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A causal link between oxidative stress and inflammation in cardiovascular and renal complications of diabetes

Clinical science (1979), 2018-08, Vol.132 (16), p.1811-1836

2018

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miR-21 promotes renal fibrosis in diabetic nephropathy by targeting PTEN and SMAD7

Clinical science (1979), 2015-12, Vol.129 (12), p.1237-1249

2015

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Dyslipidemia, Diabetes and Atherosclerosis: Role of Inflammation and ROS-Redox-Sensitive Factors

Biomedicines, 2021-11, Vol.9 (11), p.1602

2021

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Oxidative Stress and Inflammation in Renal and Cardiovascular Complications of Diabetes

Biology (Basel, Switzerland), 2020-12, Vol.10 (1), p.18

2020

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Genetic Targeting or Pharmacologic Inhibition of NADPH Oxidase Nox4 Provides Renoprotection in Long-Term Diabetic Nephropathy

Journal of the American Society of Nephrology, 2014-06, Vol.25 (6), p.1237-1254

2014

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The emerging role of NADPH oxidase NOX5 in vascular disease

Clinical science (1979), 2017-05, Vol.131 (10), p.981-990

2017

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Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b

Kidney international, 2014, Vol.85 (2), p.352-361

2014

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Are reactive oxygen species still the basis for diabetic complications?

Clinical science (1979), 2015-07, Vol.129 (2), p.199-216

2015

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Adverse renal effects of NLRP3 inflammasome inhibition by MCC950 in an interventional model of diabetic kidney disease

Clinical science (1979), 2022-01, Vol.136 (2), p.167-180

2022

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Podocyte-specific Nox4 deletion affords renoprotection in a mouse model of diabetic nephropathy

Diabetologia, 2016-02, Vol.59 (2), p.379-389

2016

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Independent of Renox, NOX5 Promotes Renal Inflammation and Fibrosis in Diabetes by Activating ROS-Sensitive Pathways

Diabetes (New York, N.Y.), 2022-06, Vol.71 (6), p.1282-1298

2022

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Diabetes and Renal Complications: An Overview on Pathophysiology, Biomarkers and Therapeutic Interventions

Biomedicines, 2024-05, Vol.12 (5), p.1098

2024

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Empagliflozin modulates renal sympathetic and heart rate baroreflexes in a rabbit model of diabetes

Diabetologia, 2020-07, Vol.63 (7), p.1424-1434

2020

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NADPH Oxidase Inhibition: Preclinical and Clinical Studies in Diabetic Complications

Antioxidants & redox signaling, 2020-08, Vol.33 (6), p.415-434

2020

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Diabetes and Kidney Disease: Role of Oxidative Stress

Antioxidants & redox signaling, 2016-10, Vol.25 (12), p.657-684

2016

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Oxidative Stress, Nox Isoforms and Complications of Diabetes—Potential Targets for Novel Therapies

Journal of cardiovascular translational research, 2012-08, Vol.5 (4), p.509-518

2012

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Endothelial NOX5 Obliterates the Reno-Protective Effect of Nox4 Deletion by Promoting Renal Fibrosis via Activation of EMT and ROS-Sensitive Pathways in Diabetes

Antioxidants, 2024-04, Vol.13 (4), p.396

2024

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Combined NOX1/4 inhibition with GKT137831 in mice provides dose-dependent reno- and atheroprotection even in established micro- and macrovascular disease

Diabetologia, 2017-05, Vol.60 (5), p.927-937

2017

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NADPH Oxidase Nox5 Accelerates Renal Injury in Diabetic Nephropathy

Diabetes (New York, N.Y.), 2017-10, Vol.66 (10), p.2691-2703

2017

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Nox (NADPH Oxidase) 1, Nox4, and Nox5 Promote Vascular Permeability and Neovascularization in Retinopathy

Hypertension (Dallas, Tex. 1979), 2020-04, Vol.75 (4), p.1091-1101

2020

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Endothelial or vascular smooth muscle cell-specific expression of human NOX5 exacerbates renal inflammation, fibrosis and albuminuria in the Akita mouse

Diabetologia, 2019-09, Vol.62 (9), p.1712-1726

2019

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437-P: NOX5 in Human Peripheral Blood Mononuclear Cells Is a Potential Biomarker for Unstable Diabetic Vascular Disease

Diabetes (New York, N.Y.), 2022-06, Vol.71 (Supplement_1)

2022

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478-P: Nox5 Aggravates Renal Injury Independent of Nox4 Expression in Diabetes

Diabetes (New York, N.Y.), 2020-06, Vol.69 (Supplement_1)

2020

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Contribution of the Renal Nerves to Hypertension in a Rabbit Model of Chronic Kidney Disease

Hypertension (Dallas, Tex. 1979), 2020-11, Vol.76 (5), p.1470-1479

2020

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479-P: Inhibition of the NLRP3 Inflammasome by MCC950 Accelerates Renal Injury in Diabetes Model

Diabetes (New York, N.Y.), 2020-06, Vol.69 (Supplement_1)

2020

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A causal link between oxidative stress and inflammation in cardiovascular and renal complications of diabetes

miR-21 promotes renal fibrosis in diabetic nephropathy by targeting PTEN and SMAD7

Dyslipidemia, Diabetes and Atherosclerosis: Role of Inflammation and ROS-Redox-Sensitive Factors

Oxidative Stress and Inflammation in Renal and Cardiovascular Complications of Diabetes

Genetic Targeting or Pharmacologic Inhibition of NADPH Oxidase Nox4 Provides Renoprotection in Long-Term Diabetic Nephropathy

The emerging role of NADPH oxidase NOX5 in vascular disease

Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b

Are reactive oxygen species still the basis for diabetic complications?

Adverse renal effects of NLRP3 inflammasome inhibition by MCC950 in an interventional model of diabetic kidney disease

Podocyte-specific Nox4 deletion affords renoprotection in a mouse model of diabetic nephropathy

Independent of Renox, NOX5 Promotes Renal Inflammation and Fibrosis in Diabetes by Activating ROS-Sensitive Pathways

Diabetes and Renal Complications: An Overview on Pathophysiology, Biomarkers and Therapeutic Interventions

Empagliflozin modulates renal sympathetic and heart rate baroreflexes in a rabbit model of diabetes

NADPH Oxidase Inhibition: Preclinical and Clinical Studies in Diabetic Complications

Diabetes and Kidney Disease: Role of Oxidative Stress

Oxidative Stress, Nox Isoforms and Complications of Diabetes—Potential Targets for Novel Therapies

Endothelial NOX5 Obliterates the Reno-Protective Effect of Nox4 Deletion by Promoting Renal Fibrosis via Activation of EMT and ROS-Sensitive Pathways in Diabetes

Combined NOX1/4 inhibition with GKT137831 in mice provides dose-dependent reno- and atheroprotection even in established micro- and macrovascular disease

NADPH Oxidase Nox5 Accelerates Renal Injury in Diabetic Nephropathy

Nox (NADPH Oxidase) 1, Nox4, and Nox5 Promote Vascular Permeability and Neovascularization in Retinopathy

Endothelial or vascular smooth muscle cell-specific expression of human NOX5 exacerbates renal inflammation, fibrosis and albuminuria in the Akita mouse

437-P: NOX5 in Human Peripheral Blood Mononuclear Cells Is a Potential Biomarker for Unstable Diabetic Vascular Disease

478-P: Nox5 Aggravates Renal Injury Independent of Nox4 Expression in Diabetes

Contribution of the Renal Nerves to Hypertension in a Rabbit Model of Chronic Kidney Disease

479-P: Inhibition of the NLRP3 Inflammasome by MCC950 Accelerates Renal Injury in Diabetes Model

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