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FDA-approved drugs that protect mammalian neurons from glucose toxicity slow aging dependent on cbp and protect against proteotoxicity

PloS one, 2011-11, Vol.6 (11), p.e27762

2011

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Glucose-Induced Metabolic Memory in Schwann Cells: Prevention by PPAR Agonists

Endocrinology (Philadelphia), 2013-09, Vol.154 (9), p.3054-3066

2013

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Identification of Novel HLA Class II-Restricted Neoantigens Derived from Driver Mutations

Cancers, 2019-02, Vol.11 (2), p.266

2019

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Reversal of diabetic nephropathy by a ketogenic diet

PloS one, 2011-04, Vol.6 (4), p.e18604-e18604

2011

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Role of Hypothalamic Creb-Binding Protein in Obesity and Molecular Reprogramming of Metabolic Substrates

PloS one, 2016-11, Vol.11 (11), p.e0166381-e0166381

2016

Volltextzugriff (PDF)

Regulation of peripheral metabolism by substrate partitioning in the brain

Endocrinology and metabolism clinics of North America, 2013-03, Vol.42 (1), p.67

2013

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Low-carbohydrate diets cause obesity, low-carbohydrate diets reverse obesity: A metabolic mechanism resolving the paradox

Appetite, 2007-03, Vol.48 (2), p.135-138

2007

Volltextzugriff (PDF)

Laparotomy in mice induces blood cell expression of inflammatory and stress genes

Journal of interferon & cytokine research, 2015-04, Vol.35 (4), p.302

2015

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Treatment of Diabetes and Diabetic Complications With a Ketogenic Diet

Journal of child neurology, 2013-08, Vol.28 (8), p.1009-1014

2013

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Abstract 3552: Personalized neoantigen cancer vaccine assembled on DC targeting antibody improves cancer immunity

Cancer research (Chicago, Ill.), 2022-06, Vol.82 (12_Supplement), p.3552-3552

2022

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Rigosertib (RIG) in combination with azacitidine (AZA) to modulate epigenetic effects and to overcome clinical resistance to hypomethylating agents (HMA) in myelodsyplastic syndromes (MDS)

Journal of clinical oncology, 2016-05, Vol.34 (15_suppl), p.7020-7020

2016

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Hypothalamic Fkbp51 is induced by fasting, and elevated hypothalamic expression promotes obese phenotypes

American journal of physiology: endocrinology and metabolism, 2012-04, Vol.302 (8), p.E987-E991

2012

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Role of glucocorticoids in mediating effects of fasting and diabetes on hypothalamic gene expression

BMC physiology, 2003-07, Vol.3 (1), p.5-5

2003

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Inhibition of Agouti-Related Peptide Expression by Glucose in a Clonal Hypothalamic Neuronal Cell Line Is Mediated by Glycolysis, Not Oxidative Phosphorylation

Endocrinology (Philadelphia), 2008-02, Vol.149 (2), p.703-710

2008

Volltextzugriff (PDF)

Estradiol impairs hypothalamic molecular responses to hypoglycemia

Brain research, 2009-07, Vol.1280, p.77-83

2009

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Hypothalamic Fkbp51 is induced by fasting, and elevated hypothalamic expression promotes obese phenotypes

American journal of physiology: endocrinology and metabolism, 2012, Vol.65 (4)

2012

Volltextzugriff (PDF)

Wistar Fatty Rat Is Obese and Spontaneously Hypertensive

Hypertension (Dallas, Tex. 1979), 1995-01, Vol.25 (1), p.146-150

1995

Volltextzugriff (PDF)

Impaired glucose signaling as a cause of obesity and the metabolic syndrome: The glucoadipostatic hypothesis

Physiology & behavior, 2005-05, Vol.85 (1), p.3-23

2005

Volltextzugriff (PDF)

d- chiro-Inositol enhances effects of hypothalamic toxin gold-thioglucose

Brain research, 2003-12, Vol.993 (1), p.172-176

2003

Volltextzugriff (PDF)

T lymphopenia in obese diabetic ( [formula omitted]) mice is non-selective and thymus independent

Life sciences (1973), 1998, Vol.62 (14), p.1243-1250

1998

Volltextzugriff (PDF)

T lymphopenia in genetically obese-diabetic wistar fatty rats: Effects of body weight reduction on T cells

Metabolism, clinical and experimental, 2000-10, Vol.49 (10), p.1261-1266

2000

Volltextzugriff (PDF)

Insufficient expression of Fas antigen on helper T cells in Behçet's disease

British journal of ophthalmology, 1996-02, Vol.80 (2), p.174-176

1996

Volltextzugriff (PDF)

Glucose Hysteresis as a Mechanism in Dietary Restriction, Aging and Disease

Interdisciplinary topics in gerontology, 2007-01, Vol.35, p.39-68

2007

Volltextzugriff (PDF)

Secrets of the lac operon. Glucose hysteresis as a mechanism in dietary restriction, aging and disease

Interdisciplinary topics in gerontology, 2007, Vol.35, p.39

2007

Volltextzugriff (PDF)

Secrets of the lac Operon

Mechanisms of Dietary Restriction in Aging and Disease, 2006, Vol.35, p.39-68

2006

Volltextzugriff (PDF)

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FDA-approved drugs that protect mammalian neurons from glucose toxicity slow aging dependent on cbp and protect against proteotoxicity

Glucose-Induced Metabolic Memory in Schwann Cells: Prevention by PPAR Agonists

Identification of Novel HLA Class II-Restricted Neoantigens Derived from Driver Mutations

Reversal of diabetic nephropathy by a ketogenic diet

Role of Hypothalamic Creb-Binding Protein in Obesity and Molecular Reprogramming of Metabolic Substrates

Regulation of peripheral metabolism by substrate partitioning in the brain

Low-carbohydrate diets cause obesity, low-carbohydrate diets reverse obesity: A metabolic mechanism resolving the paradox

Laparotomy in mice induces blood cell expression of inflammatory and stress genes

Treatment of Diabetes and Diabetic Complications With a Ketogenic Diet

Abstract 3552: Personalized neoantigen cancer vaccine assembled on DC targeting antibody improves cancer immunity

Rigosertib (RIG) in combination with azacitidine (AZA) to modulate epigenetic effects and to overcome clinical resistance to hypomethylating agents (HMA) in myelodsyplastic syndromes (MDS)

Hypothalamic Fkbp51 is induced by fasting, and elevated hypothalamic expression promotes obese phenotypes

Role of glucocorticoids in mediating effects of fasting and diabetes on hypothalamic gene expression

Inhibition of Agouti-Related Peptide Expression by Glucose in a Clonal Hypothalamic Neuronal Cell Line Is Mediated by Glycolysis, Not Oxidative Phosphorylation

Estradiol impairs hypothalamic molecular responses to hypoglycemia

Hypothalamic Fkbp51 is induced by fasting, and elevated hypothalamic expression promotes obese phenotypes

Wistar Fatty Rat Is Obese and Spontaneously Hypertensive

Impaired glucose signaling as a cause of obesity and the metabolic syndrome: The glucoadipostatic hypothesis

d- chiro-Inositol enhances effects of hypothalamic toxin gold-thioglucose

T lymphopenia in obese diabetic ( [formula omitted]) mice is non-selective and thymus independent

T lymphopenia in genetically obese-diabetic wistar fatty rats: Effects of body weight reduction on T cells

Insufficient expression of Fas antigen on helper T cells in Behçet's disease

Glucose Hysteresis as a Mechanism in Dietary Restriction, Aging and Disease

Secrets of the lac operon. Glucose hysteresis as a mechanism in dietary restriction, aging and disease

Secrets of the lac Operon

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