5_Supplement), p.A027-A027

2023

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Abstract 349: mTOR complex 2 (mTORC2) positively regulates Mcl-1 stability by suppressing GSK3-dependent and FBXW7-mediated proteasomal degradation of Mcl-1

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (19_Supplement), p.349-349

2014

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Abstract 1036: Targeting bone morphogenetic protein 6 as a therapeutic target for LKB1-inactivated lung cancer

Cancer research (Chicago, Ill.), 2022-06, Vol.82 (12_Supplement), p.1036-1036

2022

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Abstract 4047: Unraveling synergistic therapeutic strategies involving FAK inhibition and immunotherapy using anti-PD1 antibody in KRAS and LKB1-driven lung cancer

Cancer research (Chicago, Ill.), 2024-03, Vol.84 (6_Supplement), p.4047-4047

2024

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Erratum: GSK3 is required for rapalogs to induce degradation of some oncogenic proteins and to suppress cancer cell growth

Oncotarget, 2021-02, Vol.12 (3), p.251-252

2021

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GSK3 is required for rapalogs to induce degradation of some oncogenic proteins and to suppress cancer cell growth

Oncotarget, 2021-02, Vol.12 (3), p.251-252

2021

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Abstract B45: An early treatment window to target FAK-dependent collective invasion in Lkb1-mutant lung adenocarcinoma

Cancer research (Chicago, Ill.), 2016-04, Vol.76 (7_Supplement), p.B45-B45

2016

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Abstract LB-348: Developing a personalized anti-metastatic therapy to treat KRAS, LKB1-mutant lung adenocarcinoma

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.LB-348-LB-348

2016

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Abstract LB-41: Rictor undergoes GSK3-dependent, FBXW7-mediated ubiquitination and proteasomal degradation

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.LB-41-LB-41

2013

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Abstract 2803: GSK3 plays a crucial role in determining cell response to mTOR inhibitors

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.2803-2803

2012

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The E3 ubiquitin ligases [beta]-TrCP and FBXW7 cooperatively mediates GSK3-dependent Mcl-1 degradation induced by the Akt inhibitor API-1, resulting in apoptosis

Molecular cancer, 2013-11, Vol.12

2013

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Targeting adhesion signaling in KRAS, LKB1 mutant lung adenocarcinoma

JCI insight, 2017-03, Vol.2 (5), p.e90487-e90487

2017

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Abstract 1953: Elevated eukaryotic translation initiation factor 4E (eIF4E) is involved in erlotinib resistance

Cancer research (Chicago, Ill.), 2011-04, Vol.71 (8_Supplement), p.1953-1953

2011

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Rictor Undergoes Glycogen Synthase Kinase 3 (GSK3)-dependent, FBXW7-mediated Ubiquitination and Proteasomal Degradation

Maintaining Glycogen Synthase Kinase-3 Activity Is Critical for mTOR Kinase Inhibitors to Inhibit Cancer Cell Growth

Elevated expression of eukaryotic translation initiation factor 4E is associated with proliferation, invasion and acquired resistance to erlotinib in lung cancer

GSK3 is required for rapalogs to induce degradation of some oncogenic proteins and to suppress cancer cell growth

mTOR Complex 2 Stabilizes Mcl-1 Protein by Suppressing Its Glycogen Synthase Kinase 3-Dependent and SCF-FBXW7-Mediated Degradation

Live-Cell Invasive Phenotyping Uncovers ALK2 as a Therapeutic Target in LKB1-Mutant Lung Cancer

Correction: GSK3 is required for rapalogs to induce degradation of some oncogenic proteins and to suppress cancer cell growth

Abstract 865: Isolation of circulating tumors cells from genetically engineered mouse models of lung adenocarcinoma

The E3 ubiquitin ligases β-TrCP and FBXW7 cooperatively mediates GSK3-dependent Mcl-1 degradation induced by the Akt inhibitor API-1, resulting in apoptosis

Abstract A027: Defining the role of PER1 and circadian rhythm dysregulation in KRAS/LKB1-mutant lung adenocarcinoma

Abstract 349: mTOR complex 2 (mTORC2) positively regulates Mcl-1 stability by suppressing GSK3-dependent and FBXW7-mediated proteasomal degradation of Mcl-1

Abstract 1036: Targeting bone morphogenetic protein 6 as a therapeutic target for LKB1-inactivated lung cancer

Abstract 4047: Unraveling synergistic therapeutic strategies involving FAK inhibition and immunotherapy using anti-PD1 antibody in KRAS and LKB1-driven lung cancer

Erratum: GSK3 is required for rapalogs to induce degradation of some oncogenic proteins and to suppress cancer cell growth

GSK3 is required for rapalogs to induce degradation of some oncogenic proteins and to suppress cancer cell growth

Abstract B45: An early treatment window to target FAK-dependent collective invasion in Lkb1-mutant lung adenocarcinoma

Abstract LB-348: Developing a personalized anti-metastatic therapy to treat KRAS, LKB1-mutant lung adenocarcinoma

Abstract LB-41: Rictor undergoes GSK3-dependent, FBXW7-mediated ubiquitination and proteasomal degradation

Abstract 2803: GSK3 plays a crucial role in determining cell response to mTOR inhibitors

The E3 ubiquitin ligases [beta]-TrCP and FBXW7 cooperatively mediates GSK3-dependent Mcl-1 degradation induced by the Akt inhibitor API-1, resulting in apoptosis

Targeting adhesion signaling in KRAS, LKB1 mutant lung adenocarcinoma

Abstract 1953: Elevated eukaryotic translation initiation factor 4E (eIF4E) is involved in erlotinib resistance

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