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Arrhythmogenesis in Timothy Syndrome is associated with defects in Ca2+-dependent inactivation

Nature communications, 2016-01, Vol.7 (1), p.10370-12, Article 10370

2016

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A Precision Medicine Approach to the Rescue of Function on Malignant Calmodulinopathic Long-QT Syndrome

Circulation research, 2017-01, Vol.120 (1), p.39-48

2017

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Calmodulin Mutations in Human Disease

Channels (Austin, Tex.), 2023-12, Vol.17 (1), p.2165278-2165278

2023

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Apocalmodulin Itself Promotes Ion Channel Opening and Ca2+ Regulation

Cell, 2014-10, Vol.159 (3), p.608-622

2014

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Following Optogenetic Dimerizers and Quantitative Prospects

Biophysical journal, 2016-09, Vol.111 (6), p.1132-1140

2016

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The molecular basis of the inhibition of CaV1 calcium-dependent inactivation by the distal carboxy tail

The Journal of biological chemistry, 2021-01, Vol.296, p.100502, Article 100502

2021

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Infanticide vs. inherited cardiac arrhythmias

Europace (London, England), 2021-03, Vol.23 (3), p.441-450

2021

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David T. Yue: In Memoriam

Neuron (Cambridge, Mass.), 2015-03, Vol.85 (6), p.1158-1161

2015

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A bilobal model of Ca2+-dependent inactivation to probe the physiology of L-type Ca2+ channels

The Journal of general physiology, 2018-12, Vol.150 (12), p.1688-1701

2018

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Nanodomain Ca2+ of Ca2+ channels detected by a tethered genetically encoded Ca2+ sensor

Nature communications, 2012-04, Vol.3 (1), p.778-778, Article 778

2012

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Novel fluorescence resonance energy transfer‐based reporter reveals differential calcineurin activation in neonatal and adult cardiomyocytes

The Journal of physiology, 2015-09, Vol.593 (17), p.3865-3884

2015

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Protein kinase A modulation of CaV1.4 calcium channels

Nature communications, 2016-07, Vol.7 (1), p.12239-12239, Article 12239

2016

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Towards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium Channels

Current molecular pharmacology, 2015-01, Vol.8 (2), p.188-205

2015

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Arrhythmogenesis in Timothy Syndrome is associated with defects in Ca(2+)-dependent inactivation

Nature communications, 2016-01, Vol.7, p.10370-10370

2016

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Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca(2+) currents and promote proarrhythmic behavior in ventricular myocytes

Journal of molecular and cellular cardiology, 2014-09, Vol.74, p.115-124

2014

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Apocalmodulin itself promotes ion channel opening and Ca(2+) regulation

Cell, 2014-10, Vol.159 (3), p.608-622

2014

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Induced pluripotent stem cell-derived cardiomyocytes enable the development of new therapeutic strategies for the treatment of LQT8

Biophysical journal, 2023-02, Vol.122 (3), p.310a-310a

2023

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CACNA1C-Related Channelopathies

Handbook of experimental pharmacology, 2023, Vol.279, p.159-181

2023

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Mechanism of local and global Ca2+ sensing by calmodulin in complex with a Ca2+ channel

Cell, 2008-06, Vol.133 (7), p.1228-1240

2008

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Impaired CaV1.2 inactivation reduces the efficacy of calcium channel blockers in the treatment of LQT8

Journal of molecular and cellular cardiology, 2022-12, Vol.173, p.92-100

2022

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Disease-associated calmodulin variants impair regulation of neuronal voltage-gated calcium channels

Biophysical journal, 2023-02, Vol.122 (3), p.105a-105a

2023

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CaV1.2 channelopathic mutations evoke diverse pathophysiological mechanisms

The Journal of general physiology, 2022-11, Vol.154 (11), p.1

2022

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An autism-associated mutation in CaV1.3 channels has opposing effects on voltage- and Ca(2+)-dependent regulation

Scientific reports, 2016-06, Vol.6, p.27235-27235

2016

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An autism-associated mutation in CaV1.3 channels has opposing effects on voltage- and Ca2+-dependent regulation

Scientific reports, 2016-06, Vol.6 (1), Article 27235

2016

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Sodium Channel Blockade May Contribute to the Analgesic Efficacy of Antidepressants

The journal of pain, 2007-04, Vol.8 (4), p.315-324

2007

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Arrhythmogenesis in Timothy Syndrome is associated with defects in Ca2+-dependent inactivation

A Precision Medicine Approach to the Rescue of Function on Malignant Calmodulinopathic Long-QT Syndrome

Calmodulin Mutations in Human Disease

Apocalmodulin Itself Promotes Ion Channel Opening and Ca2+ Regulation

Following Optogenetic Dimerizers and Quantitative Prospects

The molecular basis of the inhibition of CaV1 calcium-dependent inactivation by the distal carboxy tail

Infanticide vs. inherited cardiac arrhythmias

David T. Yue: In Memoriam

A bilobal model of Ca2+-dependent inactivation to probe the physiology of L-type Ca2+ channels

Nanodomain Ca2+ of Ca2+ channels detected by a tethered genetically encoded Ca2+ sensor

Novel fluorescence resonance energy transfer‐based reporter reveals differential calcineurin activation in neonatal and adult cardiomyocytes

Protein kinase A modulation of CaV1.4 calcium channels

Towards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium Channels

Arrhythmogenesis in Timothy Syndrome is associated with defects in Ca(2+)-dependent inactivation

Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca(2+) currents and promote proarrhythmic behavior in ventricular myocytes

Apocalmodulin itself promotes ion channel opening and Ca(2+) regulation

Induced pluripotent stem cell-derived cardiomyocytes enable the development of new therapeutic strategies for the treatment of LQT8

CACNA1C-Related Channelopathies

Mechanism of local and global Ca2+ sensing by calmodulin in complex with a Ca2+ channel

Impaired CaV1.2 inactivation reduces the efficacy of calcium channel blockers in the treatment of LQT8

Disease-associated calmodulin variants impair regulation of neuronal voltage-gated calcium channels

CaV1.2 channelopathic mutations evoke diverse pathophysiological mechanisms

An autism-associated mutation in CaV1.3 channels has opposing effects on voltage- and Ca(2+)-dependent regulation

An autism-associated mutation in CaV1.3 channels has opposing effects on voltage- and Ca2+-dependent regulation

Sodium Channel Blockade May Contribute to the Analgesic Efficacy of Antidepressants

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