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Notch is an essential upstream regulator of NF-κB and is relevant for survival of Hodgkin and Reed-Sternberg cells

Leukemia, 2012-04, Vol.26 (4), p.806-813

2012

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Quark–gluon plasma and color glass condensate at RHIC? The perspective from the BRAHMS experiment

Nuclear physics. A, 2005-08, Vol.757 (1), p.1-27

2005

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Notch and NF-κB signaling pathways in the biology of classical Hodgkin lymphoma

Current molecular medicine, 2011-04, Vol.11 (3), p.236

2011

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High-level expression of Mastermind-like 2 contributes to aberrant activation of the NOTCH signaling pathway in human lymphomas

Oncogene, 2011-04, Vol.30 (15), p.1831-1840

2011

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Charged meson rapidity distributions in central Au + Au collisions at √sNN = 200 GeV

Physical review letters, 2005-04, Vol.94 (16), p.162301.1-162301.4

2005

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Notch pathway inhibition controls myeloma bone disease in the murine MOPC315.BM model

Blood cancer journal (New York), 2014-06, Vol.4 (6), p.e217-e217

2014

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PAX5 overexpression is not enough to reestablish the mature B-cell phenotype in classical Hodgkin lymphoma

Leukemia, 2014-01, Vol.28 (1), p.213-216

2014

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Aberrant expression of Notch1 interferes with the B-lymphoid phenotype of neoplastic B cells in classical Hodgkin lymphoma

Leukemia, 2008-08, Vol.22 (8), p.1587-1594

2008

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Notch inhibition blocks multiple myeloma cell-induced osteoclast activation

Leukemia, 2008-12, Vol.22 (12), p.2273-2277

2008

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Activated Notch1 signaling promotes tumor cell proliferation and survival in Hodgkin and anaplastic large cell lymphoma

Blood, 2002-05, Vol.99 (9), p.3398-3403

2002

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A novel mouse model for multiple myeloma (MOPC315.BM) that allows noninvasive spatiotemporal detection of osteolytic disease

PloS one, 2012-12, Vol.7 (12), p.e51892

2012

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Intrinsic inhibition of transcription factor E2A by HLH proteins ABF-1 and Id2 mediates reprogramming of neoplastic B cells in Hodgkin lymphoma

Nature Immunology, 2006-02, Vol.7 (2), p.207-215

2006

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Scanning the phases of QCD with BRAHMS

Journal of Physics G: Nuclear and Particle Physics, 2004-08, Vol.30 (8), p.S667-674

2004

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Jagged1-induced Notch signaling drives proliferation of multiple myeloma cells

Blood, 2004-05, Vol.103 (9), p.3511-3515

2004

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Aberrantly expressed c-Jun and JunB are a hallmark of Hodgkin lymphoma cells, stimulate proliferation and synergize with NF-κB

The EMBO journal, 2002-08, Vol.21 (15), p.4104-4113

2002

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PF581 FUNCTIONAL HIGH‐THROUGHPUT SCREENING FOR IDENTIFICATION OF NOTCH1 DOWNSTREAM EFFECTORS AS NOVEL THERAPEUTIC TARGETS IN MULTIPLE MYELOMA

HemaSphere, 2019-06, Vol.3 (S1), p.242-n/a

2019

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c-FLIP mediates resistance of Hodgkin/Reed-Sternberg cells to death receptor-induced apoptosis

The Journal of experimental medicine, 2004-04, Vol.199 (8), p.1041-1052

2004

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A rapamycin derivative (everolimus) controls proliferation through down-regulation of truncated CCAAT enhancer binding protein β and NF-κB activity in Hodgkin and anaplastic large cell lymphomas

Blood, 2005-09, Vol.106 (5), p.1801-1807

2005

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Inhibition of NF-κB essentially contributes to arsenic-induced apoptosis

Blood, 2003-08, Vol.102 (3), p.1028-1034

2003

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Elevated NF-κB p50 complex formation and Bcl-3 expression in classical Hodgkin, anaplastic large-cell, and other peripheral T-cell lymphomas

Blood, 2005-12, Vol.106 (13), p.4287-4293

2005

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Notch is an essential upstream regulator of NF-[kappa]B and is relevant for survival of Hodgkin and Reed-Sternberg cells.(ORIGINAL ARTICLE)

Leukemia, 2012-04, Vol.26 (4), p.806

2012

Volltextzugriff (PDF)

Trimethoprim-sulfamethoxazole exacerbates posthypoxic action myoclonus in a patient with suspicion of Pneumocystis jiroveci infection

Infection, 2004-06, Vol.32 (3), p.176-178

2004

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Sp1 as G1 cell cycle phase specific transcription factor in epithelial cells

Oncogene, 2002-02, Vol.21 (10), p.1485-1492

2002

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Particle production and flow at SIS energies

Nuclear physics. A, 1996-12, Vol.610, p.49-62

Herrmann, N.

1996

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Notch is an essential upstream regulator of NF-κB and is relevant for survival of Hodgkin and Reed-Sternberg cells

Quark–gluon plasma and color glass condensate at RHIC? The perspective from the BRAHMS experiment

Notch and NF-κB signaling pathways in the biology of classical Hodgkin lymphoma

High-level expression of Mastermind-like 2 contributes to aberrant activation of the NOTCH signaling pathway in human lymphomas

Charged meson rapidity distributions in central Au + Au collisions at √sNN = 200 GeV

Notch pathway inhibition controls myeloma bone disease in the murine MOPC315.BM model

PAX5 overexpression is not enough to reestablish the mature B-cell phenotype in classical Hodgkin lymphoma

Aberrant expression of Notch1 interferes with the B-lymphoid phenotype of neoplastic B cells in classical Hodgkin lymphoma

Notch inhibition blocks multiple myeloma cell-induced osteoclast activation

Activated Notch1 signaling promotes tumor cell proliferation and survival in Hodgkin and anaplastic large cell lymphoma

A novel mouse model for multiple myeloma (MOPC315.BM) that allows noninvasive spatiotemporal detection of osteolytic disease

Forward and midrapidity like-particle ratios from p+p collisions at s=200 GeV

Intrinsic inhibition of transcription factor E2A by HLH proteins ABF-1 and Id2 mediates reprogramming of neoplastic B cells in Hodgkin lymphoma

Scanning the phases of QCD with BRAHMS

Jagged1-induced Notch signaling drives proliferation of multiple myeloma cells

Aberrantly expressed c-Jun and JunB are a hallmark of Hodgkin lymphoma cells, stimulate proliferation and synergize with NF-κB

PF581 FUNCTIONAL HIGH‐THROUGHPUT SCREENING FOR IDENTIFICATION OF NOTCH1 DOWNSTREAM EFFECTORS AS NOVEL THERAPEUTIC TARGETS IN MULTIPLE MYELOMA

c-FLIP mediates resistance of Hodgkin/Reed-Sternberg cells to death receptor-induced apoptosis

A rapamycin derivative (everolimus) controls proliferation through down-regulation of truncated CCAAT enhancer binding protein β and NF-κB activity in Hodgkin and anaplastic large cell lymphomas

Inhibition of NF-κB essentially contributes to arsenic-induced apoptosis

Elevated NF-κB p50 complex formation and Bcl-3 expression in classical Hodgkin, anaplastic large-cell, and other peripheral T-cell lymphomas

Notch is an essential upstream regulator of NF-[kappa]B and is relevant for survival of Hodgkin and Reed-Sternberg cells.(ORIGINAL ARTICLE)

Trimethoprim-sulfamethoxazole exacerbates posthypoxic action myoclonus in a patient with suspicion of Pneumocystis jiroveci infection

Sp1 as G1 cell cycle phase specific transcription factor in epithelial cells

Particle production and flow at SIS energies

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