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Autoantibody binding and unique enzyme-substrate intermediate conformation of human transglutaminase 3

Nature communications, 2023-10, Vol.14 (1), p.6216-6216, Article 6216

2023

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High-Throughput Single-Cell Analysis of B Cell Receptor Usage among Autoantigen-Specific Plasma Cells in Celiac Disease

The Journal of immunology (1950), 2017-07, Vol.199 (2), p.782-791

2017

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Generation of circulating autoreactive pre-plasma cells fueled by naive B cells in celiac disease

Cell reports (Cambridge), 2024-04, Vol.43 (4), p.114045-114045, Article 114045

2024

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Activity-regulating structural changes and autoantibody epitopes in transglutaminase 2 assessed by hydrogen/deuterium exchange

Proceedings of the National Academy of Sciences - PNAS, 2014-12, Vol.111 (48), p.17146-17151

2014

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Strong Clonal Relatedness between Serum and Gut IgA despite Different Plasma Cell Origins

Cell reports (Cambridge), 2017-09, Vol.20 (10), p.2357-2367

2017

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Enhanced B-Cell Receptor Recognition of the Autoantigen Transglutaminase 2 by Efficient Catalytic Self-Multimerization

PloS one, 2015-08, Vol.10 (8), p.e0134922-e0134922

2015

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Igs as Substrates for Transglutaminase 2: Implications for Autoantibody Production in Celiac Disease

The Journal of immunology (1950), 2015-12, Vol.195 (11), p.5159-5168

2015

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Coeliac disease in the Trøndelag Health Study (HUNT), Norway, a population-based cohort of coeliac disease patients

BMJ open, 2024-01, Vol.14 (1), p.e077131-e077131

2024

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Separate Gut Plasma Cell Populations Produce Auto‐Antibodies against Transglutaminase 2 and Transglutaminase 3 in Dermatitis Herpetiformis

Advanced science, 2023-09, Vol.10 (25), p.e2300401-n/a

2023

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Thiol−Disulfide Exchange between Glutaredoxin and Glutathione

Biochemistry (Easton), 2010-02, Vol.49 (4), p.810-820

2010

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Autoimmunity provoked by foreign antigens: In celiac disease, exogenous gluten drives T cell-B cell interactions that cause autoimmunity

Science (American Association for the Advancement of Science), 2020

2020

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Dissecting autoimmune encephalitis through the lens of intrathecal B cells

Proceedings of the National Academy of Sciences - PNAS, 2024-02, Vol.121 (9), p.e2401337121

2024

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Autoimmunity provoked by foreign antigens

Science (American Association for the Advancement of Science), 2020-04, Vol.368 (6487), p.132-133

2020

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The Immunobiology and Pathogenesis of Celiac Disease

Annual review of pathology, 2023-01, Vol.18, p.47

2023

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Tango of B cells with T cells in the making of secretory antibodies to gut bacteria

Nature reviews. Gastroenterology & hepatology, 2023-02, Vol.20 (2), p.120-128

2023

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Coeliac disease: the paradox of diagnosing a food hypersensitivity disorder with autoantibodies

Gut, 2024-02, Vol.73 (5), p.844

2024

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Evidence That Pathogenic Transglutaminase 2 in Celiac Disease Derives From Enterocytes

Gastroenterology (New York, N.Y. 1943), 2020-08, Vol.159 (2), p.788-790

2020

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Sa1261: THE DEVELOPMENT AND VALIDATION OF A HIGH CAPACITY SEROLOGICAL ASSAY FOR COELIAC DISEASE

Gastroenterology (New York, N.Y. 1943), 2022-05, Vol.162 (7), p.S-359-S-359

2022

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Transglutaminase 2 strongly binds to an extracellular matrix component other than fibronectin via its second C‐terminal beta‐barrel domain

The FEBS journal, 2016-11, Vol.283 (21), p.3994-4010

2016

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1022 THE TOTAL PREVALENCE AND RATE OF UNDIAGNOSED CELIAC DISEASE IN AN ADULT GENERAL POPULATION – THE HUNT STUDY, NORWAY

Gastroenterology (New York, N.Y. 1943), 2023-05, Vol.164 (6), p.S-218-S-218

2023

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Selective activation of naïve B cells with unique epitope specificity shapes autoantibody formation in celiac disease

Journal of autoimmunity, 2024-05, Vol.146, p.103241-103241, Article 103241

2024

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Efficient T cell–B cell collaboration guides autoantibody epitope bias and onset of celiac disease

Proceedings of the National Academy of Sciences - PNAS, 2019-07, Vol.116 (30), p.15134-15139

2019

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Longevity, clonal relationship, and transcriptional program of celiac disease-specific plasma cells

The Journal of experimental medicine, 2021-02, Vol.218 (2)

2021

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Plasma Cells Are the Most Abundant Gluten Peptide MHC-expressing Cells in Inflamed Intestinal Tissues From Patients With Celiac Disease

Gastroenterology (New York, N.Y. 1943), 2019-04, Vol.156 (5), p.1428-1439.e10

2019

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Sa1248: SCREENING FOR ADULT CELIAC DISEASE IN THE GENERAL POPULATION

Gastroenterology (New York, N.Y. 1943), 2022-05, Vol.162 (7), p.S-353-S-353

2022

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Autoantibody binding and unique enzyme-substrate intermediate conformation of human transglutaminase 3

High-Throughput Single-Cell Analysis of B Cell Receptor Usage among Autoantigen-Specific Plasma Cells in Celiac Disease

Generation of circulating autoreactive pre-plasma cells fueled by naive B cells in celiac disease

Activity-regulating structural changes and autoantibody epitopes in transglutaminase 2 assessed by hydrogen/deuterium exchange

Strong Clonal Relatedness between Serum and Gut IgA despite Different Plasma Cell Origins

Enhanced B-Cell Receptor Recognition of the Autoantigen Transglutaminase 2 by Efficient Catalytic Self-Multimerization

Igs as Substrates for Transglutaminase 2: Implications for Autoantibody Production in Celiac Disease

Coeliac disease in the Trøndelag Health Study (HUNT), Norway, a population-based cohort of coeliac disease patients

Separate Gut Plasma Cell Populations Produce Auto‐Antibodies against Transglutaminase 2 and Transglutaminase 3 in Dermatitis Herpetiformis

Thiol−Disulfide Exchange between Glutaredoxin and Glutathione

Autoimmunity provoked by foreign antigens: In celiac disease, exogenous gluten drives T cell-B cell interactions that cause autoimmunity

Dissecting autoimmune encephalitis through the lens of intrathecal B cells

Autoimmunity provoked by foreign antigens

The Immunobiology and Pathogenesis of Celiac Disease

Tango of B cells with T cells in the making of secretory antibodies to gut bacteria

Coeliac disease: the paradox of diagnosing a food hypersensitivity disorder with autoantibodies

Evidence That Pathogenic Transglutaminase 2 in Celiac Disease Derives From Enterocytes

Sa1261: THE DEVELOPMENT AND VALIDATION OF A HIGH CAPACITY SEROLOGICAL ASSAY FOR COELIAC DISEASE

Transglutaminase 2 strongly binds to an extracellular matrix component other than fibronectin via its second C‐terminal beta‐barrel domain

1022 THE TOTAL PREVALENCE AND RATE OF UNDIAGNOSED CELIAC DISEASE IN AN ADULT GENERAL POPULATION – THE HUNT STUDY, NORWAY

Selective activation of naïve B cells with unique epitope specificity shapes autoantibody formation in celiac disease

Efficient T cell–B cell collaboration guides autoantibody epitope bias and onset of celiac disease

Longevity, clonal relationship, and transcriptional program of celiac disease-specific plasma cells

Plasma Cells Are the Most Abundant Gluten Peptide MHC-expressing Cells in Inflamed Intestinal Tissues From Patients With Celiac Disease

Sa1248: SCREENING FOR ADULT CELIAC DISEASE IN THE GENERAL POPULATION

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